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    Ocular

    Trauma

    Blok 24Abraham Adiwidjaja Sutjiono

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    BETT: Birmingham Eye Trauma Terminology

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    Term - Definition and Explanation

    Eyewall : Sclera and cornea

    Although technically the eyewall has three coats posterior tothe limbus, for clinical and practical purposes, violation ofonly the most external structure is taken into consideration)

    Closed globe injury : No full-thickness wound of eyewall Open globe injury: Full-thickness wound of the eyewall

    Contusion: There is no (full-thickness) wound.

    The injury is due to either direct energy delivery by the object

    (e.g., choroidal rupture) or the changes in the shape of theglobe (e.g., angle recession)

    Lamellar laceration: Partial-thickness wound of theeyewall

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    Term - Definition and Explanation

    Rupture: Full-thickness wound of the eyewall, caused by a bluntobject (Because the eye is filled with incompressible liquid, the impactresults in momentary increase in IOP.) The eyewall yields at itsweakest point (at the impact site or elsewhere; e.g., an old cataractwound dehisces even though the impact occurred elsewhere); theactual wound is produced by an inside-out mechanism

    Laceration: Full-thickness wound of the eyewall, caused by a sharpobject. The wound occurs at the impact site by an outside-inmechanism

    Penetrating injury: Entrance wound . If more than one wound is present, each must have

    been caused by a different agent. Retained foreign object(s) Technically a penetrating injury, but grouped

    separately because of different clinical implications

    Perforating injury: Entrance and exit wounds. Both wounds caused bythe same agent

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    OPEN GLOBE INJURY CLASSIFICATION

    Type

    A. Rupture B. Penetrating

    C. IOFB

    D. Perforating

    E. Mixed

    Grade (Visual acuity)

    A. 20/40 B. 20/50 to 20/100

    C. 19/100 to 5/200

    D. 4/200 to light perception

    E. NLP

    Pupil

    A. Positive, relative APD in injured eye

    B. Negative, relative APD in injured eye

    Zone

    I. Cornea and limbus

    II. Limbus to 5 mm posterior into sclera

    III. Posterior to 5 mm from the limbus

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    CLOSED GLOBE INJURY CLASSIFICATION

    Type

    A. Contusion B. Lamellar laceration

    C. Superficial foreign body

    D. Mixed

    Grade (Visual acuity) A. 20/40

    B. 20/50 to 20/100

    C. 19/100 to 5/200 D. 4/200 to light perception

    E. NLP

    Pupil A. Positive, relative APD in injured eye

    B. Negative, relative APD in injured eye

    Zone (see Fig. 22) I. External (limited to bulbar conjunctiva, sclera, cornea)

    II. Anterior segment (includes structures of the anterior segment and the pars plicata)

    III. Posterior segment (all internal structures posterior to the posterior lens capsule)

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    Eyelid Laceration

    Symptoms

    Periorbital pain, tearing.

    Signs

    Partial or full-thickness defect in the eyelid involving

    the skin and subcutaneous tissues. Superficial

    laceration/abrasion may mask a deep laceration or

    injury to the lacrimal drainage system (e.g., puncta,canaliculi, common canaliculus, lacrimal sac).

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    Eyelid Laceration contd

    Work-Up

    History: Determine mechanism of injury: bite, foreign body

    potential, etc.

    Complete ocular examination, including bilateral dilated

    fundus evaluation. Make sure there is no injury to the globes

    and optic nerves before attempting eyelid repair.

    Determine the depth of the laceration (can look deceptively

    superficial). Use toothed forceps or cotton-tipped applicators

    to gently pull open one edge of the wound to determine

    depth of penetration.

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    Eyelid Laceration contd

    Work-Up

    CT scan of brain and orbits (axial and coronal views, 1- to 3-

    mm sections) should be obtained when a foreign body,

    ruptured globe, or severe blunt trauma is suspected. Loss of

    consciousness necessitates CT scan of the brain. Depending

    on the mechanism of injury, the cervical spine may need to

    be cleared.

    If laceration is nasal to either the upper or lower eyelid

    punctum, even if not obviously through the canalicularsystem, perform punctal dilation and irrigation of the

    canalicular system to exclude canalicular involvement

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    Eyelid Laceration contd

    Treatment

    Consider tetanus prophylaxis

    Give systemic antibiotics if contamination or foreign body is

    suspected [e.g., dicloxacillin or cephalexin, 250 to 500 mg

    p.o., q.i.d. (adults); 25 to 50 mg/kg/day divided into four

    doses (children); for human or animal bites, consider

    penicillin V (same dose as dicloxacillin)]. Continue for 5 to 7

    days. For animal bites, if indicated, consider rabies

    prophylaxis.

    Assess eyelid laceration.

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    Eyelid Laceration contd

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    Eyelid Laceration contd

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    Conjunctival Laceration

    Symptoms:

    Mild pain, red eye, foreign body sensation; usually, a

    history of ocular trauma.

    Signs

    Fluorescein staining of the conjunctiva. The

    conjunctiva may be torn and rolled up on itself.

    Exposed white sclera may be noted. Conjunctivaland subconjunctival hemorrhages are often present.

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    Conjunctival Laceration contd

    Work-Up

    History: Determine the nature of the trauma and whether a rupturedglobe or intraocular or intraorbital foreign body may be present.Evaluate mechanism for possible foreign body involvement, includingsize, shape, weight, and velocity of object.

    Complete ocular examination, including careful exploration of thesclera (after topical anesthesia, e.g., proparacaine) in the region of theconjunctival laceration to rule out a scleral laceration or asubconjunctival foreign body. The entire area of sclera under theconjunctival laceration must be inspected. Since the conjunctiva ismobile, inspect a wide area of the sclera under the laceration. Use aproparacaine-soaked, sterile cotton-tipped applicator to manipulatethe conjunctiva. A Seidel test may be helpful. Dilated fundusexamination, especially evaluating the area underlying theconjunctival injury, must be carefully performed with indirectophthalmoscopy.

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    Conjunctival Laceration contd

    Work-Up

    Consider a CT scan of the orbit (axial and coronal views, 1-

    mm sections) to exclude an intraocular or intraorbital

    foreign body. UBM may be helpful.

    Exploration of the site in the operating room under

    general anesthesia may be necessary when a ruptured

    globe is suspected.

    Children often do not give an accurate history of trauma.

    They must be questioned and examined especially

    carefully.

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    Conjunctival Laceration contd

    Treatment

    Antibiotic ointment (e.g., erythromycin or bacitracin

    t.i.d.) for 4 to 7 days. A pressure patch may be used

    for the first 24 hours. Most lacerations will heal without surgical repair.

    Some large lacerations (>1 to 1.5 cm) may be

    sutured with 8-0 polyglactin 910 (e.g., Vicryl). When

    suturing, take care not to bury folds of conjunctiva

    or incorporate Tenon capsule into the wound. Avoid

    suturing the plica semilunaris or caruncle to the

    conjunctiva.

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    Conjunctival Laceration contd

    Conjunctival laceration vs subconjunctival

    bleeding vs scleral foreign bodies vs scleral

    rupture?

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    Scleral foreign body. (A) The obvious finding is a small subconjunctival hemorrhage.

    (B) With higher magnification and a slit illumination, the brassfragment from a .22 caliber bullet casing is seen to be transfixingthe sclera. In this case, 90% of the scleral foreign body was insidethe globe. The conjunctival laceration/hemorrhage did not overliethe scleral defect, a common situation in combined wounds of theconjunctiva and sclera.

    (C) The removed foreign body.

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    Corneal Laceration

    Can be partial or full thickness corneal

    laceration

    Parital-Thickness Laceration

    Signs

    The anterior chamber is not entered and, therefore,

    the cornea is not perforated.

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    Corneal Laceration contd

    Parital-Thickness Laceration

    Work-Up Careful slit-lamp examination should be performed to exclude

    ocular penetration. Carefully evaluate the conjunctiva, sclera, andcornea, checking for extension beyond the limbus in casesinvolving the corneal periphery. Evaluate the depth of the AC and

    compare with the fellow eye. A shallow AC indicates an activelyleaking wound or a self-sealed leak. Deeper AC in the involved eyecan be an indication of a posterior rupture. Check iris fortransillumination defects (TIDs) and evaluate lens for a cataract ora foreign body tract (must have a high level of suspicion withprojectile objects). Presence of TIDs and lens abnormalities are an

    indication of a ruptured globe. IOP should be measured only aftera ruptured globe is ruled out. Use applanation only if thelaceration site can be avoided. Otherwise, use a Tono-Pen to checkthe IOP.

    Seidel test. If the Seidel test is positive, a full-thickness lacerationis present.

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    Corneal Laceration contd

    Parital-Thickness Laceration

    Treatment

    A cycloplegic (e.g., scopolamine 0.25%) and an antibiotic

    [e.g., frequent polymyxin B/ bacitracin ointment (e.g.,Polysporin) or fluoroquinolone drops, depending on thenature of the wound].

    When a moderate to deep corneal laceration isaccompanied by wound gape, it is often best to suture

    the wound closed in the operating room to avoidexcessive scarring and corneal irregularity, especially inthe visual axis.

    Tetanus toxoid for dirty wounds

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    Corneal Laceration contd

    Follow-Up Reevaluate daily until the epithelium heals.

    Full-Thickness Laceration See Ruptured Globe and Penetrating Ocular Injury.

    Note that small, self-sealing, or slow-leakinglacerations may be treated with aqueous

    suppressants, bandage soft contact lenses,fluoroquinolone drops q.i.d. Alternatively, apressure patch and twice-daily antibiotics may beused. Avoid topical steroids.

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    Corneal Laceration

    Full-thickness corneal laceration

    with positive Seidel test

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    Chemical Injuries

    Acids vs alkalis

    Alkalies readily penetrate into the eye,

    damaging the corneal stroma and endothelium

    as well as other anterior segment structures(e.g., iris, lens, ciliary body). Most acids tend to

    remain confined to the ocular surface.

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    Chemical Injuries contd

    Work-up: elimination of residual alkali or acid from the eye;

    institution of topical and systemic medical therapy tominimize adverse sequelae.

    Treatment:

    Irrigate copiously IMMEDIATELY :

    No therapeutic differences have been identified betweennormal saline, normal saline with bicarbonate, lactatedRingers, balanced salt solution (BSS), and BSS-plus.

    Try to use other neutral fluids

    As it is impossible to overirrigate a chemically injured eye,

    irrigation for 15 to 30 minutes is recommended.

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    Chemical Injuries contd

    An eyelid speculum and topical anesthetic (e.g.,proparacaine) can be placed prior to irrigation. Upper andlower fornices must be everted and irrigated

    NEVER use acidic solutions to neutralize alkalis or vice versaas acid-base reactions themselves can generate harmful

    substrates Check the pH a few minutes after irrigation

    Debridement

    Medical therapy Topical corticosteroids

    Topical antibiotics

    Glaucoma medications

    Cycloplegics

    Sodium ascorbat

    Artificial tears

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    Chemical Injuries contd

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    Corneal and Conjunctival Foreign Bodies

    Symptoms

    Foreign body sensation, tearing, history of trauma.

    Signs

    Critical. Conjunctival or corneal foreign body with or

    without rust ring.

    Other. Conjunctival injection, eyelid edema, mild AC

    reaction, and SPK. A small infiltrate may surround acorneal foreign body; it is usually sterile. Vertically

    oriented linear corneal abrasions or SPK may

    indicate a foreign body under the upper eyelid.

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    Corneal and Conjunctival Foreign Bodies Contd

    Work-Up

    History: Determine the mechanism of injury [e.g.,

    metal striking metal, power tools or weed-whackers

    may suggest an intraocular foreign body (IOFB)].Attempt to determine the size, weight, velocity,

    force, and shape of the object. Safety goggles?

    Document visual acuity before any procedure is

    performed. One or two drops of topical anesthetic

    may be necessary to control blepharospasm and

    pain.

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    Corneal and Conjunctival Foreign Bodies Contd

    Work-Up Slit-lamp examination: Locate and assess the depth of

    the foreign body. Rule out self-sealing lacerations.Examine closely for iris tears and transillumination

    defects, lens opacities, AC shallowing, andasymmetrically low IOP in the involved eye.

    If there is no evidence of perforation, evert the eyelidsand inspect the fornices for additional foreign bodies.

    Double-everting the upper eyelid with a Desmarreseyelid retractor may be necessary. Carefully inspect aconjunctival laceration to rule out a scleral laceration orperforation. Measure the dimensions of any infiltrateand the degree of any AC reaction.

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    Corneal and Conjunctival Foreign Bodies Contd

    Work-Up

    Dilate the eye and examine the posterior segment

    for a possible IOFB

    Consider a B-scan ultrasonography (UBM), acomputed tomography (CT) scan of the orbit (axial

    and coronal views, 1-mm sections), or

    ultrasonographic biomicroscopy (UBM) to exclude

    an intraocular or intraorbital foreign body. Avoid

    magnetic resonance imaging (MRI) if there is a

    history of possible metallic foreign body.

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    Corneal and Conjunctival Foreign Bodies Contd

    Innert vs non-innert FB

    Principle: take it out in fashioned ways..

    Corneal metallic foreign body with rust ring

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    Corneal and Conjunctival Foreign Bodies Contd

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    Corneal foreign body - Treatment

    Apply topical anesthetic (e.g., proparacaine). Remove thecorneal foreign body with a foreign body spud or fine forceps

    at a slit lamp. Multiple superficial foreign bodies may be more

    easily removed by irrigation.

    Remove the rust ring as completely as possible on the firstattempt. It is sometimes safer to leave a deep, central rust

    ring to allow time for the rust to migrate to the corneal

    surface, at which point it can be removed more easily.

    Measure the size of the resultant corneal epithelial defect.

    Treat as for corneal abrasion

    Alert the patient to return as soon as possible if there is any

    worsening of symptoms.

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    Conjunctival foreign body - Treatment

    Remove foreign body under topical anesthesia. Multiple or loose foreign bodies can often be removed

    with saline irrigation.

    A foreign body can be removed with a cotton-tipped

    applicator soaked in topical anesthetic or with fine forceps. For deeply embedded foreign

    bodies, consider pretreatment with a cotton-tippedapplicator soaked in phenylephrine 2.5% to reduceconjunctival bleeding.

    Small, relatively inaccessible, buried subconjunctivalforeign bodies may sometimes be left in the eye withoutharm unless they are infectious or proinflammatory.Occasionally, they will surface with time, at which pointthey may be removed more easily.

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    Conjunctival foreign body - Treatment

    Sweep the conjunctival fornices with a glass rod

    or cotton-tipped applicator soaked with a

    topical anesthetic to catch any remaining

    pieces. A topical antibiotic (e.g., bacitracin ointment

    b.i.d.; trimethoprim/polymyxin B or

    fluoroquinolone drops q.i.d.) may be used. Artificial tears (e.g., Refresh q.i.d. for 2 days)

    may be given for irritation.

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    Follow-up

    Corneal foreign body: Follow up as with corneal

    abrasion. If residual rust ring remains,

    reevaluate in 24 hours.

    Conjunctival foreign body: Follow up as needed,or in 1 week if residual foreign bodies were left

    in the conjunctiva.

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    Hyphema

    Blood in the AC commonly accumulates in case

    of (closed as well as open) globe trauma.

    Consequences include IOP elevation, corneal

    blood staining, the formation ofanterior/posterior synechiae, cataract, and a

    wide variety of indirectly related pathologic

    changes.

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    Hyphema

    Symptoms

    Pain, blurred vision, history of blunt trauma.

    Signs

    Blood or clot or both in the AC, usually visible

    without a slit lamp. A total (100%) hyphema may be

    black or red. When black, it is called an 8-ball or

    black ball hyphema; when red, the circulatingblood cells may settle with time to become less than

    a 100% hyphema.

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    Hyphema contd

    Work-Up History: Mechanism (including force, velocity, type,

    and direction) of injury? Protective eyewear? Timeof injury? Time of visual loss? Usually the visualcompromise occurs at the time of injury; decreasingvision over time suggests a rebleed or continuedbleed. Use of medications with anticoagulantproperties [aspirin, NSAIDs, warfarin (e.g.,

    Coumadin), or clopidogrel (e.g., Plavix)]? Personal orfamily history of sickle cell disease/trait? Symptomsof coagulopathy (e.g., bloody nose-blowing,bleeding gums with tooth brushing, easy bruising)?

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    Hyphema contd

    Work-Up Ocular examination, first ruling out a ruptured globe

    Evaluate for other traumatic injuries. Document theextent (e.g., measure the hyphema height) and location

    of any clot and blood. Measure the IOP. Perform adilated retinal evaluation without scleral depression.Consider a gentle UBM if the view of the fundus is poor.Avoid gonioscopy unless intractable increased IOPdevelops. If gonioscopy is necessary, gently use a Zeiss 4mirror lens. Consider UBM to evaluate the anteriorsegment if the view is poor and lens capsule rupture,IOFB, or other anterior segment abnormalities aresuspected.

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    Hyphema contd

    Work-Up

    Consider a CT scan of the orbits and brain (axial and

    coronal views, with 1- to 3-mm sections through the

    orbits) when indicated (e.g., suspected orbitalfracture or IOFB, loss of consciousness).

    Black and Mediterranean patients should be

    screened for sickle cell trait or disease (order

    Sickledex screen; if necessary, may checkhemoglobin electrophoresis).

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    Hyphema contd

    Treatment Confine either to bed rest with bathroom privileges or

    to limited activity. Elevate head of bed to allow blood tosettle.

    Place a shield (metal or clear plastic) over the involvedeye at all times. Do not patch because this preventsrecognition of sudden visual loss in the event of arebleed.

    Atropine 1% solution b.i.d. to t.i.d. or scopolamine

    0.25% b.i.d. to t.i.d. No aspirin-containing products or NSAIDs.

    Mild analgesics only (e.g., acetaminophen). Avoidsedatives.

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    Hyphema contd

    Treatment

    Use topical steroids (e.g., prednisolone acetate 1%

    four to eight times per day) if any suggestion of iritis

    (e.g., photophobia, deep ache, ciliary flush),evidence of lens capsule rupture, any protein (e.g.,

    fibrin), or definitive white blood cells in anterior

    chamber. Reduce the frequency of steroids as soon

    as signs and symptoms resolve to reduce thelikelihood of steroid-induced glaucoma.

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    Hyphema contd

    Treatment For increased IOP:

    Start with a beta-blocker (e.g., timolol or levobunolol 0.5%b.i.d.).

    If IOP still high, add topical alphaagonist (e.g., apraclonidine0.5%, or brimonidine 0.2% t.i.d.) or topical carbonic anhydraseinhibitor (e.g., dorzolamide 2%, or brinzolamide 1% t.i.d.).Avoid prostaglandin analogs and miotics (may increaseinflammation). In children under 5, topical alphaagonists are

    contraindicated. If topical therapy fails, add acetazolamide (500 mg p.o., q12h

    for adults, 20 mg/kg/day divided three times per day forchildren) or mannitol [1 to 2 g/kg intravenously (i.v.) over 45minutes q24h]. If mannitol is necessary to control the IOP,

    surgical evacuation may be imminent.

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    Hyphema contd

    Treatment If hospitalized, use antiemetics p.r.n. for severe nausea or

    vomiting [e.g., prochlorperazine 10 mg intramuscularly (i.m.)q8h or 25 mg q12h p.r.n.; 48 hours, despite maximal medical therapy (toprevent optic atrophy).

    IOP >25 mm Hg with total hyphema for >5 days (to prevent cornealstromal blood staining).

    IOP 24 mm Hg for >24 hours (or any transient increase in IOP >30 mmHg) in sickle trait/disease patients.

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    Hyphema contd

    Follow-Up The patient should be seen daily for 3 days after initial

    trauma to check visual acuity, IOP, and for a slit-lampexamination. Look for new bleeding, increased IOP,corneal blood staining, and other intraocular injuries asthe blood clears (e.g., iridodialysis; subluxated,disclocated, or cataractous lens). Hemolysis, which mayappear as bright red fluid, should be distinguished froma rebleed, which forms a new, bright red clot. If the IOPis increased, treat as described earlier.

    The patient should be instructed to return immediatelyif a sudden increase in pain or decrease in vision isnoted (which may be symptoms of a rebleed orsecondary glaucoma).

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    Hyphema contd

    Follow-Up If a significant rebleed or an intractable IOP increase

    occurs, the patient should be hospitalized.

    After the initial close follow-up period, the patient may

    be maintained on a long-acting cycloplegic (e.g.,atropine 1% solution q.d. to b.i.d., scopolamine 0.25%q.d. to b.i.d.), depending on the severity of thecondition. Topical steroids may be tapered as the blood,

    fibrin, and white blood cells resolve. Glasses or eye shield during the day and eye shield at

    night. As with any patient, protective eyewear(polycarbonate or Trivex lenses) should be worn anytime significant potential for an eye injury exists.

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    Hyphema contd

    Follow-Up

    The patient must refrain from strenuous physical

    activities (including bearing down or Valsalva

    maneuvers) for 1 week after the initial injury orrebleed. Normal activities may be resumed 1 week

    from the date of injury or rebleed. This period

    should be extended if blood remains in the anterior

    chamber.

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    Orbital Blow-Out Fracture

    Symptoms

    Pain on attempted eye movement (orbital floor

    fracture: pain on vertical eye movement; medial

    wall fracture: pain on ab-/adduction), localtenderness, eyelid edema, binocular diplopia,

    crepitus after nose-blowing, recent history of

    trauma. Tearing may be a symptom of nasolacrimal

    duct fracture seen with medial buttress or Leforte IIfractures, but this is typically a late complaint. Acute

    tearing is usually due to ocular surface irritation

    (e.g., conjunctival chemosis, corneal abrasion, iritis).

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    Orbital Blow-Out Fracture contd

    Signs Critical. Restricted eye movement (especially in upward

    or lateral gaze or both), subcutaneous or conjunctivalemphysema, hypesthesia in the distribution of theinfraorbital nerve (i.e., ipsilateral cheek and upper lip),point tenderness, enophthalmos (may initially bemasked by orbital edema).

    Other. Nosebleed, eyelid edema, and ecchymosis.Superior rim and orbital roof fractures may show

    hypesthesia in the distribution of the supratrochlear orsupraorbital nerve (ipsilateral forehead) and ptosis.Trismus, malar flattening, and a palpable step-offdeformity of the inferior orbital rim are characteristic oftripod (zygomatic complex) fractures. Optic neuropathymay be present.

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    Orbital Fracture

    Palpebral Edema Ophthalmoplegi

    Enopthalmus

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    Orbital Blow-Out Fracture contd

    Differential Diagnosis

    Orbital edema and hemorrhage without a blow-out

    fracture: May have limitation of ocular movement,

    periorbital swelling, and ecchymosis due to soft-tissue edema and hemorrhage, but these resolve

    over 7 to 10 days.

    Cranial nerve palsy: Limitation of ocular movement,

    but no restriction on forced-duction testing. Willhave abnormal results on force generation testing.

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    Orbital Blow-Out Fracture contd

    Work-Up Complete ophthalmologic examination, including

    measurement of extraocular movements and globedisplacement. Compare the sensation of the affectedcheek with that on the contralateral side; palpate theeyelids for crepitus (subcutaneous emphysema); palpatethe orbital rim for step-offs; evaluate the globe carefullyfor a rupture, hyphema or microhyphema, traumaticiritis, and retinal or choroidal damage. Measure IOP.Check pupils and color vision to rule out a traumaticoptic neuropathy If eyelid and periocular edema limitthe view, special techniques may be necessary (e.g., useof Desmarres retractors, lateral cantholysis,examination under general anesthesia).

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    Orbital Blow-Out Fracture contd

    Work-Up

    Forced-duction testing is performed if restriction of

    eye movement persists beyond one week.

    CT orbit scans (axial and coronal views, 3-mmsections, without contrast) are obtained in all cases

    of suspected orbital fractures. Bone windows are

    especially helpful in evaluation of fractures. If there

    is any history of loss of consciousness, brain imagingis required.

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    Orbital Blow-Out Fracture contd

    Treatment

    Broad-spectrum oral antibiotics [e.g., cephalexin

    (Keflex) 250 to 500 mg p.o., q.i.d.; or erythromycin

    250 to 500 mg p.o., q.i.d.] for 7 days. The use ofprophylactic antibiotics in orbital fracture is

    controversial. Antibiotics are recommended if the

    patient has a history of sinusitis, diabetes, or is

    otherwise immunocompromised. In all otherpatients, the decision about antibiotic use is left up

    to the treating physician.

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    Orbital Blow-Out Fracture contd

    Treatment Instruct patient not to blow his or her nose.

    Nasal decongestants [e.g., pseudoephedrine (Afrin)

    nasal spray b.i.d.] for 3 days. Use is limited to 3 days tominimize the chance of rebound nasal congestion.

    Apply ice packs to the orbit for the first 24 to 48 hours.

    Consider oral steroids (e.g., Medrol dose pack) if

    extensive swelling limits examination of ocular motilityand globe position. If corticosteroids are used, systemic

    antibiotics should also be considered.

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    Orbital Blow-Out Fracture contd

    Treatment Neurosurgical consultation is recommended for all

    fractures involving the orbital roof, frontal sinus, orcribriform plate and for all fractures associated with

    intracranial hemorrhage. Otolaryngology or oralmaxillofacial surgery consultation is recommendedfor frontal sinus, midfacial, and mandibularfractures.

    Surgical repair: Immediate repair (within 24 to 72 hours)

    Repair in 1 to 2 weeks

    Delayed Repair

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    Ruptured Globe and Penetrating Ocular Injury

    Symptoms Pain, decreased vision, loss of fluid from eye. History of

    trauma, fall, or sharp object entering globe.

    Signs

    Critical. Full-thickness scleral or corneal laceration,severe subconjunctival hemorrhage (especially involving360 degrees of bulbar conjunctiva, often bullous), adeep or shallow AC compared to the fellow eye, peaked

    or irregular pupil, iris TIDs, lens material in the AC,foreign body tract in the lens, or limitation ofextraocular motility (greatest in direction of rupture).Intraocular contents may be outside of the globe.

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    Ruptured Globe and Penetrating Ocular Injury

    Signs

    Other. Low IOP (although it may be normal or

    increased), iridodialysis, cyclodialysis, hyphema (i.e.,

    clotted blood in AC), periorbital ecchymosis,vitreous hemorrhage, dislocated or subluxed lens,

    and traumatic optic neuropathy. Commotio retinae,

    choroidal rupture, and retinal breaks may be seen

    but are often obscured by vitreous hemorrhage.

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    Ruptured Globe and Penetrating Ocular Injury

    Work-Up/Treatment

    Once the diagnosis of a ruptured globe is made,

    further examination should be deferred until the

    time of surgical repair in the operating room. This isto avoid placing any pressure on the globe and

    risking extrusion of the intraocular contents.

    Diagnosis should be made by penlight, or if possible,

    by slit-lamp examination (with very gentlemanipulation). Once the diagnosis is made, then the

    following measures should be taken:

    Protect the eye with a shield at all times.

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    Ruptured Globe and Penetrating Ocular Injury

    Work-Up/Treatment Obtain CT scan of the brain and orbits (axial and coronal

    views, 1-mm sections) to rule out IOFB in most cases.

    Gentle UBM may be needed to localize posterior rupture

    site(s) or to rule out intraocular foreign bodies not visibleon CT scan (nonmetallic, wood, etc.). However, UBMshould not be done in patients with an obvious anteriorrupture for the risk of extrusion of intraocular contents. Atrained ophthalmologist should evaluate the patient

    before UBM or other manipulation is performed on aruptured globe suspect.

    Admit patient to the hospital with no food or drink (NPO).

    Place patient on bed rest with bathroom privileges. Avoid

    bending over and Valsalva maneuvers.

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    Ruptured Globe and Penetrating Ocular Injury

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    Ruptured Globe and Penetrating Ocular Injury

    Work-Up/Treatment Systemic antibiotics should be administered within 6 hours of injury.

    For adults, give cefazolin 1 g i.v. q8h or vancomycin 1 g i.v. q12h. Alsogive ciprofloxacin 400 mg p.o./i.v. b.i.d. (fourth-generationfluoroquinolones, such as gatifloxacin 400 mg q.d. or moxifloxacin

    400 mg q.d. may have better vitreous penetration). For children