syncope vonny

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    Vonny F. Goenawan

    Siloam Hospital Lippo Karawaci

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    Transient loss of consciousness with aninability to maintain postural tone followedby spontaneous recovery

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    Transient disruption of cerebral perfusionthat results from decrease of cardiac output,profound vasodilation, or both

    Syncope occurs due to

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    Accounts for 3 of ERvisits

    Accountsfor 1-6hospital admission

    Increase incidencewith advancing age

    Commonproblem

    Some causes of syncopeare potentially fatal

    sincope

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    Syncope must be differentiated from othernon syncopal conditions which cause atransient loss of consciousness

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    Cardiac arrthymias as a primary cause Bradyarrhythmias

    Sinus node disease

    AV nodal disease

    Pacemaker dysfunction

    Drug induced

    Tachyarrhythmias

    VT, torsade de pointes

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    Structural cardiac or cardiopulmonarydisease Valvular heart disease

    Acute MI or ischemia Pericardial disease/ tamponade

    Pulmonary embolus/pulmonary hypertension

    Obstuctive cardiomyopathy

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    Neurally mediated reflex syncope (NM)1. Carotid sinus syncope ( head turning,

    shaving)

    2. Situational faint Coughing/sneezing

    GI stimulation defecating, swallowing

    Micturation

    3. Vasocagal *common faint* Also called neurocardiogenic

    Often situational

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    -Upon positional change neurohormonal eventsmaintain cerebral perfusion-normally decreased venous return and subsequent

    decreased left ventricular filling increase sympathetic tone-- overly sensitive left ventricular response misinterprethypercontractility as volume overload inhibit sympatheticstimulation hypotension, bradychardia, syncope

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    Increased pressure in carotid sinusparasympathetic stimulation syncope

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    Orthostatic Automonic failure

    Primary autonomic dysfunction Pure autonomic failure rkinsons

    Secondary autonomic dysfunction Diabetic neuropathy Drugs

    Volume loss Internal bleeding, diarrhea

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    Cerebrovascular Almost never the cause of true fainting Vascular steal syndromes

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    History of syncope after head turning,shaving or while wearing a tight collar, olderpatients with unexplained presyncope or falls,negative cardiovascular and neurologic

    investigations. With patient supine massage each carotid 5-

    10 secs while monitoring BP and HR

    Positive response is asystole of 3 seconds or

    drop in systolic BP of 50 mmHg Non spesific 25% of nonsyncopal elderly

    patients will have positive response

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    Tilt patient passive (60 degrees, 45 minutes)in absence of pharmacologic provocation

    Administer Isoprotenol, nitroglycerin, tiltagain for 10 minute

    Positive resultsreproduction of patients

    typical syncopal symptoms with hypotension,bradycardia or both

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