case study in dm
TRANSCRIPT
Diabetes MellitusType II
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INTRODUCTION
WHAT IS DIABETES MELLITUS?
Diabetes is a disease in which the body doesn't produce or properly use insulin.
Insulin is a hormone produced in the pancreas, an organ near the stomach. Insulin is
needed to turn sugar and other food into energy. When you have diabetes, your body
either doesn’t make enough insulin or can’t use its own insulin as well as it should, or
both. This causes sugars to build up too high in your blood.
Diabetes mellitus is defined as a fasting blood glucose of 126 milligrams per
deciliter (mg/dL) or more. “Pre-diabetes” is a condition in which blood glucose levels
are higher than normal but not yet diabetic. People with pre-diabetes are at increased risk
for developing type 2 diabetes, heart disease and stroke, and have one of these conditions:
impaired fasting glucose (100 to 125 mg/dL)
impaired glucose tolerance (fasting glucose less than 126 mg/dL and a glucose
level between 140 and 199 mg/dL two hours after taking an oral glucose tolerance
test)
EPIDEMIOLOGY
The World Health Organization (WHO) estimates that more than 180 million
people worldwide have diabetes. This number is likely to more than double by
2030.
In 2005, an estimated 1.1 million people died from diabetes.1
Almost 80% of diabetes deaths occur in low and middle-income countries.
Almost half of diabetes deaths occur in people under the age of 70 years; 55% of
diabetes deaths are in women.
WHO projects that diabetes deaths will increase by more than 50% in the next 10
years without urgent action. Most notably, diabetes deaths are projected to
increase by over 80% in upper-middle income countries between 2006 and 2015
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TYPES OF DIABETES:
Type 1 diabetes (previously known as insulin-dependent or childhood-onset) is
characterized by a lack of insulin production. Without daily administration of
insulin, Type 1 diabetes is rapidly fatal.
Symptoms include excessive excretion of urine (polyuria), thirst
(polydipsia), constant hunger, weight loss, vision changes and fatigue.
These symptoms may occur suddenly.
Type 2 diabetes (formerly called non-insulin-dependent or adult-onset) results
from the body’s ineffective use of insulin. Type 2 diabetes comprises 90% of
people with diabetes around the world, and is largely the result of excess body
weight and physical inactivity.
Symptoms may be similar to those of Type 1 diabetes, but are often less
marked. As a result, the disease may be diagnosed several years after
onset, once complications have already arisen.
Until recently, this type of diabetes was seen only in adults but it is now
also occurring in obese children.
Gestational diabetes is hyperglycaemia which is first recognized during
pregnancy.
Symptoms of gestational diabetes are similar to Type 2 diabetes.
Gestational diabetes is most often diagnosed through prenatal screening,
rather than reported symptoms.
Impaired Glucose Tolerance (IGT) and Impaired Fasting Glycaemia (IFG) are
intermediate conditions in the transition between normality and diabetes. People with
IGT or IFG are at high risk of progressing to type 2 diabetes, although this is not
inevitable.
Causes of Type 1 Diabetes:
Autoimmune response: For unknown reasons, in some people the immune system
mistakenly destroys the insulin producing cells (called beta cells) in the pancreas.
It is thought that some people inherit genes that mistakenly tell the body to
recognize its beta cells as foreign invaders; then, when one gets the flu or some
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type of virus, as the body is busy killing the foreign virus, it also kills its own beta
cells because it mistakenly thinks the beta cells are invaders as well. Researchers
have learned that there are certain types of gene combinations that predispose
some people to type 1 diabetes.
Oxygen free radicals: These are formed as a by-product of chemical reactions in
the body, from exposure to smoke, air pollution, diet, and other things. Some
researchers believe that oxygen free radicals could contribute to the destruction of
pancreatic cells.
Chemicals and drugs: There are certain rare drugs that can cause diabetes.
Causes of Type 2 Diabetes:
Genetics
Insulin resistance: Researchers are still looking for the cause of insulin resistance,
but in essence either there are not enough receptors on the cell, or the receptors
aren't responding properly.
Aging, obesity, and lifestyle: Obesity is much more common in diabetes, and
there may be a genetic cause for obesity. We know from recent results of the
Diabetes Prevention Trial that lifestyle changes (modest weight reduction and
moderate regular exercise) can prevent the development of type 2 diabetes in
some people. Age is also a factor, partly due to the fact that as people age, they
might become more overweight and more sedentary, although that is not always
the case; some people simply inherit the defect that causes their pancreas to
decrease insulin production as they age.
Causes of Gestational Diabetes
Hormones: During pregnancy, the placenta produces hormones important for the
baby's growth; but these hormones cause some insulin resistance and make the
body resemble that of a person with type 2 diabetes.
Genetics: Researchers suspect that the genes responsible for type 2 diabetes and
gestational diabetes may be similar.
Obesity: Gestational diabetes is more common in women who are obese.
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SIGNS AND SYMPTOMS of hyperglycemia:
1. Frequent urination ( the body tries to rid itself of excess sugar through urine )
2. Thirst (to flush the system of the excess sugar).
3. Weight loss (the body breaks down stored fats to use as energy).
4. Constant hunger (trying to meet the body’s need for energy).
5. Feeling weak and tired (cells are starving, not receiving sugar for energy).
6. Itchy or dry skin
7. Blurry eyesight
8. Skin or bladder infections
9. Slow healing of cuts/bruises
10. Some people show no signs at all
Hypoglycemia also called: Low blood sugar/Insulin reaction/insulin shock/too little
sugar/too much insulin/too many diabetic pills. Low blood sugar usually comes on
quickly sometimes in minutes.
Causes of low blood sugar:
1. Skipping a meal or snack
2. Too much or wrong type of insulin or diabetic pills
3. Injecting insulin into or near muscles
4. Prescription or nonprescription medications can lower blood sugar
Signs and symptoms of hypoglycemia
1. Headache
2. Sweat
3. Feeling hungry
4. Tired/sleepy
5. Behavior change, irritable or cross
6. Blurry vision
7. Passing out (syncope)
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WHAT ARE COMMON CONSEQUENCES OF DIABETES?
Over time, diabetes can damage the heart, blood vessels, eyes, kidneys, and nerves.
Diabetic retinopathy is an important cause of blindness, and occurs as a result of
long-term accumulated damage to the small blood vessels in the retina. After 15
years of diabetes, approximately 2% of people become blind, and about 10%
develop severe visual impairment.
Diabetic neuropathy is damage to the nerves as a result of diabetes, and affects up
to 50% of people with diabetes. Although many different problems can occur as a
result of diabetic neuropathy, common symptoms are tingling, pain, numbness, or
weakness in the feet and hands.
Combined with reduced blood flow, neuropathy in the feet increases the chance of
foot ulcers and eventual limb amputation.
Diabetes is among the leading causes of kidney failure. 10-20% of people with
diabetes die of kidney failure.
Diabetes increases the risk of heart disease and stroke. 50% of people with
diabetes die of cardiovascular disease (primarily heart disease and stroke).
The overall risk of dying among people with diabetes is at least double the risk of
their peers without diabetes.
EXAMS AND TESTS
If the patient is having symptoms but are not known to have diabetes, evaluation
should always begin with a thorough medical interview and physical examination. The
healthcare provider will about symptoms, risk factors for diabetes, past medical
problems, current medications, allergies to medications, family history of diabetes or
other medical problems such as high cholesterol or heart disease, and personal habits and
lifestyle.
A number of laboratory tests are available to confirm the diagnosis of diabetes.
Finger stick blood glucose: This is a rapid screening test that may be performed
anywhere, including community-based screening programs. Fingerstick blood glucose
values may be inaccurate at very high or very low levels, so this test is only a preliminary
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screening study. This is the way most people with diabetes monitor their blood sugar
levels at home.
Fasting plasma glucose: The patient will be asked to eat or drink nothing for 8 hours
before having blood drawn (usually first thing in the morning). If the blood glucose level
is greater than or equal to 126 mg/dL without eating anything, they probably have
diabetes. If fasting plasma glucose level is greater than 100 but less than 126 mg/dL, then
the patient has what is called impaired fasting glucose, or IFG. This is considered to be
pre-diabetes. The patient does not have diabetes, but they are at high risk of developing
diabetes in the near future.
Oral glucose tolerance test: This test involves drawing blood for a fasting plasma
glucose test, then drawing blood for a second test at two hours after drinking a very sweet
drink containing 75 grams of sugar. If the blood sugar level after the sugar drink is
greater than or equal to 200 mg/dL, the patient has diabetes. If the blood glucose level is
between 140 and 199, then the patient has impaired glucose tolerance (IGT), which is
also a pre-diabetic condition.
Glycosylated hemoglobin or hemoglobin A1c: This test is a measurement of how high
blood sugar levels have been over about the last 120 days (the average life-span of the red
blood cells on which the test is based).
HOW CAN THE BURDEN OF DIABETES BE REDUCED?
Without urgent action, diabetes-related deaths will increase by more than 50% in the next
10 years.
To help prevent type 2 diabetes and its complications, people should:
Achieve and maintain healthy body weight.
Be physically active - at least 30 minutes of regular, moderate-intensity activity
on most days. More activity is required for weight control.
Early diagnosis can be accomplished through relatively inexpensive blood testing.
Treatment of diabetes involves lowering blood glucose and the levels of other known risk
factors that damage to blood vessels. Tobacco cessation is also important to avoid
complications.
Interventions that are both cost saving and feasible in developing countries include:
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Moderate blood glucose control. People with type 1 diabetes require insulin;
people with type 2 diabetes can be treated with oral medication, but may also
require insulin;
Blood pressure control;
Foot care.
Other costs saving interventions include:
Screening for retinopathy (which causes blindness);
Blood lipid control (to regulate cholesterol levels);
Screening for early signs of diabetes-related kidney disease.
These measures should be supported by a healthy diet, regular physical activity,
maintaining a normal body weight and avoiding tobacco use.
HOW IS DIABETES TREATED?
When diabetes is detected, a doctor may prescribe changes in eating habits, weight
control and exercise programs, and even drugs to keep it in check. It's critical for people
with diabetes to have regular checkups. Work closely with your healthcare provider to
manage diabetes and control any other risk factors. For example, blood pressure for
people with diabetes and high blood pressure should be lower than 130/80 mm Hg
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PATIENT’S PROFILE
Name: F.L.
Age: 57 years old
Gender: Female
Civil Status: Married
Nationality: Filipino
Religion: Roman Catholic
Address: Tagbilaran City, Bohol
Date Admitted: January 21, 2013
Chief Complain: Loss of motor strength
Attending Physician: Dr. Belonguel
Final Diagnosis: Diabetes Mellitus Type II
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NURSING HISTORY
Past Health History
According to the patient she has completed the different immunizations such as
DPT, BCG, OPV, TT and measles. She had experienced childhood illnesses such as
chickenpox, measles, colds, fever and cough.
Family Health History
According to the patient, their family has history of heart disease, hypertension,
arthritis and diabetes. There are no other diseases related aside the mentioned above.
Present Health History
Eight (8) days prior to admission, the patient had experienced body malaise with
weakness. She then had loss of motor strength a day after. The following day, she was
brought to Holy Name University at Bohol and was admitted. On January 21, 2013, she
was referred to Perpetual Succour Hospital for further management.
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GORDON’S 11 FUNCTIONAL PATTERN
1. Health Perception/Health Management Pattern
Before Hospitalization
The patient stated that being healthy is important to be able to do her activities.
She also put emphasis on the idea that giving importance to health should be made right
from the start. She manages her health by taking a bath once a day and brushing her teeth
once or twice a day.
During Hospitalization
The patient verbalized that since she is being hospitalized, there is really
something wrong with her health. She can not take a bath, brush her teeth, and even comb
her hair without assistance.
2. Nutritional-Metabolic Pattern
Before hospitalization:
The SO verbalized that the patient eats three meals a day (breakfast, lunch and
dinner) with snacks in the morning and in afternoon. She eats whatever food is served.
The patient also added that whenever she feel something bad, she just eat 5 spoons per
meal. She drinks 7-8 glasses of water and another glass of juice a day. Her weight was 75
kg.
During hospitalization:
The patient has a diabetic diet and she eats three times a day. She eats with the help
and assistance of the SO. She also drinks about 1, 120 ml of water during our shift. Her
weight was decreased to 70 kg.
3. Elimination Pattern
Before Hospitalization:
According to the SO, the patient usually defecates once a day with a color of
brown, consistency is semi- solid. She regularly voids three times during day time and
during night, the client’s arenola (medium size) is full, its color is yellow amber without
difficulty in urinating and defecating.
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During Hospitalization
The patient urinates four times during our shift about 480 ml, has a color of dark
yellow and she did not defecate.
4. Activity- Exercise Pattern
Before Hospitalization:
The patient could not perform any exercise due to body weakness. According to
the SO, she can not do any heavy works and she needs assistance in walking or when she
go outside their house. She stays at home spending 5-6 hrs. of sitting and the rest of the
time the patient perform simple activities such as watching TV, eating, lying on bed and
chatting with her children and grandchildren.
During Hospitalization
The patient is under CBR without BRP’s so she stays on her bed the whole day.
5. Cognitive-Perceptual Pattern
Before Hospitalization:
The client’s senses are at work except for her eyes, she experiences blurring
vision. Her sense of hearing is not impaired. As to her sense of taste, she has no problem.
She is well oriented with time, place, persons and events.
During Hospitalization
The patient has no problems with regards to her speaking capabilities even though
she delivers her statement in a very soft voice. She answers questions relevantly. She is
well oriented with time, place, persons around her and event.
6. Sleep-Rest Pattern
Before Hospitalization
Her typical sleep hour is from 8pm to 8am, about 10-12 hrs. her usual nap time is
30 mins.after lunch.
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During Hospitalization:
The patient was not able to sleep at night because of the vital signs taking every 2
hrs. and drug administering.
7. Role-Relationship Pattern
Before Hospitalization
Being a responsible mother, she proudly says that she was able to fulfill her duties
and responsibilities. She has a very good relationship with her family. She claims that her
family is her priority. She emphasized that since all of them do their tasks and perform
their responsibilities well, they have a strong family ties. The moment she had her illness
and other signs of aging, she started to think she could no longer perform her duty
During Hospitalization
The SO verbalized that though her mother is suffering from DM and other illness,
the patient still have a close relationship with her family .
8. Self- Perception Pattern
Before hospitalization:
She views herself as a good mother and she added that she is strong woman who
can withstand the crisis she is currently facing in life. She believes that with the support
of her family, she could surpass whatever test of life comes her way.
During hospitalization:
She never changed her perception to herself. Though she felt being bed ridden
because she could not move that easily, with her family’s unending assistance, she gets
her strength from them not to give up no matter how painful her situation is.
9. Sexual- Reproductive Pattern
Before hospitalization:
The patient verbalized that she lives with her family in a compound. She has 4
children: 2 boys and 2 girls. She had her menarche when she was in grade 6. Her
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menstruation period lasts at about 3 or 5 days and it was regular (monthly). Sometimes,
she was suffering from dysmenorrhea. She had menopause.
During hospitalization:
Due to old age and her condition, she is no longer sexually active.
10. Coping- Stress Pattern
Before hospitalization:
She copes with stress by resting and sleeping. Whenever she thinks of her
condition, she prays to God to comfort herself.
During hospitalization:
The patient copes with stress by just merely talking to her daughter and she sleeps
when she feels drowsy. She also prays as her sign that she’ll never give up.
11. Value- Belief Pattern
The patient is a Roman Catholic. When the client was not yet suffering from DM,
they go to mass as a family but when she was diagnosed with this kind of disease and due
to aging; they could no longer attend mass; so they are just having their TV mass every
Sunday.
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LABORATORY RESULTS
Urinalysis – January 21, 2013
Microscopic Examination
BLOOD CHEMISTRY – January 21, 2013
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PARAMETERS RESULTS ANALYSIS
Color Yellow Normal
Transparency Turbid Due to increase urine concentration
Reaction 6 Normal
Specific Gravity 1.020 Normal
Sugar Trace Due to decrease renal threshold
Protein Trace Due to increase breakdowns of protein;
increased osmotic diuresis
PARAMETERS RESULTS ANALYSIS
Squamous Epithelial
Cells
Few Normal
Mucus Thread Few Due to infection as a result of increase
accumulation of sugar in the urine
Pus Cells 8-10 Due to infection as a result of increase
accumulation of sugar in the urine
RBC 3-4 Due to glomerular damage as a result of
renal strictures secondary to disease process
Armorp. Urates /
Phosphates
Few Normal
PARAMETERS NORMAL FINDINGSACTUAL
FINDINGSANALYSIS
Random blood sugar 70 – 120 md/dL 463 mg/dl Increased; due to
inability of the
body to utilize
glucose as a result
of impaired insulin
production
Creatinine F: 50-100 81.4 umol/L Normal
Sodium 135-155 mmol/L 131 mmol/L Decreased; due to
osmotic dieresis
Potassium 3.6-5.5 mmol/L 4.78mmol/L Normal
January 22, 2013
PARAMETERS NORMAL FINDINGSACTUAL
FINDINGANALYSIS
Random Blood Sugar 70 – 120 md/dL 487 mg/dl Increased; due to inability
of the body to utilize glucose
as a result of impaired
insulin production
Uric Acid 178-345 mmol/L 247 mmol/L Normal
Fasting Blood Sugar 3.89-5.84 mmol/dL 21.43 mmol/dL Increased; due to inability of
the body to utilize glucose as
a result of impaired insulin
production
Cholesterol 3.87- 6.71 mmol/dL 7 mmol/dL Increased; due to
metabolization of stored fats
as a result of free cholesterol
in the blood
HDL – C Female: >1.7 mmol/L 0.7 mmol/L Decreased; due to increased
dietary fat intake
LDL – C < 3.88 mmol/L 0.63 mmol/L Normal
Triglyceride Female: 0.46 – 1.6 1.50 Normal
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January 23, 2013
PARAMETERS NORMAL FINDINGS ACTUAL FINDINGS ANALYSIS
RANDOM BLOOD
SUGAR
70 – 120 md/dL 289 mg/dl Increased; due to
inability of the body
to utilize glucose as a
result of impaired
insulin production
SODIUM 135-155 mmol/L 140 mmol/L Normal
POTASSIUM 3.6-5.5 mmol/L 4.91mmol/L Normal
HEMATOLOGY – January 21, 2013
PARAMETERS NORMAL FINDING ACTUAL FINDING ANALYSIS
WBC 5 x 10 g/L 8.9 Normal
RBC: Hemoglobin
Hematocrit
Female: 12 – 16 g/dL
Female: 37 – 48%
11.4 g/dL
35%
Decreased; due to
inadequate oxygen
supply to cells as a
result of blood vessel
constriction
Decreased; due to
inadequate oxygen
supply to cells as a
result of blood vessel
constriction
Differential Count:
Segmenters
Lymphocytes
0.60 – 0.70
0.20 - .30
0.94
0.06
Increased; due to
inflammatory response
Decreased; due to
presence of infection
January 22, 2013
PARAMETERS NORMAL FINDING ACTUAL FINDING ANALYSIS
WBC 5 x 10 g/L 11.5 Increased; due to
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inflammatory response
RBC: Hemoglobin
Hematocrit
Female: 12 – 16 g/dL
Female: 37 – 48%
13 g/dL
40%
Normal
Normal
Differential Count:
Segmenters
Lymphocytes
0.60 – 0.70
0.20 - .30
0.90
0.10
Increased; due
inflammatory response
Decreased; due to
presence of infection
HEMOGLUCO TEST (HGT)
DATE TIMENORMAL
FINDING
ACTUAL
FINDINGANALYSIS
January 22,
2013
11:45 A.M.
1:00 P.M.
8:00 P.M.
80-120
80-120
80-120
408
333
307
Increased; due to increased secretion of
insulin
January 23,
2013
12:30 A.M.
2:30 A.M.
5:00 P.M
80-120
80-120
80-120
326
487
274
Increased; due to increased secretion of
insulin
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PATHOPHYSIOLOGY Precipitating: Predisposing
- Diet - Family history- Lifestyle - Age- Obesity
Number of insulin at receptor site
Insulin / zero insulin uptake in the cell
Metabolism Protein Metabolism Fat Metabolism
Lipolysis of adipose tissue Liver is to unable Glucose cannot CHON CHON to store glucose enter the cells of catabolism synthesis
muscle and adipose tissues ketone bodies hyperlipedimia
glycogenolysis fuel polyphagi amino acid -muscle weakness incomplete oxidation deposits lipid converted to -failure to grow in the lumen glucose
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A BC
D E
hyperglycemia lungs kidneys BP persistent BP
kidneys secreted acetone & CO2 acetone excretion bursting of small CVD excess glucose to retention capillaries in eyes restore balance S/SX: blurring vision
RR
dycosuria glucose in the urine acts as osmotic diuretic ketonuria Na retention osmotic pressure
urine output H2O loss
blood volumefluid volume deficit
C.O DEHYDRATION organ perfusion
SHOCK
severe tissue anorexia paradoxin of lactic acid
eyes (retinal neuropathy) peripheral neuropathy kidney neuropathy
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A B C D E
blindness tingling sensation poor ketones in the blood wound healing
ketosis
further in the nausea & vomiting H+; PH urine output
H2O loss metabolic acidosis H+ enters cellsdiabetic coma
polyuria K+ moves out of the cell going to the blood
dehydration
hypokalemia cardiacdysrythmias
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NURSING CARE PLAN
ASSESSMENT DIAGNOSIS PLANNING INTERVENTION RATIONALE EVALUATION
Objective Data:
-warm to touch
-flushed skin
- T= 37.7 o C
- RR= 34 cpm
Altered body
temperature:
Hyperthermia r/t
increased
production of
pyrogens in the
body
At the end of one
hour, the patient will
be able to obtain
body temperature
within normal range.
>Monitor vital signs & recorded
particularly temperature
>Provided the patient continues
TSB
>Positioned the patient in a
comfortable and safe position.
>Provided proper room
ventilation by opening the fan &
window panes.
>Advised the patient’s SO to
change the patient’s clothing to
loose & light colored clothes.
>To determine basal body
temperature, to have baseline
data & to have a basis for
evaluating the effectiveness of
interventions.
>To promote surface cooling
and heat lose by evaporation
and conduction.
> Promote client safety & to
maintain patent airway to
address increased respiratory
rate.
>To promote surface cooling
& heat loss by convection.
>Loose clothing & light
colored clothing promotes
body surface cooling. Light
colored clothes are more
absorbent to address
diaphoresis.
>To replace fluids &
Goal met. The
patient was able to
obtain core body
temperature within
normal range.
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>Advised the patient to increase
fluid intake to 8-10 glasses/day.
>Instructed the client to maintain
bed rest.
>Administered antipyretics & due
medications intravenously as
ordered.
electrolyte to support
circulatory volume & tissue
perfusion and to prevent
dehydration.
>To reduce metabolic
demands & oxygen
consumption.
>To restore normal body
temperature & to treat
underlying conditions.
ASSESSMENT DIAGNOSIS PLANNING INTERVENTION RATIONALE EVALUATION
Subjective Data:
“Masakit ang katawan ko
kapag ginagalaw ko”, as
verbalized by the patient
- pain scale of 8/10
Objective Data:
facial grimace
expressive
behavior
Pain r/t musculoskeletal
impairment
At the end of the shift,
the patient will be able
to:
> report pain if
relieved/ controlled and
a decreased in pain
scale of 0/10
> Determined level of
pain from 8/10
> Positioned patient
comfortably on bed
> Provided patient
adequate rest
> Provided quiet
environment
> to be able to know
what particular
intervention to relieve
pain
> To promote wellness
> to prevent fatigue
> to be able to have
enough rest
> to alleviate pain
caused by pressure on
Goal partially met.
The patient reported
pain controlled and a
decreased in pain scale
of 5/10
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restlessness > Provided comfort
measures such as
changing the patient’s
position frequently
> Administered
analgesia as ordered
nerve endings
> to maintain
acceptable level of pain
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ASSESSMENT
DIAGNOSIS PLANNING INTERVENTION RATIONALE EVALUATION
Subjective data:
“Madali akong mapagod
kahit na mag – iba lang ako
Activity
intolerance r/t
body weakness
At the end of the
shift , the patient
will be able to:
>Participate
> Monitored vital signs
every 2 hours
> to provide baseline data; to
evaluate the degree of
condition and the
effectiveness of the
Goal met.
At the end of the shift, the
patient was able to:
>Participate willingly in
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ASSESSMENT DIAGNOSIS PLANNING INTERVENTION RATIONALE EVALUATION
Subjective Data:
“Hindi ako mapakali
kasi sumasakit ang
katawan ko”, as
verbalized by the
patient.
Objective Data:
>Facial grimace
>Difficulty in turning
>Slowed movement
>Jerky movement
Impaired physical
mobility r/t
discomfort
At the end of the
shift, the patient
will be able to:
>verbalize
understanding of
situation and
individual
treatment regimen
and safety
measures
>increase strength
and function of
affected body part
> Determined diagnosis that
contribute to immobility such
as arthritis
> Assessed degree of pain by
listening to client’s
description and facial
expression
> Noted emotional or
behavioral responses to
problems of immobility
> Supported affected body
parts by using pillows
> to determine immediate
deviation or complication on
patient’s condition
> to provide necessary
intervention to lessen client’s
discomfort
> Feelings of
frustrations/powerlessness may
impede attainment of goals
> to maintain position of
function and reduce risk of
pressure on the affected area
Goal partially met.
At the end of the shift,
the patient was able to
verbalized
understanding of
situation and
individual treatment
regimen and safety
measures but did not
increase d the strength
and function of
affected body part
ng pososyon”, as verbalized
by the patient.
Objective Data:
BP: 110/70 mmHg
RR: 24 cpm
PR: 104 bpm
Body weakness
willingly in
necessary
activities
> report
measurable
increase in
activity
intolerance
> Encouraged
expression of feelings
contributing to the
condition
> Planned care with rest
periods between
activities
> Involved client and
SO in planning of
activities as much as
possible
intervention
> to determine the necessary
intervention to be made for
the client’s condition
> to reduce fatigue and to
have enough rest periods.
> involvement of client and
SO during the plan of care
helps to attain goals
necessary activities
> report measurable
increase in activity
intolerance
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ASSESSMENT DIAGNOSIS PLANNING INTERVENTION RATIONALE EVALUATION
Subjective data:
“Paputol –putol ang
tulog ko dahil sa
pagbibigay ng gamut ko
at pagmonitor sa akin”,
as verbalized by the
patient.
Objective data:
dark circles
under eyes
frequent
yawning
restlessness
Sleep pastern
disturbance r/t
interruptions for
therapeutics and
monitoring
At the end of the shift,
the patient will be able
to:
verbalize
understanding of
sleep disturbance
report improvement
in sleep or rest
pattern
report increased
sense of feeling
rested
>Obtained feedback from
Client and SO regarding usual
bedtime, number of sleep
hours, and time of arising
>Explained to the patient and
SO the importance of taking
VS and administering
medicines.
>Arranged care to provide for
uninterrupted periods for rest
>Provided quiet environment
and comfort measures
(arranging the bed)
> to determine usual
sleep pattern and provide
comparative baseline
>to gain cooperation
from the patient
>to provide adequate
sleep and rest periods
> to provide comfort to
the patient and have
adequate sleep
Goal met.
At the end of the shift,
the patient was able to:
> verbalize
understanding of sleep
disturbance
>report improvement in
sleep or rest pattern
report increased sense
of feeling rested
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DRUG STUDY
1. Generic Name: Colchicine
Brand Name: Colsalide, Novocolchine
Classifications: Antigout
Action: Inhibits microtubule formation of lactic acid in leukocytes which decreases
phagocytosis and inflammation in joints.
Uses: Prevention or treatment for gout, gouty arthritis to arrest progression of neurologic
disability in multiple sclerosis
Contraindications: Hypersensitivity; serious GI, renal, hepatic, cardiac disorders; blood
dyscariasis
Side Effects:
Miscellaneous: myopathy, alopecia, reversible azoospermia, peripheral neuritis
GU: Hematuria, Oliguria, Renal Damage
HEMA: Anemia, thrombocytopenia
GI: nausea, vomiting, anorexia, malaise, metallic taste, cramps, peptic ulcer,
diarrhea
INTEGU: cdhills, dermatitis, pruritis, purpura, erythema
Route and Dosage: 1 tab every 1 hour
Nursing Implications:
Assessment: > Assess I and O ratio, observe for decrease in urinary output.
> Administer on empty stomach, only to facilitate absorption
Family / Client Teaching: > Instruct SO or family members to increase fluids to
3-4 L/day.
> Encourage patient or SO to report any pain, redness
hand area.
2. Generic Name: Omeprazole
Brand Name: Prilosec
Classifications: Proton Pump Inhibitor
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Action: Thought to be a gastric pump inhibitor in that it blocks the final step of acid
production by inhibiting the hydrogen, potassium, ATP system at the secretory surface of
the gastric parietal cell.
Uses: Used to treat ulcers, gastroesophageal reflux disease or heartburn
Contraindications: pain with swallowing; had heartburn for 3 months; frequent chest
pain
Side Effects: CNS: headache, dizziness, insomia, apathy
GI: diarrhea, constipation, anorexia
CV: chest pain, bradycardia, palpitation
Respi: URI, cough, bronchospasm
Derm: rash, severe generalized skin reaction including toxic epidermal
necrolysis
GU: UTI, urinary frequency, hematuria, glycosuria
Hema: thrombocytopenia, anemia
Musculo: Back Pain, Muscle Cramps and Weakness
Misc: fever, pain, gout, fatigue, malaise, weight gain
Route and Dosage: 40 mg ANST ( - )
Nursing Implications:
Assessment: > Perform skin test
> Check for vital signs
> Monitor for any episodes of chest pain
Family / Client Teaching: > The capsule should be taken 30 minutes before
eating and is to be swallowed whole; it
should not be opened, chewed or crushed.
> Review drug associated side effects; report if
diarrhea persists. Report any changes in
urinary elimination or pain and discomfort.
3. Generic Name: Vigocid (Piperacillin Sodium + Tazobactam Sodium)
Action: Bactericidal in action by inhibiting septum formulation and cell wall synthesis of
susceptible bacteria
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Use: used as antibiotic
Contraindications: Patients with a history of allergic reactions to any of the penicillins,
cephalosporins, beta-lactamase inhibitors
Side Effects: nausea nad vomiting, diarrhea, constipation, rash, red skin, allergic reaction
(hives), difficulty in sleeping, headache, diaphoresis, eczema
Route and Dosage: 4.5 g / IV every 8 hours ANST ( - )
Nursing Implications:
Assessment: > Document indications for therapy, symptom onset and weight
history
> Assess for other medical conditions that require careful
monitoring
Family / Client Teaching: > Take drug within ordered intervals to prevent
further aggravation
> Do not engage in activities that require mental
alertness.
4. Generic Name: Meloxicam
Brand Name: Mobic
Classifications: NSAID
Action: Reduces the production of prostaglandin that initiate the cause of inflammation
Uses: used to treat inflammation and pain of arthritis
Contraindications: with asthma attacks, hives or allergic reactions
Side Effects: GI: abdominal pain, diarrhea, dyspepsia, constipation, flatulence
CNS: dizziness, headache,
Route and Dosage: 15 mg / tab; 1 tab OD
Nursing Implications:
Assessment: > Perform skin test
> Document indications for therapy, onset and characteristics of
disease, ROM.
> Determine any GI bleed or ulcer history, aspirin or other
NSAID-induced asthma, urticaria or allergic type reactions.
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Family / Client Teaching: > Take exactly as directed and at the same time each
day. May take with or without food.
> Avoid activities that require mental alertness until
drug effects realized; may cause dizziness or
drowsiness.
> Report any unusual or persistent side effects
including dyspepsia, abdominal pain, dizziness and
changes in stool or skin color.
5. Generic Name:Metformin HCl
Brand Name: Glocophage
Classifications: Antidiabetic
Action: Decreases hepatic glucose production, decreases intestinal absorption of glucose
and increases peripheral uptake and utilization of glucose.
Uses: Alone as an adjunct to diet to lower blood glucose in client having NIDDM whose
blood glucose cannot be managed satisfactorily via diet alone.
Contraindications: acute or chronic metabolic acidosis, including diabetic ketoacidosis,
with or without coma. Abnormal hepatic function.
Side Effects: GI: Diarrhea, abdominal bloating, flatulence, anorexia, unpleasant or
metallic state, abnormal stools, taste disorders
CNS: lightheadedness, headache
Misc: hypoglycemia, myalgia, chest discomfort, palpitation
Route and Dosage: 500 mg / tab (tab BID during meals)
Nursing Implications:
Assessment: > Individualize dosage based on tolerance and effectiveness
> Give with meals starting at a low dose with gradual escalation.
This will reduce GI side effects and allow determination of the
minimal dose necessary for adequate control of blood glucose.
> Document age at diabetes onset, previous therapies utilized and
outcome.
Family / Client Teaching: > Take with food to decrease GI upset
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> Warn users that the inactive components in the
extended-release tablets may pass into the feces and
appear as a soft, hydrated mass.
6. Generic Name: Humulin - N
Brand Name: Isophane Insulin Human
Action: intermediate-acting insulin with slower onset of action that keep blood glucose at
a nearly normal level
Uses: indicated for treatment of patient with diabetes mellitus who require insulin for the
maintenance of glucose homeostasis
Contraindications: contraindicated during episodes of hypoglycemia; also patient with
hypersensitivity to human insulin
Route and Dosage: OD every 9 pm
Nursing Implications:
Assessment: > Monitor VS particularly BP
> Be alert for signs of hypoglycemia, loss of glucose control,
kidney, eye or foot problems
Family / Client Teaching: > Do not take if without medical advise.
7. Generic Name: Paracetamol
Brand Name: Acephen, Campain
Classifications: Antipyretic
Action: Reduces fever by direct action on the hypothalamus, heat-regulating center with
consequent vasodilatation and sweating.
Uses: Fever reduction; temporary relief of mild to moderate pain. Generally, as substitute
for aspirin when it is not tolerated.
Contraindications: Renal insufficiency, anemia, cardiac or pulmonary disease
Side Effects: rash, anorexia, nausea and vomiting, diaphoresis, abdominal pain
Route and Dosage: 500 mg, 1 tab PRN for fever
Nursing Implications:
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Assessment: > Do not exceed a dose of 4 g/ 24hr in adults and 75 mg/kg/day in
children.
> Do not take more than 5 days for pain in children, 10 days for
pain in adults, or more 3 days for fever in adults or children
without consulting Health care provider.
Family / Client Teaching:> Warn not to combine products containing
acetaminophen.
> Take only as directed and with food or milk to
decrease GI upset
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