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ADAR2 editing activity in newly diagnosed versus relapsed pediatric high-grade astrocytomas
ADAR2 editing activity in newly diagnosed versus relapsed pediatric high-grade astrocytomas
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ATM
ATM
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INTRODUCTION As part of our ongoing search for predictive tools to understand skin responses, we are evaluating tissue engineered human skin equivalent
INTRODUCTION As part of our ongoing search for predictive tools to understand skin responses, we are evaluating tissue engineered human skin equivalent
218 views
Biology, Biologists, and Society What is science? Science is a way of knowing based on experimental or observational evidence and its interpretation. Science
Biology, Biologists, and Society What is science? Science is a way of knowing based on experimental or observational evidence and its interpretation. Science
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A b Supp. Figure 1: Anoikis-resistance of MDA-MB-231 is dependent on PI3Kγ (a) Cells were prevented from attaching for 48 hours in the presence of DMSO,
A b Supp. Figure 1: Anoikis-resistance of MDA-MB-231 is dependent on PI3Kγ (a) Cells were prevented from attaching for 48 hours in the presence of DMSO,
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Supplementary Figures Supplementary Figure 1. T183 of JNK and WW domain of Pin1 are involved in the interaction. (a) HEK 293 cells transfected with plasmids
Supplementary Figures Supplementary Figure 1. T183 of JNK and WW domain of Pin1 are involved in the interaction. (a) HEK 293 cells transfected with plasmids
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(a) Representative phase-contrast photomicrographs of adherent cells and mammospheres formed after 11 days; ×10 objective. The single cell suspension from
(a) Representative phase-contrast photomicrographs of adherent cells and mammospheres formed after 11 days; ×10 objective. The single cell suspension from
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SUPPLEMENTARY FIGURES Figure S-I. Fluorescence intensity and subcellular localization of transfected EGFP fusion proteins HeLa cells were transferred into
SUPPLEMENTARY FIGURES Figure S-I. Fluorescence intensity and subcellular localization of transfected EGFP fusion proteins HeLa cells were transferred into
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NEGpre-221 pre-222pre-221/222 NEG DMSO 100 nM Fulvestrant for 72h Figure S1. Cell morphology of MCF7 cells after treatment with fulvestrant, with or without
NEGpre-221 pre-222pre-221/222 NEG DMSO 100 nM Fulvestrant for 72h Figure S1. Cell morphology of MCF7 cells after treatment with fulvestrant, with or without
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70 25 5 PI Cell number proliferating A549 19 10 71 PI Cell number 40 38 22 PI Cell number 20 13 67 PI Cell number senescent A549 proliferating MCF-7senescent
70 25 5 PI Cell number proliferating A549 19 10 71 PI Cell number 40 38 22 PI Cell number 20 13 67 PI Cell number senescent A549 proliferating MCF-7senescent
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Supplementary Figure 1 Supplemental Figure 1. Nogin siRNA potentiates BMP2-induced Smad-dependent BMP- signaling MDA-MB-231 breast cancer cells. MDA-MB-231/BREluc
Supplementary Figure 1 Supplemental Figure 1. Nogin siRNA potentiates BMP2-induced Smad-dependent BMP- signaling MDA-MB-231 breast cancer cells. MDA-MB-231/BREluc
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Relative mRNA Levels Stat 1 WT1 5nM WT1 siRNAs Treatment Stat 1 Relative mRNA Levels nM TLR3 siRNA Treatment Supplemental Figure1. Stat 1 upregulation
Relative mRNA Levels Stat 1 WT1 5nM WT1 siRNAs Treatment Stat 1 Relative mRNA Levels nM TLR3 siRNA Treatment Supplemental Figure1. Stat 1 upregulation
220 views