neuro/musculoskeletal by diana blum rn bsn metropolitan community college
TRANSCRIPT
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Neuro/Neuro/musculoskeletal musculoskeletal
By Diana Blum RN BSNMetropolitan Community College
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Selective AnatomySelective Anatomy12 cranial nerves 31 spinal nervesNeuron transmits impulses to facilitate
movement or sensationMeninges serve as protection of the
brain and spinal cordBronca’s area in frontal lobe forms
speechHypothalamus regulates water,
appetite, tempCSF: surrounds and cushions brain and
cord
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Mental statusMental statusDoes not decline with ageCaused by drugs or lack of o2 to
the brainAs we age LTM is better then
STM
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functional Assessment functional Assessment Appearance SpeechMotor functionFamily historyEthnicity Diet ADLsRight handed or left handed
◦Brain injury is more pronounced in dominant hemisphere
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Physical assessmentPhysical assessmentOrientationLOCMemory
◦ LTM (DOB)◦ STM (mode of transportation to hospital)◦ Immediate memory (repeat 3 words after 5
minutes)Attention
◦ Serial 7 testLanguage/copying
◦ Follows simple commandsCognition
◦ Current events
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Sensory assessmentSensory assessmentPain and temp
◦Cotton ball vs paper clip◦Cold vs warm
Touch◦Pt closes eyes and you touch hand
etc and then have them touch where you touched
ABNORMAL FINDINGS Propioception-position sense below injury Contralateral- loss of sensation in
opposite side of body affected
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Motor assessmentMotor assessmentHand graspsFoot strengthArm driftCoordinationGaitBalanceReflexesABNORMAL FINDINGS
tremors, weakness, paralysis, jerking muscles
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Rapid assessmentRapid assessmentGlascow coma scale: eye opening,
motor response, and verbal response◦painful stimuli
Supraorbital pressure Sternal rub Mandibular pressure Trapezius squeeze
◦LOC Decortication-hands/arms turned in Decerebration- hands/ arms turned out
◦Pupil assess Response to light
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The GCS is scored between 3 and 15, 3 being the worst
score, and 15 the best. It is composed of three parts:
Best Eye Response, Best Verbal Response, Best Motor Response
When doing a neuro assessment it is important to watch for trends indicating a decreasing LOC.
Keep in mind that when patients have ingested alcohol,
mind altering drugs, have hypoglycemia or shock with a systolic BP <80, the GCS may be invalid.
9 to 12 is a moderate injury 8 or less is a severe brain injury. 7 or less = Coma
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A client has a 5 on the Glasgow Coma Scale. When assessing this client, the nurse would expect what level of consciousness?
Sleepy or drowsy
Stuporous
Fully alert and oriented
Comatose
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Comatose
A score of 7 or less indicates a comatose client. Above that are varying degrees of consciousness.
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Coma: No eye opening, no ability to follow commands, no word verbalizations (3-8)
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Diagnostics Diagnostics Blood cultures to find
infection Xray for fx, erosion, etcPET
◦ Evaluates drug metabolism
◦ Detects alzheimer’s, epilepsy, etc
◦ No caffeine, alcohol, or tobacco 24 hrs before test
◦ NPO 6-12 hours prior◦ No insulin prior◦ Takes 2-3 hrs◦ No special follow-up
Angiography for circulation check◦ NPO for 4-6 hrs prior◦ Preop checklist◦ Remove jewelry and
hairpins◦ Neuro check and vs◦ Empty bladder before◦ After- monitor pulses, cap
refill, color, and vs
MRI◦ Signed consent◦ No food or fluid
restrictions◦ Inform of noise and offer
ear plugs◦ Check for hx of pins,
pacemaker, metal objects Lumbar puncture
◦ Needle in subarachnoid space to obtain CSF for analysis
◦ Signed consent◦ Empty bladder◦ Explain procedure◦ Lie in fetal position for
test◦ Bedrest 4-8 hours post◦ Increase fluid for 24 hours
to prevent spinal headache (3000ml)
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Diagnostics continuedDiagnostics continuedCT
◦ No food 4-6 hrs prior
◦ Fluids okay◦ Remove jewelry
and hairpins◦ Monitor for rx to
dye◦ Monitor I/O
EEG◦ Determines brain activity◦ Determines origin of
seizures◦ Dx of sleep disorders◦ Determines brain death◦ Explain procedure◦ No coffee , tea, or
stimulants◦ May be ordered as speep
deprived◦ Hair should not have
product on it◦ Takes about 1 hour◦ When done remove gel
with acetone
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Diagnostics continuedDiagnostics continuedEMG
◦ Looks at muscle activity
BRAIN SCAN◦ Locates tumors and
aneurysms◦ Explain the test◦ May need consent◦ 2 hour delay so brain
absorbs isotope◦ Must be still for
duration of test◦ 1-2 hour exam◦ f/u is to increase
fluids to promote elimination of isotope
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Traumatic Brain InjuryTraumatic Brain Injury(TBI)(TBI)
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Head Injury Head Injury Classification:Classification:
Severe Head Injury----GCS score of 8 or less
Moderate Head Injury----GCS score of 9 to 12
Mild Head Injury----GCS score of 13 to 15
(Adapted from: Advanced Trauma Life Support: Course for Physicians, American College of Surgeons, 1993).
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Superficial InjuriesSuperficial InjuriesCommonAbrasions“Goose Eggs”Lacerations
◦Scalp is very vascularXray if suspect skull fracture
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Skull FracturesSkull FracturesCategorized according to type
and severityFrequently seen in conjunction
with brain injuriesLinear skull fractures Comminuted skull fracturesBasal skull fracturesPossible associated cranial nerve
deficits
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Open Skull FracturesOpen Skull Fractures
Linear- simple clean break
Depressed - bone pressed in towards tissue
Open -lacerated scalp that creates opening to brain tissue
Comminuted - bone fragments and depresses into brain tissue
Basilar- unique fx at base of skull with CSF leaking though the ear or nose◦ Racoon eyes/Battles sign
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Closed Skull FracturesClosed Skull FracturesClosed- blunt trauma
◦Mild concussion-brief LOC◦Diffuse axonal injury- usually from MVA
May go into coma◦Contusion-bruising of brain
Site of impact (coupe) Opposite side of impact (contrecoupe)
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Intracranial HematomasIntracranial Hematomas
Epidural- bleed b/w skull and dura◦Laceration of artery or vien
Subdural-bleed below dura and arachoid layers ◦Acute, subacute, chronic
Intracerebral-accumulation of blood in brain tissue◦Blunt trauma◦Penetrating wounds ◦Acceleration/deceleration injuries
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http://www.unc.edu/~rowlett/units/scales/glasgow.htm
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Increased Intracranial Increased Intracranial PressurePressure
(ICP)(ICP)
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Increase is caused by an increase in the volume of any of the intracranial components
Drivers of increased ICP◦Hypoxia – triggers the vasodilatory
cascade◦Ischemia in acute brain injury
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Increased ICPIncreased ICPNormal ICP 10-15mmHgNormal increases occur with
coughing, sneezing, defecationLeading cause of death for head
trauma
As ICP increases cerebral perfusion decreases causing tissue hypoxia, decrease serum pH, and increase in CO2
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ICP continuedICP continued
3 types of edema◦Vasogenic: increase in brain tissue
volume ◦Cytotoxic: result of hypoxia◦Interstitial: occurs with brain
swelling
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AssessmentAssessment
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Hydrocephalus Hydrocephalus
abnormal increase in CSF volumeCauses: impaired reabsorption from
subarachnoid hemorrhage or menengitis
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Brain HerniationBrain Herniation
Increased ICP will shift and move brain tissue downward
Central Herniation◦ Downward shift to brainstem
S/S Cheyne stokes , pinpoint pupils, hemodynamic
instability
The most life threatening is Uncal because it causes pressure on the 3rd cranial nerve◦ S/S
Dilated, nonreactive pupils, ptosis, rapidly decreased LOC
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The brainThe brainHeadaches
◦3 MAIN types Migraine-genetic predisposition
s/s: sensitive scalp, anorexia, photophobia, N/V Spasming of arteries at the base of the brain
causing arterial constriction, decrease cerebral blood flow, platelets clump, and serotonin released. Other ateries release prostoglandins that cause swelling and inflammation
With aura- sensation that signals onset Most are without aura Atypical- less common Tx: tylenol, migraine medicine, beta blocker,
yoga, meditation, relaxation, etc.
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Cluster headacheCluster headache one sided headache usually felt deep
around eye. They come and goOnset is associated with relaxation,
napping or REM sleeps/s: ipsilateral (one side) tearing of
the eye, rhinorrhea(runny nose), ptosis(droopy), eyelid edema, facial sweating, miosis (abn. Constriction of eye). There may be bradycardia, pallor, increased temp.
Tx: same as migraine, wear sunglasses, O2 for 15 minutes, surgery
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Tension headacheTension headacheMuscle and shoulder tenderness,
base of skull and forehead pain. Similar s/s to migraines
Classic s/s:N/V, photophobia, phonophobia, aggravates with activity
Tx: NSAIDS,muscle relaxers
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Seizures/EpilepsySeizures/EpilepsySeizure: abnormal sudden,
excessive, uncontrollable electrical d/c of neurons w/in the brain that may result in altered LOC, motor/sensory ability, and/or behavior.◦No known cause but may be from
tumors
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Types of Seizures Tonic-Clonic: lasts 2-5 minutes
◦ Rigidity/stiffening arms/legs and Loss of Consciousness Tonic: loss of consciousness, muscle contraction and
relaxation Clonic: rhythmic jerking, may bite tongue, incontinence
◦ Post seizure lethargy Absence: more common in kids, runs in families, blank
staring, loss of consciousness (resembles daydreaming) Myoclonic: brief jerking or stiffening, symmetric or
assymetric movement Atonic (akinetic): sudden loss of muscle tone, lasts for few
seconds confusion after seizure. Partial: begin in one part of cerebral hemisphere, most
often in adults and are less responsive to medical treatment
Complex Partial: blacks out for 1-3 minutes and automatisms present (lip smacking, picking), amnesia after seizure,temporal lobe most affected
Simple partial: remains conscious, senses unusual sensation, smell, or pain before (déjà vu). Unilateral movement during seizure, and may have tachycardia, flushing, or psychic symptoms
Idopathic: account for ½ of seizures, no known cause
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Causes Metabolic disordersETOH withdrawlElectrolyte
disturbancesHeart diseaseAltered gene function
◦ Defective genes for channels that regulate ions in/out of cell
◦ Myoclonus clients are missing cystain B protein
◦ Etc.
Triggers◦ Physical activity◦ Stress◦ Fatigue◦ Alcohol or caffeine◦ Certain foods
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Epilepsy Def: chronic disorder
characterized by recurrent unprovoked seizure activity.◦May be caused from abnormality in
electrical neuronal activity, abnormal transmitters, or both.
Approximately 2 million people in the USA with epilepsy
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can be defined as abnormal, uncontrolled electrical activity in brain cells.
Nerve cells transmit signals to and from the brain in two ways by ◦(1) altering the concentrations of salts
(sodium, potassium, calcium) within the cell◦(2) releasing chemicals called
neurotransmitters (gamma aminobutyric acid). The change in salt concentration conducts the impulse from one end of the nerve cell to the other.
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At the end, a neurotransmitter is released, which carries the impulse to the next nerve cell.
Neurotransmitters either slow down or stop cell-to-cell communication (called inhibitory neurotransmitters) or stimulate this process (called excitatory neurotransmitters).
Normally, nerve transmission in the brain occurs in an orderly way, allowing a smooth flow of electrical activity. Improper concentration of salts within the cell and overactivity of either type of neurotransmitter can disrupt orderly nerve cell transmission and trigger seizure activity.
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Types of EpilepsyPrimary or idopathic
◦ Not associated with identifiable brain lesionSecondary
◦ Most common cause is brain lesion, tumor or trauma
Status epilepticus◦ Prolonged seizures that last greater than 5
minutes or repeated seizures over the course of thirty minutes. Causes:
Med withdrawl Infection Acute alcohol withdrawl Head trauma Cerebral edema Metabolic disturbances
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CONVULSIVE STATUS EPIEPTICUS IS A NEUROLOGICAL EMERGENCY AND MUST BE TREATED PROMPTLY AND AGGRESSIVELY.◦ Call 911or staff emergency◦ Get airway established if needed by RT,
Anesthesia ◦ O2 as needed◦ Establish large bore IV access◦ Start NS◦ Get ABGs◦ Transfer to ICU
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Education of seizure/epilepsy patientTeach importance of taking meds
as prescribedPromote balanced diet, rest, and
stress reduction techniquesInstruct pt. to keep a seizure
diary to identify causative factors
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Phases of seizuresPreicteral phase: aura present.. The first phase
involves alterations in smell, taste, visual perception, hearing, and emotional state. This is known as an aura, which is actually a small partial seizure that is often followed by a larger event.
Ictus: The seizure.. There are two major types of seizure: partial and generalized. What happens to the person during the seizure depends on where in the brain
the disruption of neural activity occurs. Postictal state: The period in which the brain
recovers from the insult it has experienced. Drowsiness and confusion are commonly experienced during this phase. the period in which the brain recovers from the insult it has experienced
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DiagnosticsEEGCTMRIPET
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TREATMENTNonsurgical
◦ Antiepileptic drugs◦ Seizure precautions
During: Protect the client from
injury Do not force anything
into mouth Turn client to side Loosen restrictive
clothes Do not restrain
After Take vitals Perform neuro checks Keep on side Allow rest document
Teach family◦ Info about disease◦ Info about medication◦ Support groups
available◦ Teach about alcohol
avoidance◦ To investigate state
laws pertaining to driving and working with machinery
◦ Care of seizure client
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Surgical treatmentVagal nerve stimulation
◦ For simple or complex partial seizures◦ Stimulating device is surgically placed in
the left chest wall with a lead wire on the vagus nerve
◦ Activates with hand held magnetCorpuscalostomy
◦ Used for tonic-clonic seizures◦ For those not candidates for other surgical
procedures◦ Sections of the anterior and 2/3 of the
corpus collosum are created to prevent neural discharges
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Nursing diagnosisRisk for fallsIneffective copingRisk for ineffective breathing
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Parkinson’shttp://www.youtube.com/watch?v
=TtM-aP9Gr28
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Alzheimer’s Disease
http://www.youtube.com/watch?v=Z6lA1P2tF0o&feature=related
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Stages Early
◦mild Middle
◦moderate Late
◦severe
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s/ss/sAggressiveRapid mood swingsIncreased confusion at nite
(sundowner’s)Decrease interest in personal
appearanceInappropriate clothing selectionLoss of bowel/bladderDecreased appetite
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diagnosisdiagnosisCBCBMPFolate level checkedThyroid and liver function testTest for syphilisDrug tox screening (OTC)Alcohol screeningCTMRIPETEEG
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Nursing diagnosisNursing diagnosisChronic confusionRisk for injuryDisturbed sleep pattern
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Tx Tx MedsPrevent overstimulationBe consistentReorientPromote independence Bowel/bladder trainingPromote facial recognitionSpeech therapySafety precautionsMinimize agitations
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Spinal Cord InjurySpinal Cord Injury(SCI)(SCI)
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Causes of SCICauses of SCI
Primary◦ Hyperflexion (moved forward excessively)◦ Hyperextension (MVA)◦ Axial loading (blow at top of head causes
shattering)◦ Excessive rotation (turning beyond normal range)◦ Penetrating (knife, bullet)
Secondary◦ Neurogenic shock◦ Vascular insult ◦ Hemorrhage◦ Ischemia◦ Electrolyte imbalance
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TypesTypes
Complete: spinal cord severed and no nerve impulses below level of injury◦Cervical/Thoracic
Incomplete: allow some function and movement below level of injury◦Includes:◦Central cord syndrome ◦Anterior cord syndrome◦Brown-Séquard syndrome
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CompleteComplete
Tetraplegia (quadriplegia): paralysis from neck down◦Loss of bowel and bladder control◦Loss of motor function◦Loss of reflex activity◦Loss of sensation◦Coping issues*Christopher Reeve is example of this
injury*
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IncompleteIncompleteCentral Cord Syndrome
◦Hyperextension damage to center of spinal cord
◦Greater loss of function in upper extremities
Anterior Cord Syndrome◦Cause: Direct injury to anterior spinal cord
or disrupted anterior spinal artery ◦Paralysis, loss of pain and temperature
sensation ◦Light touch, vibration, proprioception
preserved◦Prognosis for recovery is variable
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IncompleteIncompletePosterior cord lesion
◦Damage to posterior white and gray matter
◦Motor function intact, but loss of vibratory sense, crude touch, and position sensation
Brown Sequard syndrome◦Result of penetrating injury that causes
hemisection of spinal cord.◦Motor function , proprioception, vibration,
and deep touch are lost on the same side as injury (ipsilateral)
◦On the other side (contralateral) the sensation of pain, temperature and light touch are affected
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AssessmentAssessment1st -respiratory status2nd - intra-abdominal hemorrhage
(hypotension, tachycardia, weak and thready pulse)
3rd assess motor function◦ C4-5 apply downward pressure while the client shrugs◦ C5-6 apply resistance while client pulls up arms◦ C7 apply resistance while pt straightens flexed arms◦ C8 check hand grasp◦ L2-4 apply resistance while the client lifts legs from bed◦ L5 apply resistance while client dorsiflexes feet◦ S1 apply resistance while client plantar flexes feet
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ComplicationsComplicationsCerebral ischemiaDVT/PEPneumonia/AtelectasisVomiting and AspirationGI stress ulcersConstipationUTIPressure Ulcers
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Autonomic DysreflexiaAutonomic DysreflexiaSevere HTN, bradycardia, sever headache,
nasal stuffiness, and flushing Caused by noxious stimuli like distended bladder or
constipation
Immediate interventions◦ Place in sitting position◦ Call doctor ◦ Loosen tight clothes◦ Check foley tubing if present◦ Check for impaction◦ Check room temp◦ Monitor BP q10-15 minutes◦ Give nitrates or hydralazine per md order
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TreatmentTreatmentImmobilize fx- C-collarProper body alignment
◦ Traction is possibleMonitor VS q4 hr and prnNeuro checks q4 hr and prnMonitor for neurogenic shock
(hypotension and bradycardia)Prepare for possible surgeryTeach skin care, ADLs, wound
prevention techniques, bowel and bladder training, medications, and sexuality
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NRSG DX for SCINRSG DX for SCI
Ineffective tissue perfusion r/t interruption of arterial flow
Ineffective airway clearance r/t SCI
Ineffective breathing pattern r/t SCI
Impaired gas exchange r/t SCI
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HUNTINGTON’S DISEASEHUNTINGTON’S DISEASEFormerly huntington’s choreaHereditaryTransmitted as an autosomal
dominant trait at time of conception25000 people in usa have2 main symptoms are progressive
mental status changes and choreiform movements (rapid, jerky) in the limbs trunk and face
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No known cause No known treatmentOnly prevention is to not have
childrenAntipsychotics and monoamine
depleting agents used to manage movement
TX: PT, OT, speech therapy, meal planning by dietician, HHC, social work to line up community resources
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OsteoporosisOsteoporosis
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Metabolic conditionBone demineralizesEasy to fractureWrist, hip, and vertebrae are
most affected
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Osteopenia: low bone massOsteoclasic: bone resorptionDecreased bone mineral density40-45% loss in women throughout
lifespanTrebecular (Spongy bone) is lost firstThen Cortical (compact bone) lost 2nd Pathophysiology is unknown
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classesclassesGeneralized:involves many structures
◦Primary: more common Post menopausal women Men in 60s-70s
◦secondayRegional: limb involved
◦r/t fx, injury, paralysis, joint inflammation◦Immobilization greater than 8-12 weeks◦Weightless environment (astronauts)
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Health preventionHealth preventionTeach about exerciseTeach about diet rich in calciumTeach about bone healthTeach about safety
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Assessment Assessment Risk for fallsHead to toe assessment
◦Inspect and palpate vertebraeAssess painAssess for fallophobia No definitive lab testsBone scan to check density
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Nursing diagnosisNursing diagnosisRisk for fallsImpaired physical mobilityAcute or chronic pain
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InterventionsInterventionsClient education is #1Hormone replacementsCalcium supplementsMultivitaminsDiet Fall preventionExercisePain managementBraces
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Osteomalacia Osteomalacia Softening of the bone tissueInadequate mineralization of osteoid
(mature compact and spongy bone)Vitamin D deficiency is a key playerSimilar characteristics with
osteoporosisRare in USAPrevent with vitamin D, sun
exposure, and diet
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s/s: early stages : nonspecific◦Muscle weakness◦Bone pain◦Hypophosphatemia◦Hypocalcemia◦Generalized bone tenderness
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Paget’s DiseasePaget’s Disease
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Metabolic disorder of bone remodelingBone deposits that are weak, enlarged, and
disorganizedPhases:
◦ Active increased osteoclasts cause massive bone destruction Osteoclasts are multinuclear
◦ Mixed◦ Inactive 2nd phase
New bone becomes sclerotic and very hard Osteoclasts return to normal amount
2nd most common bone diseaseMost common sites are vertebrae, femur, skull,
sternum, and pelvisUnknown cause
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Assessment Assessment 80% asymptomaticAssess past history of fractures, skin color and
temp, gout, hyperparathyroidism, lethargy, hyperuricemia
Pain that is aching, deep, poor descriptionPain worsens with weight bearing and pressurePain most noticeable at nite or at restArthritis at infected jointsAssess posture, gait, and balanceAssess vision, speech, and swallowing,
hydrocephalus, Neoplasm is the dreaded complication
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Diagnostics Diagnostics Serum alk phosphate
◦Those treated for paget’s need ALP drawn 3-4 times/year
Urine hydroxyproline◦Shows bone collagen turnover and
degree of severityCalcium levels are normal or elevatedIncrease noted in uric acid
◦May initially be thought to be goutX-rays, CT, MRI, bone biopsy
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Treatment Treatment Drugs for pain reliefDrugs to decrease bone resorptionCalcitonin (thyroid hormone)Mithramycin (antineoplastic)BiphosphanatesHeat therapyGentle massageExercisePTDietOsteotomy or joint replacement
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osteomylelitisosteomylelitis
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Inflammatory processIncrease in vascularity and
edemaVessel becomes thrombosed
once inflamedIschemia is next Then necrosisSequestrium forms and retards
bone healing
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Categories Categories Exogenous: infection enters from
outsideEndogenous: infection enters from
insideContiguous: results from skin infection
The most common offending organism is pseudomonas aeruginosa
Staph, salmonella are aslo culprits
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s/s and assessments/s and assessmentPainFeverErythemaHeatSwellingAssess circulationAssess for septic shock
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Treatment Treatment Contact precautionsIV antibx therapyPICC lineUse sterile techniquesPain medsHyperbaric oxygen therapyBone graftsMuscle flapsAmputations
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Bone tumorsBone tumors
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Chondrogenic Chondrogenic Osteochondroma: most common,
benign, tumor…onsets in childhood, grows until skeletal maturity..has a bony stalk like appearance..may become malignant
Chondroma: lesion of mature hyaline cartilage of the hand and feet. Ribs, sternum, spine, and long bones can also be affected…can get at any age or gender
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Osteogenic Osteogenic Osteoid osteoma: pinkish granular
appearance..any bone affected..femur and tibia most affected
Osteoblastoma: affects vertebrae and long bones..large in size and lies in spongy bone..reddish granular appearance
Giant cell tumor: origin unknown..aggressive and extensive..affects women 20s-30s
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Assessment/ tx Assessment/ tx Assess pain Palpate involved areaCT scan and MRI done for
diagnosisInterventions
◦Meds and surgery combination◦Pain meds◦Meds taken with meals or milk◦
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Malignant bone tumorsMalignant bone tumors Primary: originate in bone / 2nd ary: mets to bone
◦ Primary Osteosarcoma: most common
◦ Large lesion, pain and swelling of short duration, warm site, central portion is sclerotic, usually mets to lung in 2 yrs then death
Ewing’s sarcoma: most malignant◦ Pain and swelling, fever, anemia, leukocytosis, pelvis and lower
extremities most affected, any age..but kids and young adults age 20s more
◦ Pelvic yields poor prognosis Chondrosarcoma: dull pain, swelling for long period..
◦ pelvis and femur fore affected
◦ Destroys bone and often calcifies
◦ Affect middle age to elders and more in men
Fibrosarcoma: from fibrous tissue; most common in long bones of legs and mets to lungs◦ Histiocytoma is most malignant type
◦ Local tenderness, with or w/o mass palpated
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Bone Mets Bone Mets Primary tumors are in prostate,
breast, kidney, thyroid, and lungFractures are major problem with
management◦Femur and acetabulum
Primarily affects those under 40
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Assess/ diagnosticsAssess/ diagnosticsAssess pain, swelling, palpate for
massesMonitor vsAssess ADLsAssess support structuresAssess coping skillsCheck ALP levels for elevationCT scanStage tumor
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Nursing diagnosis/tx Nursing diagnosis/tx PainAnticipatory grievingDisturbed body imageFearAnxiety Tx
◦Pain management, chemo, radiation, surgery, dressing changes, be active listener, establish goals, safety precautions, HHC
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Carpal tunnelCarpal tunnel
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Education Education Use ergonomic work stationsTeach client to take regular
breakss/s
◦Parathesia in hands◦Weak pinch, clumsiness, weakness◦Hand activity worsens symtoms◦Swelling may occur
Tx: nsaids, surgery
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Dupuytren’s contracturesDupuytren’s contracturesSlow progressive contractureCommon problemAffects 4th or 5th digit of the hand Trigger finger release surgery
performed to fix
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Disorders of the footDisorders of the foot
Hammertoe: fix with surgeryTarsal tunnel syndrome: ankle version
of carpal tunnelPlantar fasciitis: inflammation of the
plantar fascia located in the arch of the foot◦s/s: pain in arch, pain worsens w/ wt
bearing◦Tx: ice, rest, stretches, strapping, nsaids,
surgeryHallux valgus: aka bunion
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Rheumatoid ArthritisRheumatoid Arthritis
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Most common connective tissue disorders
Most destructive to jointsRA factors looked for in lab Assess sedrate Assess immunoglobinsMRIs performedEMGs are performed to measure
function
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Assessment/ S/S continuedAssessment/ S/S continuedJoint stiffnessSwellingPainFatigueWeight lossReddened jointsDeformity of jointsBaker’s cysts may occur and cause painDry eyes, dry mouth, dry vaginaAssess ADLs, coping, pain
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interventionsinterventions
NsaidsImmunosuppressive drugRest Proper positioningPain managementIceHeart parafin waxPlasmapheresisFish oil tablets
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Lupus Lupus Characteristic sign is butterfly
rashAlso alopecia is commonAutoimmune disorderMay have kidney involvementWomen b/w 15-40African american more than
caucasian
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s/s continueds/s continuedfeverFatigueAnorexiaWeight lossPleural effusionsPericarditisAbd painJaundice
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Assessments and txAssessments and txSteroids, nsaids Monitor wtMonitor social response
◦May become withdrawnSkin biopsy may be done for
diagnosisMonitor CBC for pancytopeniaRheumatology consultAvoid prolonged UV exposure
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GoutGoutType of arthritisUrate crystals deposit in jointsPrimary gout is most commonInflammation is key sign2nd ary is when too much uric acid
in bloodCan affect kidneysMeds to treat Pain management
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Marfan syndromeMarfan syndromeInherited, dominant autosomal traits/s: excessively tall and lanky with
elongated hands and feet, scoliosis, funnel shaped chest, glaucoma
Avg life span is 32 yearsMitral valve prolapse and AAA are
commonTx is palliative and preventitive
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Fibromyalgia Fibromyalgia
Chronic pain syndromePain is burning or gnawingHeadache and jaw pain are also
commonChest pain is commonPain control is the key
◦Muscle relaxers, nsaids, antidepressants
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glaucomaglaucoma
2 types◦Primary open angle: most common ◦Angle closure: less common..emergency
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s/ss/sOpen angle: small cresent
shaped defectAngle closure: visual fields
quickly decrease, severe pain around eye, headache, n/v, halos, blurred vision
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Macular degenerationMacular degenerationCentral vision declinesMild blurring or distortionMore rapid to produce in smokers
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Muscular distrophiesMuscular distrophies9 typesProgression is slow or fastMost common is severe X linked
recessiveDiagnosis is difficultComfort is key Treat symptoms
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