SILA THONGLAI MD.

Bangkok Eye center Bangkok Hospital

Thailand

Ocular Anatomy

1. Frontal bone

2. Zygomatic bone

3. Maxillary bone

4. Sphenoid bone

5. Ethmoid bone

6. Lacrimal bone

7. Palatine bone

1

2 3

4 5

6

7

Bony Components of Orbit

Size 30 x 40 x 45 mm

Paranasal Sinus

Ocular Anatomy

Ocular Anatomy

Extraocular Muscles

Optic Nerve

Ocular Emergencies

Trauma

Non - trauma

Blunt trauma

Penetrating trauma

Retinal arterial Perforation Orbital cellulitis

occlusion Ruptured Orbital injury

Chemical burns Acute glaucoma Corneal ulcer

Sudden congestion Corneal abrasion

proptosis Hyphema

Intraocular FB

Retinal detachment

Macular edema

( Immediately ) ( Within a few hours ) ( Within one day )

Acute Eye Conditions

Emergency Very Urgent Urgent

Ocular condiitons requiring immediate

treatment

Acute Angle-Closure Glaucoma

Central Retinal Artery Occlusion

Orbital Cellulitis

Cavernous Sinus Thrombosis

Endophthalmitis

Retinal Detachment

Toxic Causes of blindness

Nontraumatic Ocular Emergencies

Acute Dacryocystitis

Acute Dacryoadenitis

Acute Hordeolum

Preseptal cellulitis

Spontaneous subconjunctival hemorrhage

Conjunctivitis

Bacterial corneal ulcer

Viral keratoconjunctivitis

Acute hydrops of the cornea

Hyphema

Uveitis ( iritis & iridocyclitis )

Vitreous hemorrhage

Retinal hemorrhage

Central retinal vein occlusion

Optic neuritis

Ocular Emergencies

Ocular burns and trauma

Ocular Burn

Alkali Burns

Acid Burns

Thermal Burns

Burns Due to Ultraviolet Radiation

Mechanical Trauma to the Eye

Penetrating or Perforating injuries

Blunt Trauma to the Eye, Adnexa,& Orbit

1. Ecchymosis of the Eyelids

2. Lacerations of the Eyelids

3. Orbital hemorrhage

4. Fracture of the Ethmoid bone

5. Blowout Fractures of the Floor of the Orbit

6. Corneal Abrasions

7. Corneal & Conjunctival Foreign Bodies

Ocular Emergencies

Eye Examination

Visual acuity

External Eye : orbit, periorbital skin, eyelids

Confrontation visual fields

Ocular motility

Anterior Segment

Conjunctiva

Cornea

Anterior chamber

Iris

Lens

Pupils : RAPD

Eye Examination

A dilated pupil makes it easier to see the optic

nerve, macula, and retina

- 1% tropicamide ( Mydriacyl )

- 2.5% phenylephrine ( Neo-Synephrine )

PanOptic Ophthalmoscope

Indirect Ophthalmoscope

Fundus Examination

Digital palpation

Schiotz tonometer

Intraocular Pressure Measurement

Ocular Trauma

Closed Globe Open Globe

Burn

Contusion

Laceration Laceration

Penetrating Perforating

Rupture

Causes

• Trauma, Hypertension

• Valsava pressure spikes

• Spontaneous

No treatment

Resolve within 2 weeks

Subconjunctival Hemorrhage

Pain , photophobia ,

FB sensation, tearing

Conjunctival injection,

swollen eyelid

Epithelial staining defect with fluorescein

Corneal Abrasion

Conjunctival Foreign Bodies

Corneal foreign body with rust ring

Rust ring

Corneal Foreign Bodies

Remove the FB under the best magnification

Evert the eyelid to rule out additional FB

Treat resulting corneal abrasion

Referral to ophthalmologist, next day

Residual rust ring

Corneal Foreign Bodies

Disruption of blood vessels in the iris or ciliary body

Blood in anterior chamber

Traumatic Hyphema

Grade Size of Hyphema

0 No layered blood circulating red blood cells only

I Less than 1/3

II 1/3 to 1/2

III 1/2 to less than total

IV Total

Traumatic Hyphema : Classification

Traumatic Hyphema

Elevate the patient’s head

Bed rest

1% atropine one drop 3-4 times daily

1% prednisolone acetate one drop 3-4 times daily

If the globe is intact, measure IOP

Reduce IOP

Ophthalmology consult

Traumatic Hyphema : Management

Rebleeding can occur 3 to 5 days later in 30%

Uncontrolled glaucoma or blood stained cornea

requires anterior chamber “wash out”

Traumatic Hyphema : Management

Sharp or blunt trauma

R/O associated ocular injury

Remove superficial FB

Rule out deeper FB

Give tetanus prophylaxis

Lid Lacerations

Tear lid margin

Full Thickness Lid Lacerations

- Gray line

- Lash line

- Mucocutaneous junction

Laceration of lower eyelid margin Post-operative result following a primary repair

Lid Margin Repair

Refer to ophthalmologist if there are

associated ocular injuries

Lid Lacerations

Ruptured globe

Lacrimal drainage system

Levator aponeurosis

Medial canthal tendon

Tissue loss ( > 1/3 )

Lid Lacerations with tear canaliculi

Canalicular Repair

Penetrating / Ruptured Globe

Corneal or scleral lacerations

Hypotony (not always present)

Severe chemosis & hemorrhage

Intraocular contents may be outside the globe

Limitation of extraocular motility

Shallow anterior chamber

Irregular pupil

Irregular pupil

Penetrating / Ruptured Globe

Ruptured globe caused by golf ball

Penetrating / Ruptured Globe

Penetrating / Ruptured Globe : Management

Stop examination

Shield the eye (do not patch)

Give tetanus prophylaxis

NPO and systemic antibiotics

Do not apply eye ointment or eye drop

Film orbit if IOFB can’t be R/O

Refer immediately to ophthalmologist

Intraocular or Intraorbital Foreign Bodies

Ocular Trauma

Traumatic cataract

Traumatic mydriasis Traumatic lens subluxation

Traumatic lens subluxation

Traumatic Optic Neuropathy :

Classification and Mechanisms

Direct injury

- Penetrating injury from knife, projectile

- Injury from fractured bone

- Avulsion, transection

Indirect injury

- Contusion with transmission of force through bone

- Compression secondary to orbital hemorrhage or

intrasheath hemorrhage

Clinical Features of Traumatic Optic Neuropathy

Most commonly unilateral

May be overlooked in setting of significant

globe or maxillofacial trauma

Reduced visual acuity ( NLP to 20/20 )

Visual field defect : No pathognomonic defect

Normal optic disc with development of optic

atrophy

Medical Management Options

Steroids : Controversial

- Thought to limit free-radical amplification

of the injury response

- Dosages ( low, high, mega)

- May be harmful

Observation : 57% of untreated patients shown

to have 3 lines or more acuity improvement

Surgical Management Options

Lateral canthotomy and cantholysis for orbital

hemorrhage

Surgical decompression of the optic nerve

within its canal

There is no defined standard protocol of

treatment for indirect optic nerve injury .

True ocular emergency

Both acid and alkali burns can be blinding

- Acid burns tend to coagulate proteins, limiting

the depth of penetration.

- Alkali burns can rapidly penetrate the cornea,

causing damage to intraocular structures.

Chemical Ocular Injury

Immediate copious irrigation with a minimum of

1-2 L of saline or until pH is normalized ( 6.8-7.7 )

- Instill a topical anesthetic

- Use eyelid retractor

- Double eversion of the eyelids

Chemical Ocular Injury : Management

Immediate copious irrigation with a minimum of

1-2 L of saline or until pH is normalized ( 7.3-7.7 )

- Instill a topical anesthetic

- Use eyelid retractor

- Double eversion of the eyelids

Chemical Ocular Injury : Management

Ophthalmologists Referral

No corneal involvement

- ATB + steroid eye drop

Chemical Ocular Injury : Classification

Grade I Grade II

Grade III Grade IV

Chemical Ocular Injury : Management

Preservative-free artificial tears

Topical non-preserved steroid

Topical cycloplegic

Topical antibiotics

Oral analgesics

Pressure patch or bandage CL

Antiglaucoma +

Bilateral Alkali Injuries

Chemical Ocular Injury

Chemical Ocular Injury : Management

Corneal Transplantation

Keratoprosthesis

Accidental into the eye can cause the lids to

adhere and adhesive clumps to form on the cornea

Not permanently harmful to the eye

Cyanoacrylates are used occasionally directly on the

cornea to seal corneal perforations.

Cyanoacrylate Glue

Moisten the glue with eye ointment, and remove

as much as can be removed easily without causing

damage to underlying tissue

The glue will loosen and become easier to remove

in a few days.

Cyanoacrylate Glue

Non-traumatic Ocular Emergencies

Reduce the intraocular pressure

O.5% Timolol 1 drop

2-4 % Pilocarpine 1 drop every 15 minutes

20% Mannitol 250-500 ml IV drip

Acetazolamide 500 mg oral

100% Glycerin 1 cc/kg

Consult ophthalmologist

Acute Angle Closure Glaucoma

A 60-year-old woman with acute, painless loss

of vision in the right eye

Visual acuity CF – LP in 90% of cases

Opaque white retina and attenuated vessels

Central Retinal Artery Occlusion

Treatment must be initiated immediately.

Ocular massage

Inhaled carbogen ( 95% O2 and 5% CO2 )

Reduced intraocular pressure

Central Retinal Artery Occlusion

Consult ophthalmologist immediately

Anterior chamber paracentesis

Direct infusion of t-PA or urokinase in the

ophthalmic artery

Broad spectrum intravenous antibiotics

CT scan orbit

Ophthalmology & ENT consultation

Orbital Cellulitis

Subperiosteal abscess

Preseptal Cellulitis

Endophthalmitis

Urgent Neuro-ophthalmology

A 36-year-old-woman with subacute visual loss in

right eye and pain on eye movement

VA 20/200, 20/25 RAPD +ve OD

VF central scotoma OD

Retrobulbar optic neuritis

A 55-year-old man with HT and acute visual loss in RE

VA 20/100, 20/20 RAPD +ve RE

Nonarteritic anterior ischemic optic neuropathy

ESR 10 mm/hr

A 73-year-old woman with acute visual loss of right

eye, headache, anorexia and weight loss

VA 10/200, 20/25 RAPD + ve RE

ESR 94 mm/hr, high level of C - reactive protein

Arteritic anterior ischemic optic neuropathy

Pathology : Giant Cell ( Temporal ) Arteritis

A 35-year-old man with a suspicious of aneurysmal

third nerve palsy

A 40-year-old woman with sudden onset of left

third nerve palsy, visual loss and severe headache

What is the diagnosis?

VA 20/30, LP +ve RAPD LE

References Pisit Preechawat ; Ramathibodi Hospital

Birmingham Eye Trauma Terminology ;

( Kuhn F.Ocular Traumatology Berlin 2008 )

Eye Emergency Manual 2nd Edition ; NSW Deparment of Health

Australin

Manament of Ocular Emergencies 5th Revised Edition ; Raymond

Stein, Harold Stein 2010 Montreal, Quebec Canada

Ocular Emergencies ; American Family Physician Journal Sep 15,2007