natural endocrine

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Table of ContentsThe Endocrine System 5The Endocrine System: Hypothalamus, Pituitary, & Pineal Glands 8

Hypothalamus 12Pituitary gland 13Posterior pituitary gland 1Pineal Gland 18!ircadian rhythm and the Pineal 22History 23!riteria 23"rigin 2#$mportance in animals 2%$n plants 2'iological cloc( in mammals 2

)ight and the *iological cloc( 2+Enorced longer cycles 2+Human health 3-!onclusion 3#

The Endocrine System: The Pancreas &Diabetes 39The pancreas unctions in t.o modes/ 3+ 0natomy reie. 3+Physiology o the endocrine pancreas our cell types #-ia*etes mellitus 45s.eet urine56 #3

$nherited 7orms o ia*etes ellitus #9atural treatments or dia*etes #

The Endocrine System: Thyroid andParathyroid Glands 49Thyroid oerie. #+Thyroid hormones %1Thyroid hormone unctions %3$odine upta(e and control %3Thyroid dysunction %#9atural Hashimotos Treatment ;our $odine Supplements ay 'e a(ing

;ou


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ndocrine glands, on the other hand, release more than /0 ma1or hormonesdirectly into thebloodstream where they can be transorted to cells all over the body to havecollective actions at target cells elsewhere. The ma1or glands that make u the human endocrine system include the&

2 hyothalamus2 ituitary gland2 thyroid2 arathyroids2 adrenal glands2 ineal body2 reroductive glands %which include the ovaries and testes)2 there are endocrine functions of almost all organs to a minor degree, thegall bladder releaseCC3 %Cholecystokinin %CC3 or CC3'6 from 7reek chole, 8bile86 cysto, 8sac86kinin, 8move86

hence, move the bile'sac %gallbladder)) is a etide hormone of thegastrointestinal systemresonsible for stimulating the digestion of fat androtein. Cholecystokinin, reviously called ancreo(ymin, is synthesised by I'cells in the mucosal eithelium of the small intestine and secreted in theduodenum, the rst segment of the small intestine, and causes the releaseof digestive en(ymes and bile from the ancreas and gallbladder,resectively. It also acts as ahunger suressant. 9ecent evidence has suggested that it also lays ama1or role in inducingThe cost o# this boo( is 4- E$ro) The co'yright is o.ned by /0 1td) all rights reser*ed)lease ay to the ay al under deisfm.net donations. All of the books have low res for si(e and hi resversions for clarity.

If you do not have the money ay what you can, and if you cannot ay anything lease ay thecosmos back with good deeds toothers. ass on the good karma by being good to others and heling them the way I am heling youass it on.

: ; drug tolerance to oioids like morhine and heroin, and is artlyimlicated in e!eriences of ain hyersensitivity during oioid withdrawal. There are many such e!amles but this bookis 1ust about the rimary endocrine functions.< =

The Endocrine System: 2y'othalam$sPit$itary & Pineal Glands Today, we begin our e!loration of the endocrine system. In many ways, theendocrine systemcan be viewed as a artner, or comlement, to the nervous system. >hereasthe nervous system uses nerve imulses that last milliseconds to controlshort term events in the body, the endocrine system uses hormones that cansometimes take minutes, hours, or even days to take e+ect and controlevents. And sometimes those e+ects can last a lifetime.


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?nce you understand how imortant the endocrine system is in controllingevery asect of yourlife, from your moods to your se!uality to your energy levels to your ability togrow and be strong, you reali(e how imortant it is to kee it otimi(ed. Andyes, there are things you can do to kee it otimi(ed.

"ntrod$ction The endocrine system is comrised of a grou of ductless glands that secretehormones directlyinto the saces surrounding their cells. "rom there, the bloodstream icksthem u and circulates them throughout the body '' ultimately reaching theorgan or cells designed to resond to a articular hormone. It is the ductlessnature of the glands that denes them as art of the endocrine system. Asfor hormones, they are the body$s chemical messengers that tell the bodywhat to do@ and when. ormones roduced by the endocrine system arenecessary for normal growth and develoment, reroduction, andmaintaining bodily functions %homeostasis). In humans, the ma1or endocrine

glands are the hyothalamus, ituitary, ineal, thyroid, arathyroids,adrenals, the islets of Bangerhans in the ancreas, the ovaries, and thetestes.-ecretion of hormones in the endocrine system is controlled either byregulators in a articulargland that detect high or low levels of a biochemical and inhibit or stimulatesecretion or by acomle! mechanism involving the brain, the hyothalamus, and the ituitary.It should be noted again that the nervous system and the endocrine systemare comlementary'' both in terms of form and function. #oth systems share a rimary function

of coordinating the activities of the body$s many systems. "or e!amle, thenervous system tells muscles when to contract and rela!, whereas adrenalintells the body how to resond to stress or threats. The rimary di+erence isthat nerve imulses e!ecute their e+ect in milliseconds@and the e+ects tendto be short'lived. The endocrine system, on the other hand, takessubstantially longer for hormones to wend their way from the gland thatroduces them, through the bloodstream, and ultimately to the organ or cellswhere they take e+ect. In addition, the actions of hormones are much longerlasting than the milliseconds of nerve imulses. Another way of utting thisis to say that the nervous system directs the body$s short term resonses,whereas the endocrine system directs the body$s longer term resonses.

?ne other oint of note is that both systems are mutually interconnected. "ore!amle, whenthe nervous system needs to control things longer term, it acts through theendocrine system by stimulating the release or inhibition of hormonesthemselves from the endocrine organs. ?n the other hand, adrenalin,released by the adrenal glands, acts uon the brain to stimulate the ght oright resonse.


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General denitions#efore we continue, we need to lock down some imortant denitions.2 As mentioned above, endocrine glands %endo 8within8) are glands thatsecrete directly intothe saces around the cells and whose roducts are icked u and circulated

by the bloodstream.2 In contrast to the endocrine glands are the e!ocrine glands %e!o 8out8,krinein 8to secrete8).Dnlike endocrine glands, e!ocrine glands secrete into ducts, which in turn,carry thesecretions out of the glands and into the lumens %the inner cavities of atubular organ suchas blood vessels and the intestinal tract) or other body cavities or even outof the body. #yan overwhelming ma1ority, most glands in the body are e!ocrine glands, andmost e!ocrine

glands secrete their 8roducts8 outside of the body. These include sweat, oil,and mammaryglands. %>e will not be discussing the e!ocrine glands in this articular seriesof newsletters.)2 The endocrine system includes some organs that are wholly endocrine infunction such as theituitary gland, thyroid gland, arathyroid gland, adrenal glands, and inealgland. %It is theseglands in articular, along with the ancreas, that will be the focus of thisseries of newsletters.)2 ndocrine organs that have other functions as well as endocrine functions

include the ancreas, liver, ovaries, stomach, hyothalamus of the brain,small intestine, kidneys, testes, and lacenta. These are comoundglandsEorgans. %*ost of these will be covered when we e!lore their otherfunctions.)Endocrine gland locations2 The hyothalamus, ituitary gland, and ineal gland are located in thebrain.2 The thyroid gland is located in the neck, with the four arathyroid glandssituated behind it.F0 FF2 The thymus is in the chest %will be covered when we discuss the immunesystem).2 The adrenal %A3A the suraneal) glands lie on to of the kidneys.2 The ancreas, stomach, ovaries, and testes are located in and beneath theabdominal cavityand have multile functions '' some of which include endocrine functions.2ormones


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As we mentioned earlier, the endocrine system releases chemicalmessengers called hormones%hormone 8urge on8), which act on other organs in di+erent arts of thebody. +ectively, hormones are the body$s chemical messenger system ''they tell the body what to do and when. -ome hormones romote or inhibit

nerve imulses, while others %einehrine and noreinehrine, for e!amle)may act as neurotransmitters themselves in certain arts of the body. Thenagain, these hormones act as hormones %rather than as neurotransmitters) inother laces. %This will be much easier to understand when we e!lore theadrenal glands in a subseGuent newsletter.)Also, as we mentioned earlier, hormones may take seconds, minutes, orhours to work their e+ects, and their duration of action may be short' orlong'lived. ow longHConsider that once estrogen tells a fetus to become a girl, the e+ect lasts anentire lifetime unless a doctor intervenes at some oint. In general, though,hormones regulate growth, develoment, reroduction, metabolism, mood,

and tissue function.General 'ro'erties o# hormonesAlthough they may reach all the cells of the body via the bloodstream, eachof the 50J hormones in the human body a+ects only a tiny handful of verysecic cells. This selectivity is key to the functioning of the endocrinesystem. ow is it accomlishedH2 Target cells contain highly secic recetors, which are surfaceglycoroteins %roteins whichinclude a carbohydrate and a simle rotein).2 The geometry of the glycorotein molecules allows only for very secichormones to attach

to the recetor in the target cell surface. Think of it as a lock and keymechanism. !cetionsinclude&2 Chemical mimics such as !enoestrogens %etroleum'based hormonelookalikes) and syntheticgrowth hormones in meat, etc. '' which can be otent in amounts as small asa billionth of agram. These are never good.2 lant mimics such as hytoestrogens consumed in the diet or insulements, which can ll recetorsites, making them unavailable to the stronger natural hormones %or

chemical mimicsfor that matter) in the human body. This e+ect can often be used toadvantage to tone downoverly strong hormonal resonses in the human body.ach target cell has u to F00,000 recetors for a given hormone. >henthere is an e!cess of thathormone, the number of recetors decreases, reducing sensitivity. Thisreduction of sensitivity is


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known as 8down regulation.8 Also, as 1ust e!lained, chemical and hytomimics can ll recetorsites on a cell making those sites unavailable to the actual hormones '' thusdown regulating thecell. ?r in the case of some chemical mimics, u regulating them. %Kote& cells

contain recetorsfor multile hormones, not to mention neuroetides roduced by the brain,and other kinds of recetors too. Thus a single cell may actually have millions of recetor siteson its surface.)If an abnormally low number of hormone molecules is circulating, thenumber of recetor siteson individual cells will increase to raise the level of sensitivity and thuscomensate. This is knownas 8u regulation.81ocally acting hormones:

These hormones do not enter the general circulation. There are two tyes ''one of which, in articular, is of secial concern to us.2 aracrine hormones %ara 8near8) act on cells ne!t to the secreting cellswithout enteringthe bloodstream '' 1ust assing through the interstitial uid between cells.2 Autocrine hormones %auto 8self8) act on the cell that secreted them. These can lay a criticalrole in terms of our health. Cancer cells use autocrine signaling to triggergrowth. This meansthat cancer cells are autonomous. They don$t take orders from other cells inthe body. They

tell themselves what to do. That$s one of their features that makes them sodangerous.Kow that we have a basic understanding of what the endocrine system is,what it does, and how it works, let$s start making our way down through thebody and begin by taking a look at the three endocrine glands in the humanbrain& the hyothalamus, the ituitary, and the ineal glands.F/ FL

2y'othalam$s The hyothalamus is located below the thalamus and osterior to the oticchiasm. In humans,

the hyothalamus is roughly the si(e of an almond. #ut within that smallsi(e, it contains a number of small nuclei with a variety of functions. ?ne ofthe most imortant functions of the hyothalamus is to link the nervoussystem to the endocrine system via the ituitary gland. The hyothalamusactually controls the ituitary gland6 and it integrates many messages fromarts of the brain based on feedback from all over the body and tells theituitary what to do.


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Communication between the hyothalamus and the ituitary is e+ectedthrough a ortal bloodcaillary system, which connects the two glands over a very short distance. This rovides a direct venous to venous connection. The advantage of thistye of direct connection is that a ortal ow allows blood'borne molecules

from the hyothalamus to act on the ituitary before they are diluted withthe blood in larger vessels, thus it takes very, very few molecules to directthe ituitary. The hyothalamus synthesi(es and secretes neurohormones, often calledhyothalamic'releasing hormones, and these in turn stimulate or inhibit thesecretion of ituitary hormones. Among other things, the hyothalamus,through its action on the ituitary, controls body temerature, hunger, thirst,fatigue, childbirth, emotions, growth, milk roduction, salt and waterbalance, slee, weight, and circadian cycles. It is resonsive to light %thelength of the day for regulating both daily circadian and seasonal rhythms). Itis also resonsive to olfactory stimuli %including heromones), steroids,

neurally transmitted information %from the heart, stomach, and reroductivesystem, stress, changes in body temerature caused by infection, and blood'borne stimuli %including letin and ghrelin %aetite regulating hormones),angiotensin, insulin, ituitary hormones, cytokines, and glucose, etc.)"or the most art, the hyothalamus functions retty much roblem free forthe vast ma1ority of eole. owever, any of the following can cause it tomalfunction& anore!ia, bulimia, malnutrition, too much iron, bleeding, headtraumas, infections, inammation, genetic disorders, tumors,radiation, andsurgery.

Pit$itary glandAt one time, the ituitary gland, also called the hyohysis, was oncethought to be the 8mastergland8 that controlled all the other endocrine glands. #ut, as mentionedabove, we have sincelearned that the hyothalamus actually controls the ituitary gland6 and itintegrates many messages from arts of the brain based on feedback fromall over the body and tells the ituitary what to do. In any case, the twoglands are tightly integrated. Together, they regulate all rocesses having todo with rimitive reactions, such as stress, rage, ight, body temerature,thirst, hunger, se!ual activity, and survival in general. And between them,they secrete F: hormones. The ituitary is about F cm in diameter, and it lies in the sella turcica%8Turkish saddle8) at the baseof the brain, directly behind the otic chiasm. It is divided into twoembryologically and functionallydi+erent arts& the anterior ituitary and the osterior ituitary.mbryologically refers towhat tissue the gland develoed 8out of8 starting as an embryo. The anteriorituitary evolved


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anatomically u from the oor of the mouth. The osterior ituitary, on theother hand, evolveddownward from the base of the brain. In fact, the two arts of the ituitarydon$t even talk to eachother.

nterior 'it$itary The anterior ituitary gland is also called the adenohyohysis, and it makesu ;5M of the ituitarygland '' the remaining /5M belonging to the osterior ituitary. -evenreleasing hormones%including growth'hormone'releasing hormone and growth'hormone'inhibiting hormone) aresecreted by the hyothalamus and are resonsible for the release orinhibition of the anteriorituitary hormones. They are generally controlled by negative feedbackmechanisms.

?nce triggered by the hyothalamus, hormones released by the anteriorituitary ow into theF4 F5general circulation for action in far arts of the body. Bike the hyothalamus,anterior ituitaryhormones are also controlled by negative feedback from the brain and thetarget organ. Thatis, when the target organ resonds to the activating hormone from theituitary, it will releaseits own hormone back into the blood, which will travel back to the brainthrough the circulatory

system, which in turn triggers the hyothalamus to turn o+ roduction of thestimulating hormone in the anterior ituitary. "or e!amle, the ituitarystimulates the thyroid to release thyroid hormones, which travel throughoutthe bloodstream stimulating metabolism in select arts of the body asreGuired. Through the negative feedback loo, the brain learns that themetabolism has been activated enough %in other words, that enough thyroidhormones have been released) and tells the hyothalamusEituitary to stostimulating the thyroid. This comletes the negative feedback loo.Princi'al anterior 'it$itary hormones2 Thyroid'stimulating hormone %T-) stimulates the thyroid gland to releasethyroid hormones,which tend to uregulate metabolism.2 "ollicle'stimulating hormone %"-) and luteini(ing hormone %B) togetherstimulate the release of estrogen and rogesterone, which cause thematuration of ova in the female andserm cells in the male, as well as the release of testosterone.2 rolactin %B) stimulates the roduction of milk by the breasts. As a sidenote, rolactin can


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cross the lacenta'blood barrier, causing 8witch$s milk,8 or milk roductionfrom a baby$s niles.2 Adrenocorticotroic hormone %ACT) stimulates the release of adrenalcortical hormones bythe adrenal glands.

2 *elanocyte'stimulating hormone %*-) causes increased skinigmentation.2 uman growth hormone %h7, or somatotroin) stimulates body growthand regulates metabolicrocesses. igh h7 increases the growth of the skeleton during childhood,and it maintainsmuscle and skeletal si(e in the adult. -ince h7 is robably the best knownhormone roducedby the ituitary '' and in the news constantly because of its illegal useamong amateurand rofessional athletes looking for a cometitive edge '' let$s take a look at

this articularhormone in a little more detail.2$man Gro.th 2ormone The re1uvenating owers of growth hormone %7) are no secret to thewealthy and rofessional athletes& for the last L0'40 years, 7 has beenavailable from doctors, reGuires two in1ections a day, and costs u to NF,ould that it were soO Although the e+ects are more

subtle for most eole, they are nevertheless wide ranging&2 "at loss %F4 ercent on average after si! months, without dieting)2 limination of cellulite2 igher energy levels and enhanced se!ual erformance2 9egrowth of heart, liver, sleen, kidneys, and other organs that shrink with2 age2 7reater heart outut and lowered blood ressure2 Imroved cholesterol role, with higher PB %8good8) cholesterol and2 lower BPB %8bad8) cholesterol2 -uerior immune function2 Increased e!ercise and athletic erformance

2 #etter kidney function2 -tronger bones2 "aster wound healing and recovery from in1ury2 Qounger, tighter skin2 air regrowth The most imortant function of 7, however, is telling the liver to roduceinsulin'like growth factor F %I7"'F), the main key to anti'aging. -ecically,the benets of 7 can be measured in terms of how much it increases the


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body$s roduction of I7"'F %above a /0 ercent increase starts to besignicant in terms of e+ectiveness). There is some concern that, because it increases I7"'F levels in the body, 7may increase therisk of rostate cancer. A simle reality check, however, calls these

observations into Guestion."irst, both 7 and I7"'F levels decline as we age, yet the incidence ofrostate cancer increasesas these levels decline '' the e!act oosite of the e!ressed concern. Inaddition, in numerousstudies involving thousands of atients receiving growth hormone over manyyears, there wereno observed increases in rostate cancer. In fact, based on real'lifeobservation, there is evidence that growth hormone sulementation mayreduce the risk of rostate cancer.F: F;

-ulementing with 7rowth ormone*ost sulement formulas will increase I7"'F levels by a minimum of /0ercent, with some even aroaching F00 ercent. #ut kee in mind that 1ustone L0'minute aerobic session can easily increase I7"'F levels by F00ercent, and a solid session of weight training can increase levels byan incredible 400''


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meant it could only be administered by in1ection, which reGuired a doctor andwas very e!ensive.#ecause of the cost, growth hormone in1ections became known as the secretyouth formula of movie stars, athletes, and the very rich.

"or most eole, then, the best alternative to 7 in1ections is the use ofamino acid'based recursor formulas %also called a 7 secretagogues). Tyically, these formulas contain ingredients such as glutamine, tyrosine,7A#A, arginine, and lysine. Although not as owerful as growth hormonein1ections, these formulas can be Guite e+ective, rovided your ituitary isfunctioning well, and they carry none of the downside of in1ections. Things that sometimes go wrong with the anterior ituitary gland Kotsurrisingly, since the ituitary is so involved with regulating growth, some of the key roblemsassociated with a malfunctioning ituitary are related to growth. Theseinclude&

2 ituitary dwarsm& Bow levels of h7 during the growth years causes bone'growth'late closure before normal si(e is achieved. Also, many organs aresmaller than normal, and the erson has a childlike stature. "ortunately,in1ections of synthetic h7 roduced by recombinant PKA technology inbacteria can revent this if diagnosed in time.2 ituitary giantism& yosecretion of h7 during childhood causes longbones and tall staturebut otherwise normal roortions.2 Acromegaly& Dsually caused by functioning ituitary tumors in the alreadynormal adult, itcauses thickening of bones of the face, hands, and feet %bones can get

longer after the closureof growth centers) and thickening of the tongue, eyelids, and nose. Andre the7iant, thewell known wrestler and actor, was one of the world$s best known e!amlesand was, in fact,often billed as The ighth >onder of the >orld. Another famous %ossiblee!amle) mighthave been the biblical 7oliath, who was slain by Pavid with a slingshot. Infact, there is a veryinteresting theory, with real scientic basis, that rooses that 7oliath$sacromegaly might

actually account for how he was slain by Pavid. According to the theory, aituitary tumor,because of the ituitary$s lacement right behind the otic chiasm, cansometimes lace ressure on eriheral vision nerve bers, resulting intunnel vision. If 7oliath had that condition, which sometimes does occur withacromegaly, he would have been blind to Pavid, if Pavid aroached fromthe side, and a rock hurled from the side would hit the temle at the thinnestart of the skull, thus stunning the giant. ?nce stunned and on the ground,


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Pavid would then be able to safely aroach his now helless victim and cuto+ his head. And thus the legend was born '' or so the theory goes.

Posterior 'it$itary glandAs I mentioned earlier, the osterior ituitary gland %A3A theneurohyohysis) is anatomically

derived from a down growth of the brain and is not technically a gland sinceit does not synthesi(e F< F= hormones, but rather, stores and secretes twohormones actually made in the brain. These twohormones, o!ytocin and vasoressin, are transorted from the brain in smallackets for storagein the osterior ituitary '' to be released as needed.2 ?!ytocin %o!ytocia 8raid child birth86 A3A itocin) enhances the strengthof uterine contractionsand stimulates the e1ection of milk after delivery. It may also foster maternalinstinctsand se!ual leasure during and after intercourse. Kow synthesi(ed andreadily available, it isoften given to women to hel them have stronger contractions and e!el thefetus in a moretimely manner@when necessary.2 Rasoressin %Antidiuretic hormone, AP) decreases urine roduction byincreasing re'absortion by the kidneys, a useful trick when su+ering fromdehydration. The e+ect, though, is to raise blood volume and, therefore, toraise blood ressure. Alcohol inhibits AP secretion,thus roducing rofuse urination after a drinking binge, which leads tosevere dehydration,and the severe dehydration leads to the headache and thirst associated witha hangover.

Pineal Gland The ineal gland is about the si(e of a grain of rice, is shaed like a tiny inecone %hence its name), and is located in the center of the brain in a tinycave, behind and above the ituitary gland. "or years, mystics considered itto be the seat of the mystical third eye, whereas the medical communityconsidered it vestigial and, thus, retty much non'functioning. -ince then,the mystics have not necessarily been refuted, but the medical communityhas been. The ineal gland is now known to be the ma1or source of melatoninroduction in the body. It is full si(e in children, a si(e it maintains throughout

adulthood '' although its weight can dro signicantly starting with uberty.And it is not unusual for the gland to literally calcify in many adults. Thegland most likelylays a signicant role in se!ual maturation, circadian rhythm and inducingslee, and in seasonal a+ective disorder and deression. In animals, it laysa key role in hibernation.%elatonin


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The trigger for roduction and release of melatonin is total darkness '' anylight in the room willinhibit this rocess. Today, however, living in a world with nightlights in thebedroom or streetlights sneaking through the window, we actually have aneidemic of eole with insuScient melatonin roduction, even at a very

young age. The roblem doesn$t 1ust come from light falling on our eyeswhile we slee, but from light falling on any art of the body. ven if youwear an eye'mask, if any light is falling on your arms or chest or feet, that$senough to slow melatonin roduction. >ithout articial light, we wouldnormally be in total darkness


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2 *ood levator '' Kighttime melatonin levels are low in eole with ma1orderessive andanic disorders. Individuals with mood swings or who are melancholic alsohave lower melatonin levels. #oth seasonal a+ective disorder %-AP) andcyclic deressions are related to the eaks and valleys of melatonin levels.

Third eye>hile the hysiological function of the ineal gland remained unknown untilrecently, mysticaltraditions and esoteric schools, have long considered the ineal gland to bethe connecting linkbetween the hysical and siritual worlds@and the seat of e!trasensoryercetion. I am not here to argue the siritual Gualities of the ineal gland,nor talk about its e!trasensory caabilities, e!ceting one& its sensitivity tolight.As medically theori(ed, the ineal gland resonds to the ebbs and ow oflight entering our eyes during the day. In the evening, the ineal gland reacts

to the diminishing levels of daylight and starts to roduce melatonin, whichis then released into the blood and ows through the body /0 /F making usdrowsy. Its secretion eaks in the middle of the night during our heaviesthours of slee.In the morning, bright light shining through the eyelids reaches the inealgland which reacts byswitching o+ the roduction of melatonin, thus removing the desire to slee.And we wakeO#ut this descrition is incomlete in one signicant asect. As it turns out,the ineal gland canbe diminished not only by light shining on the eyelids, but by light shining

anywhere on the body.Biterally, light striking any art of your skin can reduce roduction ofmelatonin from the inealgland. It seems the ineal can 8see without eyes.8 ow$s that for -H venmore interesting is the fact that in some lower vertebrates the ineal glandactually has a well'develoed eye'like structure and is considered by somescientists to be the evolutionary forerunner of the modern eye. In othervertebrates, though not organi(ed as an eye, it functions as a light recetor e+ectively a third eye. In any case, the key when it comes to the ineal glandand melatonin is that it$s imortant to slee in a darkened room, with no lightcoming through the curtains or night lights turned on in the room. And

wearing eyeshades won$t hel as the ineal can sense any light shining onyour skin. "ailure to slee in a darkened room will inhibit melatoninroduction, which resents a series of health roblems, not the least of whichis an inability to slee deely. #ut beyond that, if continued for too long, itwill literally shut down the ineal and cause it to atrohy. At that oint, youronly choice will be to use melatonin sulements.// /L


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The ineal gland %also called the ineal body, eihysis cerebri, eihysis orthe 8third eye8) is asmall endocrine gland in the vertebrate brain. It roduces melatonin, ahormone that a+ects the modulation of wakeEslee atterns andhotoeriodic %seasonal) functions.

ircadian rhythm and the Pineal-ome features of the human circadian biological clockA circadian rhythm is an endogenously driven roughly /4'hour cycle inbiochemical, hysiological, or behavioural rocesses. Circadian rhythms havebeen widely observed, in lants, animals,fungi and cyanobacteria %seebacterial circadian rhythms). The term 8circadian8 comes from the Batincirca, meaning 8around8, and diem or dies, meaning 8day8. The formal studyof biological temoral rhythms such as daily, tidal, weekly, seasonal, andannual rhythms is called chronobiology. Although circadian rhythms areendogenous %8built'in8, self'sustained), they are ad1usted %entrained) to theenvironment by e!ternal cues called(eitgebers, the rimary one of which isdaylight.

2istory The earliest known account of a circadian rocess dates from the 4th century#C, when Androsthenes, a shi catain serving underAle!ander the 7reat,described diurnal leaf movements of the tamarind tree.FU The rst recorded observation of an endogenous circadian oscillation was bythe "rench scientist Vean'VacGues d$?rtous de *airan in F;/=. e noted that/4'hour atterns in the movement of the leaves of the lant *imosa udicacontinued even when the lants were ket in constant darkness, in the rste!eriment to attemt to distinguish an endogenous clock from resonses to

daily stimuli./ULUIn F


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constant conditions with a eriod of about /4 hours. The rationale for thiscriterion is to distinguishcircadian rhythms from simle resonses to daily e!ternal cues. A rhythmcannot besaid to be endogenous unless it has been tested in conditions without

e!ternal eriodic inut.L. The rhythms can be ad1usted to match the local time %entrainable). Therhythm can be resetby e!osure to e!ternal stimuli %such as light and heat), a rocess calledentrainment. The rationalefor this criterion is to distinguish circadian rhythms from other imaginableendogenous/4'hour rhythms that are immune to resetting by e!ternal cues and, hence,do not serve theurose of estimating the local time. Travel across time (ones illustrates theability of the human

biological clock to ad1ust to the local time6 a erson will usually e!erience 1et lag beforeentrainment of their circadian clock has brought it into sync with local time.4. The rhythms maintain circadian eriodicity over a range of hysiologicaltemeratures %e!hibittemerature comensation). -ome organisms live at a broad range oftemeratures, and the/4 /5thermal energy will a+ect the kinetics of all molecular rocesses in theircell%s). In order tokee track of time, the organism$s circadian clock must maintain a roughly

/4'hour eriodicitydesite the changing kinetics, a roerty known as temeraturecomensation.

6riginhotosensitive roteins and circadian rhythms are believed to haveoriginated in the earliest cells, with the urose of rotecting the relicatingof PKA from high ultraviolet radiation during the daytime. As a result,relication was relegated to the dark. The fungusKeurosora, which e!iststoday, retains this clock'regulated mechanism.Circadian rhythms allow organisms to anticiate and reare for recise andregular environmental changes6 they have great value in relation to theoutside world. The rhythmicity aears to be as imortant in regulating andcoordinating internal metabolic rocesses, as in coordinating with theenvironment.=U This is suggested by the maintenance %heritability) ofcircadian rhythms in fruit ies after several hundred generations in constantlaboratory conditions,F0U as well as in


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creatures in constant darkness in the wild, and by the e!erimentalelimination of behavioural but not hysiological circadian rhythms in Guail.FFUF/U The simlest known circadian clock is that of the rokaryotic cyanobacteria.9ecent research has demonstrated that the circadian clock of-ynechococcus

elongatus can be reconstituted in vitro with 1ust the three roteins of theircentral oscillator. This clock has been shown to sustain a //' hour rhythmover several days uon the addition of AT. revious e!lanations of therokaryotic circadian timekeeer were deendent uon a PKAtranscritionEtranslation feedback mechanism.In F=;F, 9onald V. 3onoka and -eymour #en(er rst identied a geneticcomonent of the biological clock using the fruit y as a model system. Three mutant lines of ies dislayed aberrant behaviour& one had a shortereriod, another had a longer one, and the third had none. All three mutationsmaed to the same gene, which was named 8eriod8.FLU The same genewas identied to be defective in the slee disorder "A-- %"amilial advanced

slee hase syndrome) in human beings thirty years later, underscoring theconserved nature of the molecular circadian clock through evolution. *anymore genetic comonents of the biological clock are now known. Theirinteractions result in an interlocked feedback loo of gene roducts resultingin eriodic uctuations that the cells of the body interret as a secic timeof the day.A great deal of research on biological clocks was done in the latter half of the/0th century. Itis now known that the molecular circadian clock can function within a singlecell6 i.e., it is cellautonomous.F4U At the same time, di+erent cells may communicate with each other

resulting ina synchronised outut of electrical signaling. These may interface withendocrine glands of thebrain to result in eriodic release of hormones. The recetors for thesehormones may be located far across the body and synchronise the eriheralclocks of various organs. Thus, the information of the time of the day asrelayed by the eyes travels to the clock in the brain, and, through that,clocks in the rest of the body may be synchronised. This is how the timing of,for e!amle, sleeE wake, body temerature, thirst, and aetite arecoordinately controlled by the biological clock.

"m'ortance in animalsCircadian rhythmicity is resent in the sleeing and feeding atterns ofanimals, including human beings. There are also clear atterns of core bodytemerature, brain wave activity, hormone roduction, cell regeneration andother biological activities. In addition, hotoeriodism, the hysiologicalreaction of organisms to the length of day or night, is vital to both lants andanimals, and


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the circadian system lays a role in the measurement and interretation ofday length. Timely rediction of seasonal eriods of weather conditions, food availabilityor redator activity is crucial for survival of many secies. Although not theonly arameter, the changing length of the hotoeriod %$daylength$) is the

most redictive environmental cue for the seasonal timing fhysiology andbehavior, most notably for timing of migration, hibernation and reroduction."m'act o# light7dar( cycle The rhythm is linked to the lightdark cycle. Animals, including humans, ketin total darkness for e!tended eriods eventually function with afreerunningrhythm. ach 8day8, their slee cycle is ushed back or forward, deendingon whether their endogenous eriod is shorter or longer than /4 hours. Theenvironmental cues that reset the rhythms each day are called (eitgebers%from the 7erman, 8time'givers8).F:U It is interesting to note that totally'blind subterranean mammals %e.g., blind mole rat -ala! s.) are able tomaintain their endogenous clocks in the aarent absence of e!ternal

stimuli. Although they lack image'forming eyes, their hotorecetors %detectlight) are still functional6 as well, they do surface eriodically.F;U"reerunning organisms that normally have one or two consolidated sleeeisodes will still havethem when in an environment shielded from e!ternal cues, but the rhythm is,of course, notentrained to the /4'hour lightdark cycle in nature. The sleewake rhythmmay, in these circumstances, become out of hase with other circadian orultradian rhythms such as metabolic, hormonal, CK- electrical, orneurotransmitter rhythms.F


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/: /;Butterfy migration The navigation of the fall migration of the astern Korth American monarchbuttery %Panausle!ius) to their overwintering grounds in central *e!ico uses a time'

comensated sun comass that deends uon a circadian clock in theirantennae.//U/LU

"n 'lantsPiagram showing a small ortion of the transcritional feedback loo inArabidosis.BQ and CCAF are considered negative elements due to itsreression against T?CF in the morning while T?CF is considered a ositiveelement because it results in increased transcrition of BQ and CCAF duringthe evening because of its accumulation.lant circadian rhythms tell the lant what season it is in and when to owerfor the best chance of attracting insects to ollinate them and can includeleaf movement, growth, germination, stomatalE gas e!change, en(ymeactivity, hotosynthetic activity, and fragrance emission./4UCircadianrhythms occur as a biological rhythm with light, are endogenously generatedand self sustaining,and are relatively constant over a range of ambient temeratures. Circadianrhythms featurea transcritional feedback loo, a resence of A- roteins, and severalhotorecetors that netune the clock to di+erent light conditions.Anticiation of changes in the environment changes the hysiological statethat rovides lants with an adative advantage./5U A better understandingof lant circadian rhythms has alications in agriculture such as helingfarmers stagger cro harvests thus e!tending cro availability, and to secureagainst massive losses due to weather.Clocks are set through signals such as light, temerature, and nutrientavailability, so that theinternal time matches the local time. Bight is the signal and is sensed by awide variety of hotorecetors.9ed and blue light are absorbed through several hytochromes andcrytochromes.?ne hytochrome, hyA, is the main hytochrome in dark'grown seedlings,but raidly degrades in light to roduce CryF. hytochromes # are morestable with hy# the main hytochrome in light'grown seedlings. Thecrytochrome %cry) gene is also a light'sensitive comonent of the circadianclock. Crytochromes F/ %involved in blueDRA) hel to maintain the eriodlength in the clock through a whole range of light conditions./4U/5U The central oscillator generates a self'sustaining rhythm and is made of twogenes& CCAF %Circadianand Clock Associated F) and BQ %Bate longated yocotyl) that encodeclosely related


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*Q# transcrition factors that regulate circadian rhythms in Arabidosis.>hen CCAF and BQ are overe!ressed %under constant light or darkconditions) lants become arrhythimcal and m9KA signals reducecontributing to a negative feedback loo. CCAF and BQ e!ression oscillatesand eaks in early morning while T?CF oscillates and eaks in early evening.

"rom ast observations and studies, it is hyothesised that these threecomonents model a negative feedback loo in which over'e!ressed CCAFand BQ reress T?CF and over'e!ressed T?CF is a ositive regulator CCAFand BQ./5U

8iological cloc( in mammalsPiagram illustrating the inuence of light and darkness on circadian rhythmsand related hysiology and behaviour through the surachiasmatic nucleusand the ineal in humans. The rimary circadian 8clock8 in mammals is located in thesurachiasmaticnucleus %or nuclei)%-CK), a air of distinct grous of cells located in the hyothalamus.Pestruction of the -CK results in the comlete absence of a regular sleewake rhythm. The -CK receives information about illumination through theeyes. The retina of the eye contains 8classical8 hotorecetors %8rods8 and8cones8), which are used for conventional vision. #ut the retina also containsseciali(ed ganglion cells which are directly hotosensitive, and ro1ectdirectly to the -CK where they hel in the entrainment of this mastercircadian clock. These cells contain the hotoigment melanosin and their signals follow aathway called theretinohyothalamic tract, leading to the -CK. If cells from the -CK areremoved and cultured, they maintain their own rhythm in the absence ofe!ternal cues. The -CK takes the information on the lengths of the day and night from theretina, interretsit, and asses it on to the ineal gland, a tiny structure shaed like a inecone and located ontheeithalamus. In resonse, the ineal secretes the hormonemelatonin.-ecretion of melatonineaks at night and ebbs during the day and its resence rovidesinformation about night'length.-everal studies have indicated that ineal melatonin feeds back on -CKrhythmicity to modulatecircadian atterns of activity and other rocesses. owever, the nature andsystem'level signicanceof this feedback are unknown./< /= The circadian rhythms of humans can be entrained to slightly shorter andlonger eriods than the


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arth$s /4 hours. 9esearchers at arvard have recently shown that humansub1ects can at least beentrained to a /L.5'hour cycle and a /4.:5'hour cycle %the latter being thenatural solar day'nightcycle on the lanet *ars)./:U

Determining the h$man circadian rhythm The classic hase markers for measuring the timing of a mammal$s circadianrhythm are&2 melatonin secretion by the ineal gland2 core body temerature/;U2 lasma level of cortisol./


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et al. in /00/ comared cortisol and temerature data to eight di+erentanalysis methodsof lasma melatonin data, and found that 8methods using lasma melatonindata may be consideredmore reliable than methods using C#T or cortisol data as an indicator of

circadian hase inhumans.8/


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It is thought that the direction of the light may have an e+ect on entrainingthe circadianrhythm6LLU light coming from above, resembling an image of a bright sky,has greater e+ect thanlight entering our eyes from below.

According to a /0F0 study comleted by the Bighting 9esearch Center,daylight has a direct e+ecton circadian rhythms and, conseGuently, on erformance and well'being. Theresearch showedthat students who e!erience disrution in lighting schemes in the morningconseGuently e!eriencedisrution in sleeing atterns. The change in sleeing atterns may lead tonegativelyimacted student erformance and alertness. 9emoving circadian light in themorning delays thedim light melatonin onset by : minutes a day, for a total of L0 minutes for

ve days.L5UEn#orced longer cycles*odern research under very controlled conditions has shown the humaneriod for adults to be 1ust slightly longer than /4 hours on average. C(eisler et al. at arvard foundthe range for normal,healthy adults of all ages to be Guite narrow& /4 hours and FF minutes Y F:minutes. The 8clock8resets itself daily to the /4'hour cycle of the arth$s rotation.L:U The /


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and researchers are allowed to assess the e+ects of circadian hase onasects of slee and wakefulnessincluding slee latency and other functions.4/Uarly research into circadian rhythms suggested that most eole referred aday closer to /5

hours when isolated from e!ternal stimuli like daylight and timekeeing.arly investigators determinedthe human circadian eriod to be /5 hours or more. They went to greatlengths to shieldsub1ects from time cues and daylight, but they were not aware of the e+ectsof indoor electriclights.4LU The sub1ects were allowed to turn on light when they were awakeand to turn it o+ when they wanted to slee. lectric light in the evening delayed theircircadian hase. These resultsbecame well'known.L:U 9esearchers allowed sub1ects to kee electric

lighting on in the evening,as it was thought at that time that a coule of :0> bulbs would not have aresetting e+ecton the circadian rhythms of humans. *ore recent research has shown thatadults have a built'inday, which averages 1ust over /4 hours, that indoor lighting does a+ectcircadian rhythms and thatmost eole attain their best'Guality slee during their chronotye'determined slee eriods.

2$man health Timing of medical treatment in coordination with the body clock maysignicantly increase eScacyand reduce drug to!icity or adverse reactions. "or e!amle, aroriatelytimed treatmentwith angiotensin converting en(yme inhibitors %ACi) may reduce nocturnalblood ressure andalso benet left ventricular %reverse) remodelling.44UA short na during the day does not a+ect circadian rhythms.A number of studies have concluded that a short eriod of slee during theday, a ower'na,does not have any measurable e+ect on normal circadian rhythms, but candecrease stress andimrove roductivity.45U4:U There are many health roblems associated with disturbances of the humancircadian rhythm,such as seasonal a+ective disorder %-AP), delayed slee hase syndrome%P--) and othercircadianrhythm disorders.4;U Circadian rhythms also lay a art in the reticularactivating system,


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which is crucial for maintaining a state of consciousness. In addition, areversal in the sleewakecycle may be a sign or comlication of uremia,4


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L/ LL The ineal gland is located near to the center of the brain, between the twohemisheres, tuckedin a groove where the two rounded thalamic bodies 1oin. Dnlike much of therest of the brain, the

ineal gland is not isolated from the body by the blood'brain barrier system.It is reddish'gray andabout the si(e of a ea %< mm in humans), located 1ust rostro'dorsal to thesuerior colliculus andbehind and beneath the stria medullaris, between the laterally ositionedthalamic bodies. It isart of the eithalamus. It is a midline structure, and is often seen in lainskull Z'rays, as it is oftencalcied. Calcication is tyically due to intake of the uoride found in waterand toothaste. Itwas the last endocrine gland to have its function discovered.

Metaphysics The ineal gland$s location dee in the brain seems to intimate hiddenimortance. In the daysbefore its function as a hysical eye that could see beyond sace'time wasdiscovered, it was considereda mystery linked to suerstition and mysticism. Today it is associated with the si!th chakra whose awakening is linked torohecy and and increasedsychic awareness as consciousness ascends.Chakras -iraling >heels or Cones of nergyF/ Around -iraling Cones of Creation

The ineal gland, or third eye, is located in the geometric center of the brain. This correlates to thelocation of the 7reat yramid in the center of the hysical lanet.All'-eeing'ye and ye -ymbol This ineal gland is activated by Bight, and it controls the various bio'rhythms of the body. It worksin harmony with the hyothalamus gland which directs the body$s thirst,hunger, se!ual desireand the biological clock that determines our aging rocess. >hen it awakens,one feels a ressureat the base of the brain.

>hile the hysiological function of the ineal gland has been unknown untilrecent times, mysticaltraditions and esoteric schools have long known this area in the middle ofthe brain to be theconnecting link between the hysical and siritual worlds. Considered themost owerful andhighest source of ethereal energy available to humans, the ineal gland hasalways been imortant


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in initiating suernatural owers. Peveloment of sychic talents has beenclosely associatedwith this organ of higher vision.>hen awakened, the third acts as a $stargate$ thatL4 L5

sees beyond -ace'Time into Time'-ace. To activate the $third eye$ is to raise one$s freGuency and moving into higherconsciousness ' all isa consciousness e!erience erceived through the ye of Time or Third ye.*editation, Risuali(ation Qoga, and all forms of ?ut of #ody travel, oen the Third ye and allow you to$see$ beyondthe hysical. As you ractice, you will get it faster and more freGuently. Qoursychic abilities willincrease as well as your dream time messages. Qou may rst begin with youreyes closed, but as

you ractice, you will be able to oen your third eye by focusing yourattention and receiving messageswith your hysical eyes oen. lanetary vibrationEfreGuency is acceleratinge!onentially,allowing souls to eer into other realms far more easily than in the ast."reGuency will continueto rise until consciousness evolves out of the hysical in the ne!t few years. The ineal gland corresonds with divine thought after being touched by thevibrating light of 3undalini. starts its ascent towards the head center after resonding to thevibrations from the

$light in the head.$ The light is located at the to of the sutratma, or $soulthread$, which assesdown from the highest lane of our being into the hysical vehicle

oncl$sion>e$ll ause here and ick u our discussion of the endocrine system in thene!t newsletter withan e!loration of the thyroid and arathyroid glands. ?ne of the interestingthings you$ll noticeis that as we move down through the body, you$ll nd that you haverogressively more otionsfor altering the behavior of your endocrine glands. That said, you cannevertheless consider usingthe following sulements to assist the hyothalamus, the ituitary, and theineal glands in theotimal erformance of their basic functions.2 h7 secretagogue '' available throughout the internet2 Timed release melatonin


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2 yothalamus *7 from -tandard rocess '' available throughout theinternet2 ituitrohin *7 from -tandard rocess '' available throughout the internetL: L;L< L=

The Endocrine System: ThePancreas & Diabetes-everal months ago, we e!lored the anatomy and hysiology of theancreas in terms of its rolein the digestive rocess. #ut the ancreas is one of a handful of organs in thebody that functionsin two distinct modes. It is not only an e!ocrine digestive organ, but it alsofunctions as art of the endocrine system and, to a signicant degree, controls the metabolism

of sugar in the bodyand its use as a source of energy for every single cell and organ in the body.In this newsletter, wee!amine the endocrine functions of the ancreas. As an endocrine organ, theancreas roducestwo sugar'regulating hormones& insulin and glucagon. After reviewing thefunctions of insulin andglucagon and the four cell tyes that comrise the endocrine ancreas, we$lle!amine in detail themain disease associated with the ancreas, diabetes mellitus.

The 'ancreas #$nctions in t.o modes)As mentioned above, the ancreas functions in two distinctly di+erentmodes. It is both an e!ocrinedigestive organ that secretes digestive 1uicesand en(ymes into the duct of >irsung that runsdown the middle of the ancreas and emties into the duodenum at thehead of the ancreas.#ut the ancreas is also an endocrine organ, roducing insulin, glucagon,and somatostatin thatow directly into the bloodstream, eventually reaching virtually every cell inthe body.

natomy re*ie.>e e!lored the anatomy and hysiology of the ancreas in some detail inour newsletter focusedon that toic, but a Guick review would be aroriate before discussing thegland$s endocrinefunction.hysically, the ancreas is located in the uer abdominal cavity, towardsthe back '' in the C curve


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of the duodenum. It is about F/ inches long and taers from right to left.%9emember, anatomicallyseaking, left and right are referenced from behind the body so they areactually reversedin most diagrams that view the body from the front.) The thick art, the

head, comrises almost50M of the mass of the ancreas and lies to the right, nestled in the C'curveof the duodenum. Asfor the body of the ancreas, it moves u and to the left, taering into whatis known as the tail of the ancreas, which terminates at the 1unction of the sleen.40 4FAs might be susected for such an imortant organ, the ancreas is richlysulied with arteriesand veins. It is served by branches from the heatic artery, thegastroduodenal artery, the ancreaticoduodenal

artery, the suerior mesenteric artery, and the slenic artery.Kinety'nine ercent of the ancreas is made of acini, clusters of cells thatresemble a many'lobed8berry8 %acinus is Batin for berry). The acini roduce e!ocrine digestive 1uicesthat ow out of theacini through small ducts that eventually 1oin together and feed into theduodenum through theancreatic duct. #ut today, we are not interested in that ninety'nine ercent.>e are interested inthe one ercent of the ancreas that is made u of several million cellsscattered throughout the

ancreas, groued together in globules known as islets of Bangerhans. It isthese cells that containthe endocrine functioning of the ancreas. A healthy human ancreascontains about one millionsuch globules, which are distributed throughout the organ like tiny islets in avast ocean of acini'' hence their name. Their combined mass is a mere F to F.5 grams.

Physiology o# the endocrine 'ancreas #o$rcell ty'esA single islet of Bangerhans is actually comrised of four distinct tyes of

cells %alha, beta, delta,and gamma), two of which are rimary& alha and beta.l'ha cellsAlha cells constitute /0M of the islet$s cells. They secrete the hormoneglucagon, a olyetideof /= amino acids, which raises blood sugar to maintain normal levels. "orthe most art, glucagon


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does not resent the same roblems as insulin and will not raise blood sugarmuch above normal''


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to seed the return of normal levels of blood sugar. All of glucagon$s actionstend to counterthose of insulin, which works to reduce the level of glucose in the blood.Incidentally, glucagon,like insulin, is readily available thanks to genetically engineered bacteria and

recombinant PKAtechnology. This is done by inserting the human gene for insulin into . colibacteria, which then8grow8 genuine, bio'identical, human insulin in culture tanks. "or thosesGueamish about . coli,this rocess is also done by some manufacturers using yeast instead ofbacteria.8eta cells#eta cells constitute aro!imately


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aid in recovery and rest. 7lucagon itself causes insulin release to balance itse+ect in a negativefeedback loo. And nally, gastric inhibitory etide %7I) from theenteroendocrine cells of thesmall intestine resonds to glucose in the lumen of the gut, thereby signaling

the 8rearatory8release of glucose'deendent insulin from ancreatic beta cells. It should benoted that the e+ectof 7I on the ancreas is diminished by Tye / diabetes.And nally, beta cells also roduce insulin'like growth factors %secically,I7"'/), which is found inmany body tissues at concentrations far higher than insulin itself. It sharesthe molecular structureand shae of insulin and is involved in growth. As a side note, I7"'F%roduced in the liver) andI7"'/ are used by cancer cells to stimulate growth.

Delta cellsPelta cells constitute less than FM of ancreatic islets. They secretesomatostatin, the samegrowth'hormone'inhibiting hormone secreted by the hyothalamus. Thishormone inhibits insulinrelease and slows absortion of nutrients from the 7I tract.Gamma cells ;F cells<7amma cells also constitute less than FM of ancreatic islets. They secrete aancreatic olyetidethat inhibits the release of somatostatin. In other words, Pelta cells and7amma cells work to

regulate each other.Diabetes mellit$s ;s.eet $rine<Piabetes mellitus is actually not one disease, but a grou of disorders inwhich glucose levels areelevated in the blood. It is called a rotean %widesread) disease because ita+ects every systemin the body. %"or more on this concet, check out Piabetes '' The cho +ect'' highly recommended.)#y itself, it ranks somewhere between fourth and si!th as a leading cause ofdeath in

the D- '' and climbing the charts throughout the rest of the world. #ut whenconsidered as ama1or factor in cardiovascular disease and kidney failure, its true imact isrobably much higher.Its name, sweet urine, comes from the fact that it was originally diagnosedby tasting %not testing)the atient$s urine. The word 8mellitus8 is Batin for honey'sweet. levatedglucose levels make the


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immune suression, and insulin ums. To this oint, none of thesealternatives is without signicantroblems. ?n the other hand, although it cannot be controlled with diet ande!ercise,there are indeed alternative otions that can rove helful. >e$ll talk about

those a little later.In addition to the immediate roblems associated with e!cess blood sugar,diabetes also resentsother roblems. "or e!amle, atients are in a chronic state of starvation,unable to use nutrientswithout in1ections of insulin. In addition, cataracts of the lens of the eye anddiabetic retinoathyare related to high blood sugar. The e!cess sugar di+uses into the eye andforms a cloudy glycoroteinwith the lens. Another roblem associated with diabetes is if the body isunable to utili(e

blood sugar as energy for the cells of the body, it will try and convert asmuch of the e!cess glucoseas ossible into fat to store the energy. This not only leads to fatty livers, butto an e!cess of fat inthe blood. igh levels of fat in the blood, over long eriods, leads toatherosclerosis. ?ther hysicalroblems related to high blood liids and blood vessel damage %also causedby blood sugar)include strokes, heart attacks, kidney failure, eriheral vascular disease,and increased rates of infection '' not to mention, a high rate of amutation. %Again, check out

Piabetes '' The cho +ect.) There is another roblem associated with Tye I diabetes. -ince diabeticscannot use glucose forenergy e+ectively, their bodies shift to using fatty acids to roduce cellularenergy. This results inan e!cess of fatty acid wastes called ketones. 3etones are very, very acidic,and they cause a shiftto acidity in the blood. This condition is called ketoacidosis. Qou can smellacetone on the breathof a diabetic su+ering from ketoacidosis. Dncorrected, ketoacidosis is raidlyfatal.

It$s robably worth mentioning that low'carb diets work by turning dietersinto 8controlled8 diabeticsso that their bodies can shift from sugar burning to fat burning. +ectively,low'carb dietsinterrut the3rebs cycle by denying the body the F00 grams of glucose itneeds to rime the umfor sugar burning. As I mentioned, this rocess essentially turns dieters intocontrolled low'level


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diabetics and roduces a mild form of ketoacidosis. As a side note, if a dietereats rotein and fat,then triggers the 3rebs cycle, all e!cess material will be turned into fatanyway '' so ultimately,little is gained unless one chooses to remain ermanently a low level

diabetic."or more on low'carb diets, check out my series of newsletters on thesub1ect, Bow Carb Cra(iness.Ty'e "" diabetesAt one time, Tye II diabetes was known as adult onset diabetes becausealmost all its victimstended to be over 40 years of age. #ut those days are long gone, and now,thanks to catastrohicdietary changes in the develoed world %and with develoing countriesstruggling to imitate us) Tye II diabetes is now aearing in many children. -o it has been renamed.

It is now called noninsulin'deendent diabetes and accounts for some =0M of all diabetes cases. In fact,children nowaccount for /0M of all newly'diagnosed cases of Tye II diabetes and, liketheir adult counterarts,are usually overweight. -adly, it is almost always a self'inicted disease ''most often triggered byhigh glycemic diets and e!cessive weight. "ortunately, because it is self'inicted, it is usually muchmilder than Tye I diabetes %at least if caught in the early stages) and ismuch easier to control. In

fact, many atients have normal insulin levels. The roblem is that becausethe body has had toum out so much insulin over time to combat the high glycemic foodsdominating so many diets,the cells of the body have become rogressively less sensitive to the actionof insulin. They have,to use the common term, become insulin resistant.Although virtually every single cell in the body survives by convertingglucose to energy, skeletal4: 4;muscle is the ma1or 8sink8 for removing e!cess glucose from the blood and

converting it into glycogen).#ut in a Tye II diabetic, the ability of skeletal muscle to remove glucose fromthe bloodand convert it into glycogen may be only /0M of normal. This, again, iscalled insulin resistance."ortunately, vigorous e!ercise increases the ability of skeletal muscle totransort glucose across


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its cellular membrane, thus reducing the e+ect of insulin resistance. ?r tout it another way,eole who lead sedentary lives are more likely to develo Tye II diabetes.-ymtoms of Tye II diabetes are similar to that found in Tye I and includethe three olys mentioned

above. Treatment otions include&2 "or most atients '' diet, weight loss, and e!ercise.2 "or some atients '' harmaceutical drugs.2 "or a few atients '' insulin in1ections.?n the other hand, if atients are la! and do not control their disease earlyon, symtoms becomemore severe over time. It is as though after years of uming out insulin inan e+ort to overcomethe atient$s insulin resistance, the beta cells become e!hausted.Kote& there is a close relative of Tye II diabetes called gestational diabetes.

It usually results fromtransient elevations in blood glucose during regnancy. It causes the sameroblems as Tye IIdiabetes for the fetus.

"nherited Forms o# Diabetes %ellit$sA very small number of cases of diabetes result from mutant genes inheritedfrom one or botharents. These genes can cause diabetes in several di+erent ways.2 -ome mutant genes revent the body from actually manufacturing insulin.2 ?ther genes cause insulin recetor sites on cells to malfunction.2 -till another mutation revents the body from manufacturing glucokinase,an en(yme essentialfor glycolysis, the rst ste in converting glucose into AT, which energi(esevery single cellin the body.2 And yet another mutation messes u the sodium'otassium ummechanism %used to transortlarge molecules into and out of cells) in the beta cells of the ancreas so thatthe insulinthey create can never leave the cell and make its way into the bloodstream.In other words,the insulin is there, but unusable.>hile the symtoms of inherited diabetes usually aear in childhood oradolescence, atientswith inherited diabetes di+er from most children with Tye / diabetes in thattheir families havea history of similar roblems and they are not necessarily obese. #ut again,inherited diabetesreresents only a small ercentage of diabetic atients.


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=at$ral treatments #or diabetesDltimately, Tye I and Tye II diabetics end u at the same lace even thoughthey arrive therethrough very di+erent means. In Tye I diabetes, you end u with high bloodsugar because your

body can$t roduce enough insulin to drive the sugar into cells where it canbe used for energyroduction. In Tye II diabetes, your body can roduce more than enoughinsulin %at least in thebeginning), but because cells become resistant to the e+ects of that insulin,sugar stays in theblood because it can$t get transorted into the cells of the body. Thus, thealternative methods fordealing with both tyes of diabetes are similar '' with a coule of additionsfor Tye I diabetes todeal with the autoimmune factor.=at$ral 'rotocol #or dealing .ith diabetes#eyond modifying your diet and e!ercising, you might want to inhibitabsortion of high glycemicfoods, without creating unwelcome resonses in the intestinal tract, such asthose e!eriencedusing metformin. This drastically reduces the amount of insulin your bodyreGuires and minimi(esthe chances of having both sugar and insulin sikes. It can be accomlishedwith the followingherbs&

2 Koal cactus2 7ymnema sylvestreKaturally reverse insulin resistance so less insulin is reGuired. Again, thebenets for both Tye Iand Tye II diabetes are obvious&2 3on1ac mannan2 Cinnulin "2 Chromium 7T"2 ?mega'L fatty acids9eair beta cells in the islets of Bangerhans in the ancreas to otimi(einsulin roduction reserves

as oosed to forcing the cells to dramatically overroduce as withglyburide, which leadsto inevitable burn out. This is a 8sine Gua non8 for Tye I diabetes and isessential if you want torevent rolonged Tye II diabetes from 8burning out8 the beta cells throughforced overroductionof insulin.2 7ymnema sylvestre


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2 Alha lioic acid or 9 lioic acidBower blood sugar levels through roer diet and herbal sulementation&2 "enugreek e!tract2 *omordica charantia2 Corosolic acid

2 *ulberry9educe stress. 9emember, adrenaline suresses the release of insulin.S'ecic #or Ty'e " diabetes-ince it is strongly susected that Tye I diabetes results from an out ofcontrol immune systemthat attacks and destroys the beta cells in the islets of Bangerhans, it isessential that you try andmodulate your immune system to minimi(e, or even eliminate, this factor.4< 4=ImmunomodulatorsKatural immunomodulators retrain your immune system to not overreact ''

and without deadlyside e+ects.2 B'carnosine2 Cetylmyristoleate %C*?)2 Transfer "actor found in bovine colostrums2 7insengTaking on viruses There is steadily mounting evidence that a virus may be resonsible fortriggering the autoimmuneresonse that causes Tye I diabetes. If so, then you will want to useantiathogens to hel

reduce or eliminate that viral load.2 7arlic2 ?live leaf 2 inc2 7raefruit seed e!tract2 >ild mountain oil of oreganodditional ste'sIf you clicked the link to Piabetes& the cho +ect, you know that diabetesotentially a+ects almostevery organ in the body '' many of which, as they degrade, can e!acerbatethe original diabeticroblem. Therefore, anyone su+ering from diabetes will want to do whateveris necessaryto rotect those organs.rotect organs and roteins from damage caused by higher than normallevels of sugar through ami!ture of antio!idants and nutraceuticals such as&2 B'Carnosine


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2 Acetyl'l'carnitine2 P*A2 CoXF02 Alha lioic acid or 9 lioic acid2 #enfotiamine

rotect organs from damage caused by higher than normal insulin levels bycleaning the blood byusing&2 A blood cleansing formula2 roteolytic en(ymes2 ?mega'L fatty acidsAnd that concludes our e!loration of the endocrine functions of theancreas. In our ne!t newsletter,we will conclude our e!loration of the endocrine system by e!amining theadrenal glands.

The Endocrine System: Thyroidand ParathyroidGlandsIn our last newsletter, we began an e!loration of the endocrine system bye!amining the threeendocrine glands in the brain& the hyothalamus, the ituitary, and theineal gland. In this issue,we move down the body to e!amine the ve endocrine glands found in theneck& the thyroid

and the four arathyroid glands. The thyroid gland regulates the rate andintensity of the body$schemicalEmetabolic reactions, and the arathyroid glands regulate theamount of calcium andhoshorus in the blood. As it turns out, malfunctions in these glands are notthat uncommon,can roduce serious roblems such as over e!citement of the muscle andnervous systems, bonydeminerali(ation, high calcium levels, duodenal ulcers, kidney stones, andbehavioral disorders.And if left unchecked, they can kill you. "ortunately, there are things you can

do to minimi(ethe chances of these roblems occurring in the rst lace, or relieving themthrough alternativemeans if you get them.>ith that in mind, let$s begin by looking at the thyroid gland.

Thyroid o*er*ie.


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In essence, the thyroid gland is the thermostat of the body. It regulates boththe rate and intensityof chemicalEmetabolic rocesses. It is one of the largest endocrine glands inthe body andsecically controls how Guickly the body uses energy, how it makes

roteins, and the body$ssensitivity to other hormones. The function of the thyroid gland is to takeiodine and convert itinto thyroid hormones '' rimarily, thyro!ine %T4) and triiodothyronine %TL).Kormal thyroid cellsaccumulate and retain iodidefar, far more eSciently than do any other cellsin the body. *ost cellsdon$t absorb iodine at all, but some, including thyroid cancer cells and breasteithelial cells, canto a limited degree. Thyroid cells combine iodine and the amino acid tyrosine%as bound to thyroglobulin)

to make TL and T4. %>e will cover this rocess in more detail a little later.) TLand T450 5Fare then released into the bloodstream and transorted throughout the body,where they controlmetabolism %i.e., the conversion of o!ygen and calories to energy). very cellin the body deendsuon thyroid hormones for regulation of their metabolism.Anatomically seaking, the thyroid is a buttery shaed gland %two largerlobes connected by anarrower isthmus) located between the Adam$s ale and the clavicle. >hen

viewed from thefront of the body, the thyroid totally covers the trachea. Kevertheless, anormal thyroid gland cannotbe felt e!ternally. If a doctor can 8see8 it or 8feel8 it when touching the neckwith his ngers,it$s enlarged. Dnder normal circumstances, it$s soft and at.Kot surrisingly for such an imortant organ, it is richly serviced by multilearteries and veins,which makes surgery on the thyroid that much more diScult. In addition,surgeons face furthercomlications since the nerves that service the vocal cords run right ne!t to

the arteries that rovideblood to the thyroid. #ottom line is that the thyroid is intricately entwinedwith key nervesand blood vessels. And it$s not 1ust surgery on the thyroid that resentsroblems. Tracheotomies,for e!amle, must be erformed either above or below the thyroid gland. It isalso the main reason


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doctors refer to 8kill8 the thyroid with radioactive iodine rather than removeit surgically %arocedure we will talk more about later).At the micro level, the thyroid is rimarily comrised of sheres calledfollicles. The follicles themselves

are rimarily comosed of two tyes of cells&F. ?n the outside circumference of the follicles are the cuboidal follicularcells. The follicular cellsroduce two iodine based comounds, thyro!ine %tetraiodothyronine, alsoknown as T4) andtriiodothyronine %also known as TL). ?n the inside circumference, or lumen of the follicle, isa brush border comosed of hairlike e!tensions %not visible in the slidebelow). This allows forthe easy deosit and removal of key hormonal comonents into the follicularlumen %see slide

below) as reGuired for roduction of TL and T4./. The arafollicular cells %C cells) sit scattered about the outer edge of thefollicles on to of thefollicular cells and roduce calcitonin, a minor regulator of calcium in thebody.

Thyroid hormones>hen talking about thyroid hormones, we$re actually talking about four bio'chemicals&2 Thyroglobulin is a rotein %not a hormone) roduced by the thyroid. It issynthesi(ed fromamino acids and iodide and stored in the follicular lumen as colloid and usedentirely withinthe thyroid gland in the roduction of the thyroid hormones.2 TL %triiodothyronine) a+ects almost every hysiological rocess in thebody, including growthand develoment, metabolism, body temerature, and heart rate. roductionof TL and itsrohormone, T4 %TL is actually roduced by the breakdownEconversion of T4), is activated bythyroid'stimulating hormone %T-), which is released from the ituitarygland. As a side note,the L in its name refers to the fact that it contains L iodine atoms.2 T4 %thyro!ine, A3A tetraiodothyronine) is the rohormone from which thebody e!tracts TL.It is synthesi(ed from residues of the amino acid tyrosine, found inthyroglobulin. very cell inthe body deends uon the thyroid hormones TL and T4 for regulation oftheir metabolism.


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The normal thyroid gland roduces about e will talk more about this later.) If iodine is not resent insuScient amounts,the body will develo a benign goiter %enlargement of the thyroid) over time.

It is common inareas where iodine does not naturally occur in food.In the early F=00$s, >estern countries began adding iodine to salt to combatthis roblem. Andit worked, in the sense that goiters are now uncommon in the >estern world.#ut using iodi(edsaltresents its own roblems. -urrisingly, a number of 8older8 societiesrecogni(ed the connectionbetween iodine and goiters. The ancient 7reeks, for e!amle, consumediodine'rich seaweedto successfully combat goiters '' without the roblems associated with

iodi(ed salt. -ometimesgrandma really does know best.As seen in the slide above, the thyroid stores something called colloid %whichis manufactured inthe follicular cells) in the center %lumen) of the follicles in large Guantities.Although colloid containssome TL and T4, it is rimarily comrised of thyroglobulin, which isconverted to TL and T4


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and released into the body when triggered by thyroid stimulating hormone%T-), released by theituitary. In fact, a healthy thyroid stores about a three'month suly ofthyroglobulin at any givenmoment in time.

As we touched on in our last newsletter, thyroid'stimulating hormone %T-)from the anteriorituitary regulates the rocesses via a negative feedback loo. That is to say,thyroid releasing hormone%T9) from the hyothalamus stimulates the ituitary to release T- into thebloodstream,which stimulates thyroid follicular cells to add iodine to the amino'acid%tyrosine) comonent of thyroglobulin %which, once again, is stored as colloid within the lumen of thethyroid follicles).?nce converted, the TL and T4 hormones are released into the bloodstream.

This arrangementessentially works as a reserve system for thyroid hormones, allowing it torelease active hormonesinto the body on an as needed basis. As more thyroid hormones areroduced, blood levels of TL and T4 rise. Dltimately, these hormones make their way through thebloodstream back to thehyothalamus, telling the hyothalamus that enough is enough and to storeleasing T9, whichstos the ituitary from releasing T- '' shutting down the cycle.It should be noted that the thyroid hormones are slow acting. Dnlike

adrenalin, for e!amle, ittakes awhile for anything to haen with thyroid hormones.

Thyroid hormone #$nctions Thyroid hormones regulate the following activities&2 ?!ygen utake %they u regulate it).2 7ross basal metabolic rate %they u regulate it).2 *aintenance of body temerature.2 Intracellular metabolism %microscoic rotein synthesis, liid breakdown,and cholesterolbreakdown.) atients who are hyothyroid, for e!amle, will have higherlevels of cholesterolin the blood because of reduced thyroid u regulation. atients who arehyerthyroid will oftenbe thin and have lower levels because of too much u regulation.2 7rowth and develoment6 that is, body growth rate and nervous systemdeveloment.2 Thyroid hormones also enhance the e+ects of catecholamines, accountingfor high blood ressure,


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nervousness, sweating, and fast heart rate in hyerthyroid atients.

"odine $'ta(e and controlIodide %I') ions circulating in the blood are actively taken into follicular cellsthrough caillariesand become traed in the endolasmic reticulum inside the follicular cells.

?nce iodine is resent,the follicles begin synthesi(ing thyroglobulin. Resicles %small transortmembranes) transortsome of the iodide further into the follicles, where it is combined withthyroglobulin to roducethe amino acid tyrosine. This combination of thyroglobulin and tyrosine isbound into colloid,which can be transformed into TL and T4 as needed.54 55Incidentally, the thyroid$s ability to tra iodine can be used clinically.2 Bow levels of radioactive iodine %I'FLF), combined with !'ray e!osure, canma thyroid function.2 igher levels of I'FLF will irradiate and destroy thyroid tissue, whenneeded, without damagingsurrounding cells. This can be used to kill o+ some of the thyroid to downregulate its function,or kill the whole organ. >hen 3risten was diagnosed with 7raves$ diseasemany yearsago, this was the rst thing they wanted to do to her. -he chose to go analternative route. AGuarter of a century later, she still has her thyroid, and it functions erfectly.%At some oint, Iwill get her to tell her comlete story in a newsletter '' when she can nd thetime.)o Theoretically, if doctors give a hyothyroid atient some synthetic thyroidhormones, they$renot too concerned if they give a little too much, since the negative feedbackloo will ick u thee!tra hormone in the blood and sto the ituitary from releasing more T-. If they give too little,the functioning cells in the thyroid will be stimulated to release enough to8to8 levels o+. #ut thatassumes that there$s still some functioning thyroid cells in the neck toresond to stimulation if necessary. If the thyroid has been surgically removed or killed o+ by I'FLFirradiation, then there isno self'regulatory mechanism. Trying to mechanically balance thyroid levelswith harmaceuticalsat that oint is almost imossible, and eole are forced to live the rest oftheir lives in a borderline


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sychotic state. It was for that reason that 3risten chose a natural solution.2 Cancerous cells will not tra iodine when normal thyroid tissue is resent. Tumors must besurgically removed or thyroid tissue must be destroyed with higher levels ofI'FLF, before

I'FLF can be re'administered to destroy cancerous thyroid cells.Thyroid dys#$nction The two main tyes of thyroid disease fall into hyerthyroidism %7raves$disease), and hyothyroidism%ashimoto$s thyroiditis).2y'erthyroidismyerthyroidism causes increased heart rate, increased blood ressure, highbody temeratureand sweating, nervousness, diarrhea, heat intolerance, and weight lossdesite high caloric intake.In other words, the metabolic rocesses are u regulated to dangerouslevels. Also, it can lead tosevere neurotic behavior. 7raves$ disease, a secic form ofhyerthyroidism, is an autoimmunedisorder in which antibodies mimic the e+ects of T- but are not constrainedby the negativefeedback system for turn'o+ and control6 thus, they continue to drive thethyroid to release stimulating TL and T4 hormones without letu. This disease causes goiter, enlargementof the thyroid,and e!ohthalmos %bulging eyeballs caused by the build'u of fat behind the

eye). Curing thediseases %often involving the destruction or removal of the thyroid followedby the lifelong administrationof synthetic hormones) may not cure e!ohthalmos, which may leave theeyes oen toin1ury. >hen talking about 7raves$ disease and bulging eyes, the late actor,*arty "eldman almostimmediately comes to mind.2y'othyroidismyothyroidism is a condition in which the thyroid gland does not makeenough thyroid hormone.

arly symtoms include&2 #eing more sensitive to cold2 Constiation2 Peression2 "atigue or feeling slowed down2 eavier menstrual eriods2 Voint or muscle ain2 aleness or dry skin


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2 Thin, brittle hair or ngernails2 >eakness2 >eight gain %unintentional) There are two fairly common causes of hyothyroidism. The rst is a result of inammation of the

thyroid gland which leaves a large ercentage of the cells of the thyroiddamaged %or dead) and incaableof roducing suScient hormone. The most common cause of thyroid glandfailure, however,is called autoimmune thyroiditis %aka ashimoto$s thyroiditis), a form ofthyroid inammationcaused by the atient$s own immune system. %Think of it as the i side of7raves$ disease.)Pr. Bee covers hyothyroidism in >hat Qour Poctor *ay Kot Tell Qou about*enoause. "irst, heoints out that thyroid roblems are far more common in women than in men

'' a strong indicatorthat we$re dealing with an estrogen issue. Then he oints out that for mostwomen, when theystart using rogesterone cr[me, their need for thyroid sulements is greatlyreduced '' and ofteneven eliminated. Kote& 1ust because it is more common in women, does notmean that men cannothave estrogen roblems also '' caused by e!osure to chemical estrogens.If you su+er from hyothyroidism, removing your thyroid or blasting it withradiation or trying tobalance it out with synthetic medication are not your only otions. There are

natural rogesteronecr[mes %for both men and women), which easily can be found by searchingthe net. Also, immunomodulatorssuch as cetyl'myristoleate and B'carnosine might make sense in case theroblemis associated with an autoimmune disorder. And nally, thyroid e!tracts suchas -tandard rocess$ Thytrohin *7 can be helful in rebuilding lost thyroid function.5: 5;retinismyothyroidism during fetal develoment totally disruts normal

develoment atterns, leadingto dwarsm, mental retardation, and hysical deformities. %Kow usuallycalled 8thyroid dwarsm.8)Thyroid cancerCancerous thyroid tumors %nodules) are most often associated with atientswho have had their


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faces irradiated %at one time this was done to treat acne '' really), but thesecancers are easily curableby simly removing the cancerous nodules. ?ther risk factors include&

=at$ral 2ashimoto>s Treatment ?o$r "odine

S$''lements %ay8e %a(ing ?o$ +orse A Voice O Caution When Treating ashimoto!s "aturally WithIodine#>hen a atient is diagnosed with low thyroid roduction, medically referredto as hyothyroidism,one of the rst things commonly rescribed is iodine sulementation. This islogical andvalid on the surface. Iodine is a comonent in thyroid hormone roduction.>orldwide iodinedeciency is a widesread roblem. In areas of the world where iodine isdecient, iodine is thecause of many if not most cases of hyothyroidism. In areas where iodine isnot decient in thefood suly, including the Dnited -tates, iodine is not usually the cause oflow thyroid.In areas of the world with adeGuate iodine in food, or where salt is iodi(ed,the most commoncause of low thyroid is ashimoto$s Autoimmune Thyroid. It is estimated thatu to =0 ercent of cases of hyothyroidism in the Dnited -tates is from this autoimmunemechanism.Autoimmune thyroid is not rimary hyothyroidism6 instead it is a form ofhyothyroidism that iscaused by immune destruction. The amount of iodine that is contained isiodine sulements andthyroid natural suort roducts act like gas on a burning re.In a /004 article in the 1ournal Thyroid, the author stated 8...the e!losivemi! of iodine, T? Ab,and /0/ necessary for thyroid hormone synthesis, inadvertently rovide thetrigger for the autoimmunethyroid resonse.8 It is this misguided inclination to give everyhyothyroidism atienthigh doses of sulemental iodine that leads to increased thyroid glanddestruction, and moresu+ering on the atient$s art. *ost natural healthcare ractitioners ossessa very limited understandingof autoimmune hysiology and continue giving iodine sulements for allcases of


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hyothyroidism, in the same way as the medical community uses thyroidrelacement hormonesas a 8blanket8 treatment for all low thyroid conditions.If you have been diagnosed with ashimoto$s disease then you need to takean inventory of your

sulements. 9emove sulements that contain iodine, as it is most likelyaggravating your condition,and is most likely one of many things working against you as you try toroerly treat andmanage your condition. >e get lenty of iodine from the iodini(ation of saltand from a normalbalanced diet.3 2erbs #or 2y'erthyroidism ?o$ %ight +ant To onsiderTa(ing*any eole with hyerthyroidism look to take nutritional sulementsandherbs to hel manage

their symtoms naturally andEor restore their health back to normal. Thetruth is that curinghyerthyroidism involves more than 1ust taking sulements and herbs, asthere are manyother comonents to consider. I\m talking from self e!erience, as Iersonally was diagnosedwith 7raves\ Pisease in the ast, which is an autoimmune hyerthyroidcondition. >hile mostendocrinologists label 7raves\ Pisease as being incurable, I successfullyrestored my health backto normal by following a natural hyerthyroid treatment rotocol.

art of this natural hyerthyroid treatment rotocol did involve takingnutritional sulementsand herbs. I\m not going to discuss all of the di+erent sulements andherbs I took when I followedsuch a rotocol, but I am going to focus on three herbs which can lay animortant rolein natural symtom management, which is imortant when trying to restorethe health of anyerson who has hyerthyroidism. I ersonally took two of the followingherbs. -ome eole withhyerthyroidism might only need to take one of them, while others mightneed to take all three.In order to gure out e!actly which of these herbs you should take and thesecic dosage youneed, it\s best to consult with a cometent natural endocrine doctor.Anyway, let\s take a look at the following three herbs&#ugleweed. >hen I was diagnosed with 7raves\ Pisease, #ugleweed was oneof the rimary herbs


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resonsible for my recovery. ?bviously I took numerous nutritionalsulements and herbs besidesthis one, and these others were all benecial. owever, #ugleweed is a greatherb thatis secic for hyerthyroidism, and it did a wonderful 1ob of managing my

symtoms naturally.>ithout this herb, I\m retty certain I would have needed to take anti'thyroiddrugs to hel withthe symtoms I was e!eriencing.?f course this doesn\t mean that this herb should always be used as arelacement for anti'thyroiddrugs. It really does deend on the severity of the symtoms, as while I hada high ulse rateand heart alitations, I didn\t consider my symtoms to be life threatening.?n the other hand, it5< 5=

still was a risk for me to not manage the symtoms using rescrition drugs,and this is the mainreason why I can\t recommend for anyone with hyerthyroidism or 7raves\Pisease to not takeanti'thyroid drugs. This is only a decision you can make on your own,although it is wise to consultwith your endocrinologist, as well as a cometent natural endocrine doctorfor some guidance.*otherwort. This is another sulement which can hel eole withhyerthyroidism. I didn\t begintaking this herb immediately, as I started by taking #ugleweed %along with

some other sulementsand herbs). And while the #ugleweed did hel a great deal with thesymtoms, I still washaving some noticeable heart alitations, and so I began taking both the#ugleweed and the*otherwort together. Taking both of these herbstogether did a

natural endocrine

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