what?, why?, and how? to refer imran jawaid. introduction what?, why? and how? to refer

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What?, Why?, and How? to refer Imran Jawaid

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What?, Why?, and How? to refer

Imran Jawaid

INTRODUCTIONWhat?, Why? and How? To refer

The day job..OPTOMETRISTS OPHTHALMOLOGISTS

Asymptomatic Symptomatic

No ocular pathology Ocular pathology

Screening / disease detection Treatment / disease management

Detect abnormality Diagnosis and investigation

Spectacles and contact lenses No refractive correction

Retail pressures (ATV, CR etc.) No retail pressures

9-5pm 24 hours

Career progression not based on ability to detect/manage ocular abnormality

Career progression based on ability to manage ocular abnormality

CHANGING TIMESWhat? Why? and How ?to refer

“Call to action” – The future...

• Improve IT links between community optical practices and the rest of the NHS and primary care

as well as improved systems in hospitals

• Address capacity issues in hospital eye clinics to save patients from unnecessary blindness and vision impairment

• Maximise the use of the skills in the eye care pathway by ensuring that patients are treated in the appropriate place by the appropriate professional at the appropriate time, whether in the community or in the hospital

• Procure community schemes at greater scale to reduce procurement and commissioning costs and direct more resource to clinical care.

• Improve communication and relationships between the multiple professions through better commissioning to achieve a more integrated eye care pathway and better patient care

ARE WE READY FOR THE CHANGE?What?, Why?, and How to refer

Bridging the gap

• Improve knowledge base• Improve feedback to referring optometrists• Increased exposure to ocular abnormality• Improve understanding of disease

management• Improve understanding of new treatments

and diagnostic equipment

Common problems

• Raised IOP• Flashes and floaters• Retinal haemorrhages• Red eye

Elevated IOPs

22T29 HistoryAsymptomatic?Angle closure sxs? (acute/intermittent)Photophobia?Visual change?Previous Trauma?Medications? (Steroid/Topiramate)Ocular treatments (Laser/Buckle)Ocular History (Ischaemic retina)

Pressure..

• History – Symptomatic vs Asymptomatic

• Anterior Segment signs– Red eye– KS, KPs and corneal pathology– AC activity and depth– Iris atrophy / TIDs / NVI– Significant cataract

Pressure..

• Gonioscopy• www.gonioscopy.org

• Redmond-Smith Index• Van Herrick

Flashing lights and floaters

Flashing lights and floaters...

• History– Myopia– Family history– Visual field defect– Fellow eye status

• Age – 6% under 50, 53% over 50, 67% over 65

Examination

• IOP• AC activity• Vitreous – tobacco dust?, RBC?, vitritis?• Fundus

– Weiss ring– Vitreous or pre-retinal hge– Peripheral retina

• Tear/break +/- SRF• Peripheral degeneration - Lattice

Breaks..

Retinal Haemorrhage

• Anatomy– Inner 2/3 - CRA – Outer 1/3 - Choroidal circulation– CRA divides into superior and inferior branches which

each divide into nasal and temporal branches– Functionally these are end-arteries– Central foveal avascular zone contains outer retinal layers

only. Blood supply to this area is derived from the underlying choroidal circulation.

– Post arteriole retinal capillaries - NFL – Pre-venular capillaries - INL

Pre-retinal haemorrhage...

Subhyaloid haemorrhages are found between the inner limiting membrane and the posterior hyaloid face. A pre-retinal haemorrhage is located between the inner limiting membrane and nerve fibre layer. Both bleeds mask the underlying vessels

Flame haemorrhage

Hypertensive retinopathy, vein occlusion, AION, disc swelling amongst othersRecording blood pressure is essential Marker for the site of future nerve fibre and corresponding visual field lossLocation should be accurately documented and further investigations performed

Pale centred linear haemorrhage

Conditions in which Roth spots are observed * subacute bacterial endocarditis* leukaemias* anaemia* anoxia* carbon monoxide poisoning* hypertensive retinopathy* pre-eclampsia* diabetic retinopathy* neonatal birth trauma* shaken baby syndrome

Dot and blot..

Compact middle layers of the retina give rise to the dot and blot-like appearance of these haemorrhagesOften there is associated oedema and if the macula is involved may give rise to diminished acuity. Blot haemorrhages are often larger and darker.Alert the examiner to search for other features of ocular ischaemia –namely venous changes, cotton wool spots and neovascularisation.

Sub-retinal Haemorrhage

Between neuro-sensory retina and RPE. They are dark in colour and the retinal vasculature is clearly visible aboveBleed can be large in area and variable in shape Sub-RPE bleeds, (between RPE and Bruch’s membrane of the choroid), have a more confined arrangement as there are tight junctions between RPE cells Commonest cause is choroidal neovascularisationOther causes include trauma, tumours and retinal angiomas

Red eye

• History– Pain

• FB sensation/ache/ deep pain

– Photophobia– V/A– Lacrimation / discharge– Associated symptoms

• Nausea and vomiting/ frontal headache

– C/L wear

Examination

• Pattern of redness– Diffuse and superficial– Diffuse and deep– Circum-corneal– Sectoral

• Reduced vision• Pupils• NaFl

PainPhotophobiaV/ADischargeAssoc. Sxs.C/L wear

Episcleritis

Scleritis

How to refer• S Situation: • Identify yourself the site/unit you are calling from • Identify the patient by name and the reason for your report • Describe your concern

• B Background: • Give the patient's reason for attendance• Explain significant medical/ocular history

• A Assessment: • Clinical impressions, concerns

R Recommendation: • Explain what you need - be specific about request and time frame • Make suggestions • Clarify expectations

THANK YOUQUESTIONS?

What?, why? and how to refer