level 1 neurointegration class training manual
TRANSCRIPT
CLEAR MIND CENTER®
CREATING EEG SENSORY SOLUTIONSEQUIPMENT - TRAINING - CLINICAL SET-UP
LEVEL 1NEUROINTEGRATION
CLASS
TRAININGMANUAL
Clear Mind Center5587 Merrimac DriveSarasota Fl 34231
949.222.1020Denise; X1001Tech: X1003
CLEAR MIND CENTER®
CREATING EEG SENSORY SOLUTIONSEQUIPMENT • TRAINING • CLINICAL SETUP
O2O1
Short Term MemorySpatial/Object Retrieval
Vigilance AreaSelective & Sustained
Attentional Area
Sensory andMotor Functions
Personality- Emotional Tonality (anger, sadness)
Categorization & OrganizationVisualization
Auditory Cortex
Visual processingSpatial Sketch Pad, Vigilance
Personality- Excessive Self-concern, Victim MentalityAgnosia, Apraxia, Context Boundaries, Rumination
Visual ProcessingProcedural Memory
Dreaming
F4
C4
T4
P4
O2
Working MemoryVerbal Episodic Retrieval
Facial Recognition, Planning & Problem Solving
Sensory andMotor Functions
Language ComprehensionVerbal Understanding
Wernicke’s Area- Inner VoiceLong Term Memory
Declarative & Episodic ProcessingEvent Sequencing- VisualizationAmygdala/Hippocampal Area
Short Term Memory ProblemsInformation Organization
ProblemsSelf- boundaries
Excessive Thinking
Visual ProcessingProcedural Memory
Dreaming
F3
10-20 Correlations of Function
C3
T3
P3
O1
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Brainwave TrainingUsing The Clear Mind
NeuroIntegrator System
By Richard Soutar, Ph.D.Copyright 2009
What Is Neurofeedback and when is it used?
• Neurofeedback is a method of training brainwaves to alter the structure and function of the brain.
• It is used to help people reduce symptoms of a variety of disorders including ADHD, Depression, Anxiety, TBI, Stroke, Seizure as well many others .
Training Brainwaves
• People can learn to change their brainwaves .
• By practicing on a regular basis they can change the activity level of different areas of the brain.
• The brain can be made stronger and more efficient through training.
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How is it done?• Individuals are hooked up to a
computer using wires and sensors.
• The computer reads their brainwaves.
• When their brainwaves begin to appear properly ordered it feeds back that information to the individual.
• This feedback appears in the form of a game, movie or exercise that tells them when they are training just right.
• Through Operant Conditioning individuals learn to change brain structure and function.
Operant Conditioning
• The individual feels rewarded for his or her training efforts during neurofeedback.
• This reward process is called reinforcement in the psychology of Behaviorism.
• We learn to ride a bicycle in the same manner through the same mechanisms.
Learning Is Permanent
• Once we learn something it becomes a permanent part of our behavior.
• Follow up studies in neurofeedback show that the effects continue for up to 30 years.
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How long does it take?
• Trainees typically come for 20-40 sessions of training.
• Trainees come twice a week or more.
• Each session is 30-45 minutes long.
Training Averages of First Seven Sessions
When do clients begin to feel better?
• Each individual responds differently to neurofeedback. Their sensitivity varies.
• The greater their sensitivity to neurofeedback the more quickly they feel its effects.
• Sensitivity to drugs often predicts sensitivity to neurofeedback.
• Some individuals feel changes in 1 to 5 sessions.
• More typically noticeable changes begin to occur around 15 to 20 sessions.
Pre Train Symptoms
Post Train Symptoms
Changing Structure & Function
• MRI Research shows that EEG Biofeedback changes the structure and function of the brain.
• Functional magnetic resonance imaging investigation of the effects of neurofeedback training on the neural bases of selec tive attention and response inhibition in children with attention-defi cit/hyperactivity disorder.
• Beauregard M , Lévesque J .
• Centre de Recherche en Neuropsychologie et Cognition, Département de Psychologie, Université de Montréal, Montréal, Canada. [email protected]
• Two functional magnetic resonance imaging (fMRI) experiments were undertaken to measure the effect of neurofeedback training (NFT), in AD/HD children, on the neural substrates of selective attention and response inhibition. Twenty unmedicated AD/HD children participated to these experiments. Fifteen children were randomly assigned to the Experimental (EXP) group whereas the other five children were randomly assigned to the Control (CON) group. Only subjects in the EXP group underwent NFT. EXP subjects were trained to enhance the amplitude of the SMR (12-15 Hz) and beta 1 activity (15-18 Hz), and decrease the amplitude of theta activity (4-7 Hz). Subjects from both groups were scanned one week before the beginning of NFT (Time 1) and 1 week after the end of NFT (Time 2), while they performed a "Counting Stroop" task (Experiment 1) and a Go/No-Go task (Experiment 2). At Time 1, in both groups, the Counting Stroop task was associated with significant activation in the left superior parietal lobule. For the Go/No-Go task, no significant activity was detected in the EXP and CON groups. At Time 2, in both groups, the Counting Stroop task was associated with significant activation of the left superior parietal lobule. This time, however, there were significant loci of activation, in the EXP group, in the right ACC, left caudate nucleus, and left substantia nigra. No such activation loci were seen in CON subjects. For the Go/No-Go task, significant loci of activation were noted, in the EXP group, in the right ventrolateral prefrontal cortex, right ACcd, left thalamus, left caudate nucleus, and left substantia nigra. No significant activation of these brain regions was measured in CON subjects. These results suggest that NFT has the capacity to functionally normalize the brain systems mediating selective attention and response inhibition in AD/HD children.
• PMID: 16552626 [PubMed - indexed for MEDLINE]
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Success Rates
• Depression- 90%
• AD/HD- 90%
• OCD- 80%
• Anxiety- 75%
• Bipolar- 60%
• Autism- 30-45%
Brainwaves• Electrical activity recorded
from the brain is referred to as EEG or brainwaves.
• The brain produces up to 30 watts of power to accomplish its routines.
• Brainwaves cycle from positive peaks to negative troughs.
• On average they may go as high as 30-40 microvolts (or millionths of a volt) or as low as 1 microvolt.
Neurons in the cortex generate electrical activity from synaptic interaction.
Synaptic Gap Electrical potential builds prior to neurons firing.
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EEG is from the summated pre and post synaptic potentials in the cortex
Synapses generate electrical fields called dipoles
The EEG: Peak Value vs Peak To Peak
• EEG is a form of alternating current.
• It can be measured in terms of Volts or Power.
• Voltage is measured in terms of how high it swings above and below the 0 value line.
• The volt is defined as the potential differenceacross a conductor when a current of one amperedissipates one watt of power.
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Amplitude vs Power• Amplitude is the peak to peak
measurement of voltage of the EEG.
Power is the computation of energy in that peak to peak voltage.
Frequency
• A cycle is when a brainwave swings from zero to a positive peak, then down to a negative peak, then back to zero.
• One cycle every second is called one cycle per second or one hertz.
• Frequency is the number of cycles that occur in one second.
Frequency Spectrum
• The brain produces most of its activity in the frequencies between one and 24 cycles per second.
• Recent research indicates high frequencies up to 40 cycles per second or 40hz can be important as well.
• This entire range of frequencies is called a frequency spectrum.
• Your equipment will display this spectrum in a series of bar graphs running from low to high. Cycles Per Second
Am
plitu
de
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Component Bands
• The frequency spectrum is divided into component bands.
• The most basic divisions from low to high are delta, theta, alpha and beta.
• Delta is 1-4hz, theta is 4-8hz, alpha is 8-12hz and beta is 13hz and higher.
Component Bands & Function• Delta: Related to sleep and brain stem functions. Also Fascicular
Continuity and neural integration.Can indicate white matter damage.
• Theta: Related to memory, emotion, and emotional brain (Limbic System) functions.
Can indicate cortical lesions or dysfunction.• Alpha: Related to the brain at rest or routine activities.
Can indicate processing incapacities.• Beta: Related to activation and processing in the Cortex of the
brain.Can indicate hyperactivation of cell columns and over-processing.
Function & Location
• The cortex is the source of cognitive processing which appears as beta.
• When it is in stand-by mode it produces alpha.
• The brain stem does most of the housekeeping for the body and the brain and is a major source of delta.
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The Brain: General Orientation
• Frontal LobesST Memory Emotional Valencing
Attention Emotional Inhibition
• Parietal LobesBody Awareness Location
Association Arousal
• Occipital LobesVision Arousal
• Temporal LobesMemory Comprehension
Major Convergence Zone
• CerebellumBalance Motor Sequencing
• Brain StemPrimary Arousal Consciousness
The Limbic System
• The limbic system is below the cortex.
• It is considered the source of emotional activity.
• It is the major source of theta in a healthy brain.
SMR & Motor Function
• When SMR (13-15hz) appears over the sensorimotor strip, the sensory flow from the thalamus to the cortex is reduced (gated) (Sterman & Bowersox, 1981).
• The body is calmed and the somatic system reduces in tone.
• The cortex is alert but not heavily processing.
• The sensorimotor strip is mapped to the body and can be trained locally.
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Delta & The Brain Stem
• The reticular activation system (RAS) adjusts basic arousal in the brain.
• The primary neuromodulator mechanisms are norepinephrine and acetylcholine.
• Resulting vigilance activity is most readily observed at P4 and F4.
• Delta is an indicator of functional continuity.
The Normal Distribution
• In general there is a normal distribution or layering of brainwaves when the eyes are closed.
• Alpha is highest, then theta, then delta, then beta.
• Beta is about one half of alpha. Theta is about 2/3 of alpha.
0
2
4
6
8
10
12
14
16
18
20
Delta
Theta
Alpha
Beta
Delta 4 6 8 8 7
Theta 6 8 10 12 11
Alpha 8 11 14 16 18
Beta 4 6 7 6 5
Fp1 Fz Cz Pz Oz
This trainee is normalizing their delta theta layering (note theta increasing over alpha between 13 and 16 minutes..
The Eyes Open Distribution
• The eyes open distribution is different from eyes closed.• Delta is highest, then theta, then alpha, then beta.• If the distribution is different, then it is abnormal and usually
indicates something is wrong.
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Abnormal Distribution
• When there is a disorder in the brain, the distribution becomes disturbed.
• Neurofeedback is designed to train the brain to a more normal distribution.
• The brain never completely returns to normal but adjusts for a closer approximation.
Note how low this trainee’s alpha (light blue) is at the start of the session. The alpha increases and the alpha to beta ratio (alpha/beta) improves over the training session. Delta and theta come closer to normal as well.
Anxiety Disorder With Low Alpha
Exercising The Brain• The abnormal distribution
reflects problems in brain structure and function.
• Neurofeedback is a method of exercising the brain in order to change its function and eventually its structure over time.
• Neurofeedback is a form of training and learning.
• The brainmaps at the right show the normalization of theta over a 15 session training period.
Pre Map with high theta
Post Map with normal theta
Optimal performance Zone
• The brain has an optimal performance zone.
• This zone is represented as the Normal Range in the figure to the right.
• If the brain operates outside this optimal zone and is too fast or too slow, then problems occur.
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Brain too fast
• The red shaded area shows the front of the brain as being overactive and producing too much beta.
Brain too slow
• The red shaded area show the front of the brain as being underactive (especially the left side) and producing too much alpha.
Instability
• With instabilities the whole brain may shift back and forth between too slow and too fast.
• The shift may be very rapid in a matter of seconds or very slowly over a period of months.
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Training Component BandsWe can train component bands up or down with neurofeedback to adjust the brains distribution.
The client is trying to stay below the threshold line in theta,
And above the threshold line in beta.
Asymmetry• Asymmetry occurs when
one hemisphere has more magnitude or power than another.
• It can also occur between the anterior and posterior regions of the brain.
Beta greater in the right then in the left hemisphere
Dominant Frequency• Dominant frequency is
determined by computing which frequency band in a given component band contains the most power.
• In the Alpha component band of 8-12hz, the peak frequency of a healthy individual is between 9.5 and 10.5hz.
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Distributions Of Disorder
• LD appears as posterior elevated delta.
• ADHD appears as elevated frontal theta.
• Depression appears as elevated central or frontal alpha.
• Anxiety appears as elevated frontal beta often in conjunction with diminished alpha.
• Depression also appears as more slowing on the left, while anxiety appears as increased activity on the right.
Phase is the relationship between waves at two locations.
Coherence is about the consistent relationship of phase over time.
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Coherence
• Coherence is about the phase relationship between two EEG waves over time. If the phase relationship is consistent, then coherence is high. If the phase relationship is erratic, than coherence is low. If two locations have high coherence, then they are considered to be communicating more than if they are low in coherence. Too much coherence is a traffic jam in the brain, while too little may be due to noise or poor connections.
The Brainmap
• The brainmap is much like a weather map in that it provides us with information about what frequencies or component bands are high or low at different locations.
Map Patterns• Here is a typical magnitude
(average amplitude) map of one trainee.
• Each of his component frequency bands, delta, theta, alpha, or beta, is represented by a circle.
• If his component band is normal in a given location it is green. Note C3 in the alpha band is green.
• If his component band is abnormal it will be a different color. Note C4 in the alpha band is red or high.
• Red and yellow indicate high and light blue or dark blue indicate low.
• Note that P4 in beta is light blue or low.
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Asymmetry
• Maps also tells us whether the brain is out of balance front to back or left to right.
• When the brain is disordered it shows problems with this balance.
• This natural balance is called symmetry.
• When there is an abnormal balance it is referred to as asymmetry.
Notice all red on one side is excess and out of balance
Notice that theta is higher in the front than the back
Brain Too Slow
• In depression alpha is higher than normal.
• Alpha is also higher in the left hemisphere than the right.
• Note that the asymmetry section of the map shows this left hemisphere dominance.
Brain Too Fast
• Anxiety appears as low alpha or high beta.
• Beta is high on the right side.
• Anxiety and depression commonly appear in head injury.
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Connectivity• A map can also tell us how
well connected different areas of the brain are compared to a normal level of connectivity.
• This level of connectivity is known as coherence.
• Red indicates too much connectivity usually resulting in lack of flexibility.
• Blue indicates too little connectivity indicating too much flexibility.
• In either case communication between brain locations is poor.
Normal
Abnormal
Dominant Frequency Analysis• A slowed alpha frequency can indicate
slowing of general brain processing due to physiological imbalances or depression.
• Hypothyroid or toxins affecting liver function may result in slowed alpha.
Arousal Level & Disorder Stratification
• Brain Too Fast: BetaAnxiety, OCD, Mania, Worry
• Brain Too Slow: AlphaDepression, Lethargy, Fibromyalgia, Hypothyroid,Toxins, Hepatic Issues, Drug Burnout.
• Brain Very Slow: ThetaADHD, Head Injury, Toxic Encephalopathy, Cortical Damage
• Brain Extremely Slow: DeltaTBI, LD, Dementia, White Matter Damage.
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Pre & Post Maps
• As we train we can periodically remap individuals to see how much they have changed.
• We can also determine if we need to change protocols for future sessions to improve training.
• To the right is an example of a pre and post map. Note the reduction in abnormal theta in particular.
Pre Map Session 1
Post Map Session 15
Common Mode Rejection
• Detects the difference between active and reference electrode.
• Subtracts the difference between active and reference.
• Common mode rejection ratio (CMRR) measures the quality of the subtraction.
• Rejects unwanted signals common to both amplifier inputs.
Differential Amplifier Dynamics
• Site A= 7uv + Site B = 0uv results in 7uv on your training screen.
• Site A= 7uv + Site B = 7uv results in 0uv on your training screen.
• Site A = 7uv + Site B = -7uv results in 14uv on your training screen.
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How do I conduct a session?Overview
• 1. Seat the client in front of the training screen.
• 2. Review how they are feeling and list their present symptoms.
• 3. Select the appropriate protocol for training.
• 4. Hook the trainee up.• 5. Start up the training program.• 6. Record a baseline if necessary.• 7. Train the trainee for the
recommended time period.• 8. Record the results.• 9. Disconnect the trainee.• 10. Display the training results and
encourage the trainee.
Hook the client up.
• Place the ground wire on one ear.
• Place the reference wire on the other ear.
• Be consistent in placing the same wire on the same ear at each session.
• Place the active leads on the designated training locations- such as F3-F4.
• Inspect the quality of the raw EEG to be sure the impedance is correct.
The 10-20 system is a co-ordinate system for the scalp
• Electrode placement is based on the 10-20 system.
• Protocols are described in terms of the 10-20 system.
• The 10-20 system is not based on neuroanatomy.
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Prepping the skin
• Before placing an electrode at a site, the skin must be cleaned of oil and dirt.
• Use an alcohol swab and apply a small amount of Nu-prep (a mild abrasive).
• Gently scrub the area where the electrode is to be applied.
Getting at the scalp
• Be sure to part the hair with the thumb and forefinger.
• You should be able to see the white of the scalp.
• Holding the hair in place with one hand scrub the white of the scalp.
• It is a good idea to hold the electrode lead in the hand scrubbing the scalp.
Applying Paste
• Paste conducts electrical impulses from the skin to the electrode.
• A plastic knife is often used to scoop paste from the jar.
• Apply a pea sized dab of paste to each electrode.
• Be sure to use a generous portion of paste.
• Gently press the electrode to the skin site until it sticks.
• Paste, not metal, should be touching the skin.
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Determining a 10-20 location
• One of the best ways to learn the 10-20 locations is to place an electrocap on a volunteer and use it as a reference to practice placing electrodes on a second volunteer.
Disconnect the Client.
• Remove the electrodes.
• Wipe the electrode sites clean.
• Clean each electrode with the proper solution.
• Provide the trainee with a tissue to wipe their ears.
Eyes Closed: Eye Movement
• This is up and down eye movement.
• It inflates the average amplitude of delta.
• Have clients gently place a finger over each closed eye to monitor and control their eye movement.
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Eyes Closed Artifact 2
• This is left to right eye movement. Notice how the two lines come together.
• Use the same procedure as for the previous artifact.
Amplifier Clipping
• When the signal gets to big from eye movement the amplifiers cut off and generate a square looking wave.
EMG Artifact
• T3-T4 is the most likely location to find muscle artifact.
• A large number of individuals with disorder clench their teeth.
• It is often impossible to stop this unconscious habit.
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Adding Entrainment
• Entrainment is a passive form of EEG Biofeedback.
• Brainwaves are stimulated using light and sound.
• As a standalone technology, it must be used daily.
• Creates a powerful synergy when used with neurofeedback.
Entrainment Equipment
• Entrainment equipment uses glasses that flash light at specific brainwave frequencies into the brain.
• It may also deliver audio tones at the same rate to assist in the entrainment process.
• The NeuroIntegrator combines both neurofeedback and entrainment in one technology.
VE Stimulates The Visual Cortex
• Entrainment glasses flash lights at brainwave frequencies.
• The optic nerve responds and sends impulses to the thalamus.
• The thalamus conveys the information to the visual cortex
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Dual Visual Channels
• The visual pathways for the left and right hemisphere respond with some independence to the light pulses.
• The left visual field excites the right cortex and the right visual field excites the left cortex.
Areas Of Entrainment 1
• A pet scan of areas that are initially activated by VE.
• Note the visual cortex and Lateral Geniculate Nucleus LGN are activated
Default NetworkStructural Core
The Default Network of the Brain Then Resonates With Entrainment
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The Hub SystemThe Cortical Network8 Anatomical Subregions
• Posterior Cingulate• Precuneus• Cuneus• Paracentral Lobule• Isthmus of the Cingulate• Superior Temporal
Sulcus• Inferior Parietal Cortex• Superior Parietal CortexHighest elevated fiber counts & densities
(node degree and strength)
Visual Network
• The visual network is highly complex and distributed throughout the brain.
• Different VE frequencies resonate with different regions of the brain.
VE & Harmonics
• A 10hz photic stimulation will often induce elevations in EEG frequencies with associated harmonics and sub-harmonics.
• These harmonics will often resonate with specific brain regions.
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Baseline vs VEEffect of 10hz Entrainment
Baseline vs VE 2
Note increase in beta due to harmonics.
Client 2 VE Asymmetry Shift
Note Alpha dominance shifts to the right
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Train Without VE vs Train With VE
NFB With vs Without EntrainmentClient CC
• 0-8 min Without VE
• 0-16 min With VE Good Run
• 16-24 min With VE Good Run
• Client takes VE Home
NFB & EntrainmentResponse Patterns
Trial B in each Session 1- 11 is NFB with VE
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Nick NFB vs NFB+VERH alpha increase/Theta decrease
Symptom ChangesPre Post Fibromyalgia
Using Maps to Measure Network Activity
• Magnitude can measure cortical activation.
• Reduced cortical activation indicates reduction of function or damage.
• Magnitude indirectly measures synchrony at the micro level.
Magnitude reflection of damage due to stroke: Note Elevated Delta & Theta
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Lesions & Network Degredation
• Over a century of lesion studies in neurology provide insights to what occurs when network nodes are damaged.
• Provides insights to network characteristics.
• Provides insights to functional connectivity.
Avg 25 Stroke Patients With Speech Articulation Problems
LH Stroke Client- Insular Cortex
Examples of CorrelationsChild Diagnosed With ADHD
Symptom Location Correlation
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Comparisons To Psychological Evaluations
WISC-IV Test Data- Subscale Score
Verbal Comprehension• Similarities 7*• Vocabulary 6*• Comprehension 10Perceptual Reasoning• Block Design 12• Picture Concepts 7*• Matrix Reasoning 12Working Memory• Digit Span 10• Letter-Number Sequence 8*Processing Speed• Coding 17• Symbol Search 8*
Correlate Map Predictions• Auditory Verbal Sequence*• Auditory Tone Processing*• Short Term Verbal Memory• Dialogue Organization
• Short Term Visual Memory• Spatial Sequencing*
• Event Sequence*• Auditory Verbal Sequence*• Procedural Memory*• Problem Solving • Short Term Memory
• Motivation Problems• Attention
Monitoring Dietary ChangesPre Post Maps: Gluten Free Diet
Seizure Disorder: Nine Months On Gluten Free Diet- No Seizures
Using The NeuroMapDatabase Site
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Clients or Staff Log On To The Site
Fills Out History & Personal Info
Fill Out Cognitive Emotional Questionnaire
Fill Out Physical Symptom Questionnaire
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Metabolic Report Output
Predictive Problem Location Map Is Generated
Upload Data and Download Map With Report In Minutes
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Review Discriminants
qEEG Is Automatically Correlated With Cognitive Emotional Questionnaire
Protocols Are Automatically Generated From The Map
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Use The Symptoms ChecklistTo Track Client Changes Over Time
Compare Client ProgressUsing Pre Post Graph Analysis
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nA
lcoh
olis
m O
veru
seA
lcoh
olis
m_O
veru
se_2
CH
.bxd
2-C
hann
elB
EO
Ent
rain
10H
zF3
& F
4D
ecre
ases
soc
ial a
nxie
ty &
"bin
ge" d
rinki
ngA
nxie
ty F
ear
Anx
iety
_Fea
r_1C
H.b
xd1-
Cha
nnel
B I
GE
CE
ntra
in 1
3Hz
Fpo2
Trai
ns th
e rig
ht a
myg
dala
(sto
res
fear
, dep
ress
ion)
Anx
iety
Inso
mni
aA
nxie
ty_I
nsom
nia_
2CH
.bxd
2-C
hann
elB
EC
Ent
rain
10H
zC
z &
Pz
Anx
iety
Cau
sing
Inso
mni
aA
nxie
ty O
bses
sive
Beh
avio
rA
nxie
ty_O
bses
sive
_Beh
avio
r_2C
H.b
xd2-
Cha
nnel
BE
CE
ntra
in 1
0Hz
C3
& C
4A
nxie
ty c
ausi
ng O
bses
sive
beh
avio
r / O
CD
Anx
iety
Pan
ic D
isor
der
Anx
iety
_Pan
ic_D
isor
der_
1CH
.bxd
1-C
hann
elB
EC
Ent
rain
10H
zFp
2A
nxie
ty A
ssoc
. with
Pan
ic D
isor
der
Anx
iety
Soc
ial
Anx
iety
_Soc
ial_
2CH
.bxd
2-C
hann
elB
EO
Sup
pres
s 21
-37
Hz
F3 &
F4
Soc
ial A
nxie
ty /
"Fre
e Fl
oatin
g A
nxie
ty" /
Lan
guag
e pr
oces
sing
Anx
iety
Tra
umat
ic M
emor
yA
nxie
ty_T
raum
atic
_Mem
ory_
2CH
.bxd
2-C
hann
elB
EC
Ent
rain
10H
zT3
& T
4A
nxie
ty A
ssoc
. with
Tra
umat
ic M
emor
yD
epre
ssio
n A
nxie
tyD
epre
ssio
n_A
nxie
ty_2
CH
.bxd
2-C
hann
elB
I G
EC
Ent
rain
13H
zF3
& F
p1W
ith A
nxie
ty /
Rea
ctiv
e D
epre
ssio
n / A
gita
ted
Dep
ress
ion
Dep
ress
ion
Cog
nitiv
eD
epre
ssio
n_C
ogni
tive_
1CH
.bxd
1-C
hann
elY
EC
Ent
rain
17H
zF7
Cog
nitiv
e D
epre
ssio
nD
epre
ssio
n E
ndog
enou
sD
epre
ssio
n_E
ndog
enou
s_2C
H.b
xd2-
Cha
nnel
YE
CE
ntra
in 1
7Hz
C3
& C
4E
ndog
enou
s de
pres
sion
/ "D
epre
ssio
n w
ithou
t a g
ood
reas
on"
Dep
ress
ion
Gen
etic
Dep
ress
ion_
Gen
etic
_1C
H.b
xd1-
Cha
nnel
YE
CE
ntra
in 1
7Hz
Fpo2
Gen
etic
Dep
ress
ion;
invo
lves
righ
t am
ygda
laD
epre
ssio
n R
eact
ive
Dep
ress
ion_
Rea
ctiv
e_2C
H.b
xd2-
Cha
nnel
YE
CE
ntra
in 1
7Hz
F3 &
Fp1
Rea
ctiv
e D
epre
ssio
n, "S
omet
hing
hap
pene
d to
cau
se d
epre
ssio
n an
d an
xiet
y"C
hron
ic F
atig
ue (F
ocus
)C
hron
ic_F
atig
ue_1
CH
.bxd
1-C
hann
elY
EO
Ent
rain
17H
zFz
Focu
s pr
oble
ms
rela
ted
to C
FSC
hron
ic F
atig
ue (M
emor
y)C
hron
ic_F
atig
ue_2
CH
.bxd
2-C
hann
elY
EO
Ent
rain
17H
zT3
& T
4M
emor
y pr
oble
ms
rela
ted
to C
FSFi
brom
yalg
iaFi
brom
yalg
ia_2
CH
.bxd
2-C
hann
elY
GE
OE
ntra
in 1
3Hz
C3
& C
4M
uscl
e P
ain
Inso
mni
a A
ll N
ight
Inso
mni
a_A
ll_N
ight
_1C
H.b
xd1-
Cha
nnel
BE
CE
ntra
in 1
0Hz
Fz"S
leep
ing
All
Nig
ht L
ong"
Inso
mni
a O
nset
Inso
mni
a_O
nset
_1C
H.b
xd1-
Cha
nnel
Y G
EC
Ent
rain
13H
zC
zO
nset
/ "F
allin
g A
slee
p"In
som
nia
Rel
axat
ion
Inso
mni
a_R
elax
atio
n_2C
H.b
xd2-
Cha
nnel
BE
CE
ntra
in 1
0Hz
O1
& O
2H
elps
rela
xatio
n be
fore
goi
ng to
bed
- re
med
iate
insu
ficie
nt a
lpha
Tens
ion
Hea
dach
eTe
nsio
n_H
eada
che_
2CH
.bxd
2-C
hann
elB
IE
CE
ntra
in 1
3Hz
C3
& C
4M
enst
rual
/ P
MS
/ M
uscl
e Te
nsio
n / P
TSD
Mig
rain
e S
ensi
tivity
Mig
rain
e_S
ensi
tivity
_2C
H.b
xd2-
Cha
nnel
BE
CS
uppr
ess
21-3
7 H
zP
3 &
P4
Com
mon
/ C
lass
ical
Mig
rain
e / "
Sen
sitiv
e to
ligh
t & n
oise
" / S
enso
ry S
timul
iM
emor
y - D
ecla
rativ
eM
emor
y_D
ecla
rativ
e_2C
H.b
xd2-
Cha
nnel
YE
OE
ntra
in 1
7Hz
T3 &
T4
Hel
ps d
ecla
rativ
e m
emor
y (N
ames
, Add
ress
es, D
ates
, Pho
ne N
umbe
rs)
Mem
ory
- W
orki
ngM
emor
y_W
orki
ng_1
CH
.bxd
1-C
hann
elY
EO
Ent
rain
17H
zFz
Abs
ent m
inde
dnes
s
Gla
sses
Cod
eB
lue
BG
reen
GIn
digo
.EC
IO
rang
eO
Red
RW
hite
WY
ello
wY
Blu
eGre
en.E
CB
G
Eye
s O
pen
EO
Eye
s C
lose
dE
C
NI2
Bas
ic-S
H13
12/4
/200
8 7:
21 P
MN
I Des
ign-
Aug
08.x
ls
Sess
ion
Des
ign
Cha
nnel
Gla
sses
Freq
uenc
yEy
esPl
acem
ent
Not
esA
DD
Atte
ntio
n (F
ocus
)A
DD
_Atte
ntio
n_2C
H.b
xd2-
Cha
nnel
YE
ntra
in 1
7Hz
EO
FPz
& F
zFo
cus
prob
lem
s w
ithou
t Hyp
erac
tivity
AD
D A
lert
AD
D_A
lert_
2CH
.bxd
2-C
hann
elY
Ent
rain
15H
zE
OFP
z &
Fz
Focu
s pr
oble
ms
- pro
vide
s m
oder
ate
entra
inm
ent
AD
D R
elax
edA
DD
_Rel
axed
_2C
H.b
xd2-
Cha
nnel
YE
ntra
in 1
2Hz
EO
FPz
& F
zFo
cus
prob
lem
s - t
rain
s fo
r a c
alm
focu
sed
stat
eA
DD
Ove
rfocu
sA
DD
_Ove
rfocu
s_1C
H.b
xd1-
Cha
nnel
BS
uppr
ess
21-3
7 H
zE
CFz
AD
D O
verfo
cuse
dA
DD
Ove
rfocu
sA
DD
_Ove
rfocu
s_2C
H.b
xd2-
Cha
nnel
BS
uppr
ess
21-3
7 H
zE
CF3
& F
4A
DD
Ove
rfocu
sed
AD
HD
Foc
usA
DH
D_F
ocus
_2C
H.b
xd2-
Cha
nnel
YE
ntra
in 1
3Hz
EO
FPz
& F
zFo
cus
& In
atte
ntio
nA
DH
D H
yper
activ
ityA
DH
D_H
yper
activ
ity_2
CH
.bxd
2-C
hann
elY
Ent
rain
15H
zE
OC
3 &
C4
Hyp
erac
tivity
, Foc
usA
DH
D Im
puls
ivity
AD
HD
_Im
puls
ivity
_2C
H.b
xd2-
Cha
nnel
YE
ntra
in 1
3Hz
EO
Fp2
& F
4Im
puls
ivity
, Foc
usA
DH
D A
nxie
tyA
DH
D_A
nxie
ty_2
CH
2-C
hann
elB
Sup
pres
s 21
-37
Hz
EO
Fz &
Fp2
Hyp
erac
tivity
, Anx
iety
AD
HD
Dep
ress
ion
AD
HD
_Dep
ress
ion_
2CH
.bxd
2-C
hann
elY
Ent
rain
15H
zE
OFz
& F
3D
epre
ssio
n as
soci
ated
with
AD
HD
Add
ictio
nA
ddic
tion_
1CH
.bxd
1-C
hann
elB
Sup
pres
s 21
-37
Hz
EC
FzD
rug
addi
ctio
n, g
ambl
ing
Add
ictio
nA
ddic
tion_
2CH
.bxd
2-C
hann
elB
Sup
pres
s 21
-37
Hz
EC
F3 &
F4
Dru
g ad
dict
ion,
gam
blin
gA
lcoh
olis
m A
ddic
tion
Alc
ohol
ism
_Add
ictio
n_1C
H.b
xd1-
Cha
nnel
BE
ntra
in 1
0Hz
EC
O2
Chr
onic
alc
ohol
ism
/ ad
dict
ion
Alc
ohol
ism
Ove
ruse
Alc
ohol
ism
_Ove
ruse
_2C
H.b
xd2-
Cha
nnel
BE
ntra
in 1
0Hz
EO
F3 &
F4
Dec
reas
e so
cial
anx
iety
& "b
inge
" drin
king
Ang
erA
nger
_2C
H.b
xd2-
Cha
nnel
BE
ntra
in 1
0Hz
EO
F3 &
F4
"Bad
Tem
per"
Ano
rexi
aA
nore
xia_
2CH
.bxd
2-C
hann
elB
Sup
pres
s 21
-37
Hz
EO
C3
& C
4R
educ
e an
xiet
y ab
out b
ody
imag
eA
nxie
ty F
ear
Anx
iety
_Fea
r_1C
H.b
xd1-
Cha
nnel
BE
ntra
in 1
3Hz
EC
Fpo2
Anx
iety
Ass
oc. w
ith fe
arA
nxie
ty In
som
nia
Anx
iety
_Ins
omni
a_1C
H.b
xd1-
Cha
nnel
BE
ntra
in 1
0Hz
EC
Fp2
Anx
iety
Cau
sing
Inso
mni
aA
nxie
ty In
som
nia
Anx
iety
_Ins
omni
a_2C
H.b
xd2-
Cha
nnel
BE
ntra
in 1
0Hz
EC
Cz
& P
zA
nxie
ty C
ausi
ng In
som
nia
Anx
iety
Lea
rnin
g D
isor
der
Anx
iety
_Lea
rnin
g_D
isor
der_
2CH
.bxd
2-C
hann
elB
Sup
pres
s 21
-37
Hz
EO
Fp1
& F
p2A
nxie
ty A
ssoc
. with
Lea
rnin
g D
isor
ders
Anx
iety
Obs
essi
ve B
ehav
ior
Anx
iety
_Obs
essi
ve_B
ehav
ior_
1CH
.bxd
1-C
hann
elB
Ent
rain
10H
zE
CFz
Anx
iety
cau
sing
Obs
essi
ve b
ehav
ior
Anx
iety
Obs
essi
ve B
ehav
ior
Anx
iety
_Obs
essi
ve_B
ehav
ior_
2CH
.bxd
2-C
hann
elB
Ent
rain
10H
zE
CC
3 &
C4
Anx
iety
cau
sing
Obs
essi
ve b
ehav
ior
Anx
iety
Obs
essi
ve B
ehav
ior
Anx
iety
_Obs
essi
ve_B
ehav
ior_
2CH
.bxd
2-C
hann
elB
Ent
rain
10H
zE
CC
z &
Pz
Anx
iety
cau
sing
Obs
essi
ve b
ehav
ior
Anx
iety
PTS
DA
nxie
ty_P
TSD
_1C
H.b
xd1-
Cha
nnel
BE
ntra
in 1
0Hz
EC
Fpo2
Anx
iety
Ass
oc. w
ith P
TSD
Anx
iety
Pan
ic D
isor
der
Anx
iety
_Pan
ic_D
isor
der_
1CH
.bxd
1-C
hann
elB
Sup
pres
s 21
-37
Hz
EC
Fp2
Anx
iety
Ass
oc. w
ith P
anic
Dis
orde
rA
nxie
ty S
ocia
lA
nxie
ty_S
ocia
l_1C
H.b
xd1-
Cha
nnel
B B
GS
uppr
ess
21-3
7 H
zE
OFz
Soc
ial A
nxie
ty /
"Fre
e Fl
oatin
g A
nxie
ty"
Anx
iety
Soc
ial
Anx
iety
_Soc
ial_
2CH
.bxd
2-C
hann
elB
BG
Sup
pres
s 21
-37
Hz
EO
F3 &
F4
Soc
ial A
nxie
ty /
"Fre
e Fl
oatin
g A
nxie
ty"
Anx
iety
Tra
umat
ic M
emor
yA
nxie
ty_T
raum
atic
_Mem
ory_
2CH
.bxd
2-C
hann
elB
Ent
rain
10H
zE
CT3
& T
4A
nxie
ty A
ssoc
. with
Tra
umat
ic M
emor
yA
sper
gers
(Iso
late
d)A
sper
gers
_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EO
T6D
iffic
ulty
Und
erst
andi
ng O
ther
s E
mot
ions
Aud
itory
Pro
cess
ing
Aud
itory
_Pro
cess
ing_
2CH
.bxd
2-C
hann
elY
Ent
rain
17H
zE
OT5
& T
6D
istra
cted
Eas
ily b
y A
udito
ry s
timul
iA
utis
m E
xpre
ssio
n E
mot
ions
Aut
ism
_Exp
ress
ion_
Em
otio
ns_1
CH
.bxd
1-C
hann
elY
Ent
rain
17H
zE
OF8
Aut
ism
Spe
ctru
m /
Exp
ress
ing
own
Em
otio
nsA
utis
m E
xpre
ssio
n S
peec
hA
utis
m_E
xpre
ssio
n_S
peec
h_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EO
F7A
utis
m S
pect
rum
/ E
xpre
ssin
g id
eas,
Spe
akin
gA
utis
m U
nder
stan
d O
ther
sA
utis
m_U
nder
stan
d_O
ther
s_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EO
T6A
utis
m S
pect
rum
/ U
nder
stan
ding
Oth
ers
Em
otio
nsA
utis
m U
nder
stan
ding
Spe
ech
Aut
ism
_Und
erst
and_
Spe
ech_
1CH
.bxd
1-C
hann
elY
Ent
rain
17H
zE
OT5
Aut
ism
Spe
ctru
m /
Und
erst
andi
ng S
peec
hB
alan
ce S
wal
low
ing
Bal
ance
_Sw
allo
win
g_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EO
Oz
Bal
ance
and
/or S
wal
low
ing
Pro
blem
sB
rain
Brig
hten
erB
rain
Brig
hten
er_2
CH
.bxd
2-C
hann
elY
Ent
rain
15H
zE
OC
3 &
C4
Bra
in B
right
ener
Sha
rpen
s m
emor
y an
d co
ngni
tion
Chr
onic
Fat
igue
Chr
onic
_Fat
igue
_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EO
FzC
ause
d by
Ani
exty
or C
hron
ic in
fect
ions
/ to
xins
Chr
onic
Fat
igue
Chr
onic
_Fat
igue
_2C
H.b
xd2-
Cha
nnel
YE
ntra
in 1
7Hz
EO
T3 &
T4
Cau
sed
by A
nxie
ty a
nd C
hron
ic s
tress
Chr
onic
Pai
nC
hron
ic_P
ain_
1CH
.bxd
1-C
hann
elB
Sup
pres
s 21
-37
Hz
EC
Cz
With
Anx
iety
/ "T
rain
s th
e P
oste
rior C
ingu
late
"C
lum
sy L
eft H
and
Clu
msy
_Lef
t_H
and_
1CH
.bxd
1-C
hann
elY
Ent
rain
17H
zE
OC
4"P
oor G
olf S
kills
"C
lum
sy R
ight
Han
dC
lum
sy_R
ight
_Han
d_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EO
C3
Incr
ease
s de
xter
ityC
ompr
ehen
sion
Und
erst
andi
ngC
ompr
ehen
sion
_Und
erst
andi
ng_1
CH
.bxd
1-C
hann
elY
Ent
rain
17H
zE
OP
3"U
nder
stan
ding
wha
t it m
eans
"C
onfid
ence
Pro
blem
sC
onfid
ence
_Pro
blem
s_2C
H.b
xd2-
Cha
nnel
YE
ntra
in 1
7Hz
EO
F3 &
F4
Lack
of C
onfid
ence
Dem
entia
Dem
entia
_2C
H.b
xd2-
Cha
nnel
YE
ntra
in 1
7Hz
EO
P3
& P
4C
hron
ic /
Alz
heim
ers
type
Dep
ress
ion
Anx
iety
Dep
ress
ion_
Anx
iety
_2C
H.b
xd2-
Cha
nnel
B I
GE
ntra
in 1
3Hz
EC
F3 &
Fp1
With
Anx
iety
/ R
eact
ive
Dep
ress
ion
/ Agi
tate
d D
epre
ssio
nD
epre
ssio
n B
iPol
arD
epre
ssio
n_B
ipol
ar_1
CH
.bxd
1-C
hann
elB
IE
ntra
in 1
3Hz
EC
Fpo2
Trai
ns th
e rig
ht a
myg
dala
(sto
res
fear
, dep
ress
ion)
- M
ixed
Dep
ress
ion/
Anx
iety
Dep
ress
ion
BiP
olar
Dep
ress
ion_
Bip
olar
_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
5Hz
EC
Fpo2
Trai
ns th
e rig
ht a
myg
dala
(sto
res
fear
, dep
ress
ion)
- D
eepe
r Dep
ress
ion
Dep
ress
ion
Cog
nitiv
eD
epre
ssio
n_C
ogni
tive_
1CH
.bxd
1-C
hann
elY
Ent
rain
17H
zE
CF7
Cog
nitiv
e D
epre
ssio
nD
epre
ssio
n E
ndog
enou
sD
epre
ssio
n_E
ndog
enou
s_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EC
F8E
ndog
enou
s de
pres
sion
/ "D
epre
ssio
n w
ithou
t a g
ood
reas
on"
Dep
ress
ion
End
ogen
ous
Dep
ress
ion_
End
ogen
ous_
2CH
.bxd
2-C
hann
elY
Ent
rain
17H
zE
CC
3 &
C4
End
ogen
ous
depr
essi
on /
"Dep
ress
ion
with
out a
goo
d re
ason
"D
epre
ssio
n G
enet
icD
epre
ssio
n_G
enet
ic_1
CH
.bxd
1-C
hann
elY
Ent
rain
17H
zE
CFp
o2G
enet
ic D
epre
ssio
n; T
rain
s th
e rig
ht a
myg
dala
Dep
ress
ion
Man
iaD
epre
ssio
n_M
ania
_2C
H.b
xd2-
Cha
nnel
B I
Ent
rain
12H
z E
CC
z &
Fz
BiP
olar
- M
anic
Pha
seD
epre
ssio
n P
TSD
Dep
ress
ion_
PTS
D_1
CH
.bxd
1-C
hann
elY
Ent
rain
17H
zE
CFp
o2Tr
ains
the
right
am
ygda
la (s
tore
s fe
ar, d
epre
ssio
n)D
epre
ssio
n R
eact
ive
Dep
ress
ion_
Rea
ctiv
e_2C
H.b
xd2-
Cha
nnel
YE
ntra
in 1
7Hz
EC
F3 &
Fp1
Rea
ctiv
e D
epre
ssio
n, "S
omet
hing
hap
pene
d to
cau
se d
epre
ssio
n an
d an
xiet
y"D
iffic
ulty
Initi
atin
g M
ovem
ent
Diff
icul
ty_I
nitia
ting_
Mov
emen
t_2C
H.b
xd2-
Cha
nnel
BS
uppr
ess
21-3
7 H
zE
OP
3 &
P4
Par
kins
onia
n /
Aki
nesi
a "D
iffic
ulty
Initi
atin
g M
ovem
ent"
Dys
lexi
aD
ysle
xia_
1CH
.bxd
1-C
hann
elY
Ent
rain
17H
zE
O1/
2 B
etw
een
P3
& T
5A
ngul
ar G
yrus
Em
otio
nal C
ontro
lE
mot
iona
l_C
ontro
l_P
robl
ems_
1CH
.bxd
1-C
hann
elY
Ent
rain
17H
zE
OF8
Ove
r rea
ct o
r und
er re
act
Epi
sodi
c D
ysco
ntro
lE
piso
dic_
Dys
cont
rol_
2CH
.bxd
2-C
hann
elY
Ent
rain
17H
zE
OT3
& T
4M
ay o
ver r
eact
, und
er re
act o
r hav
e m
eltd
owns
Exc
essi
ve D
aytim
e S
leep
ines
sE
xces
sive
_Day
time_
Sle
epin
ess_
1CH
.bxd
1-C
hann
elY
Ent
rain
17H
zE
OFp
2M
ay n
eed
to a
ddre
ss in
som
nia
Exc
essi
ve T
alki
ngE
xces
sive
_Tal
king
_1C
H.b
xd1-
Cha
nnel
BS
uppr
ess
21-3
7 H
zE
OF7
Hyp
erve
rbos
ityFe
arFe
ar_1
CH
.bxd
1-C
hann
elB
Ent
rain
13H
zE
CFp
o2Tr
ains
the
right
am
ygda
la (s
tore
s fe
ar, d
epre
ssio
n)Fi
brom
yalg
iaFi
brom
yalg
ia_2
CH
.bxd
2-C
hann
elY
GE
ntra
in 1
3Hz
EO
C3
& C
4M
uscl
e P
ain
Flas
hbac
ksFl
ashb
acks
_2C
H.b
xd2-
Cha
nnel
BS
uppr
ess
21-3
7 H
zE
CF3
& F
4A
ssoc
. with
PTS
DH
andw
ritin
g P
robl
ems
Han
dwrit
ing_
Pro
blem
s_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EO
C3
Impr
oves
Leg
ibili
tyH
earin
g P
robl
ems
Hea
ring_
Pro
blem
s_2C
H.b
xd2-
Cha
nnel
YE
ntra
in 1
7Hz
EO
T3 &
T4
Impr
oves
Acu
ityH
yper
Em
otio
nalit
yH
yper
_Em
otio
nalit
y_1C
H.b
xd1-
Cha
nnel
BS
uppr
ess
21-3
7 H
zE
CF8
Exc
essi
ve e
mot
iona
lity
IBS
Anx
iety
IBS
_Anx
iety
_2C
H.b
xd2-
Cha
nnel
BS
uppr
ess
21-3
7 H
zE
CP
3 &
P4
For u
nder
lyin
g A
nxie
ty
NI2
Des
ign-
SH
14
12/4
/200
8 7:
21 P
MN
I Des
ign-
Aug
08.x
ls
Sess
ion
Des
ign
Cha
nnel
Gla
sses
Freq
uenc
yEy
esPl
acem
ent
Not
esIB
S D
epre
ssio
nIB
S_D
epre
ssio
n_1C
H.b
xd1-
Cha
nnel
I YE
ntra
in 1
5Hz
EC
Fpo2
For u
nder
lyin
g D
epre
ssio
nIn
cont
inen
ceIn
cont
inen
ce_1
CH
.bxd
1-C
hann
elY
Ent
rain
17H
zE
OO
zIn
cont
inen
ce /
Bed
wet
ting
Inso
mni
a A
ll N
ight
Inso
mni
a_A
ll_N
ight
_1C
H.b
xd1-
Cha
nnel
BE
ntra
in 1
0Hz
EC
Fz"S
leep
ing
All
Nig
ht L
ong"
Inso
mni
a A
ll N
ight
Inso
mni
a_A
ll_N
ight
_2C
H.b
xd2-
Cha
nnel
BE
ntra
in 1
0Hz
EC
F3 &
F4
"Sle
epin
g A
ll N
ight
Lon
g"In
som
nia
Ons
etIn
som
nia_
Ons
et_1
CH
.bxd
1-C
hann
elY
Ent
rain
13H
zE
CC
zO
nset
/ "F
allin
g A
slee
p"In
som
nia
Ons
etIn
som
nia_
Ons
et_2
CH
.bxd
2-C
hann
elY
Ent
rain
13H
zE
CC
3 &
C4
Ons
et /
"Fal
ling
Asl
eep"
Inso
mni
a R
elax
atio
nIn
som
nia_
Rel
axat
ion_
2CH
.bxd
2-C
hann
elB
Ent
rain
10H
zE
C01
& O
2H
elps
rela
xatio
n be
fore
goi
ng to
bed
- re
med
iate
insu
ficie
nt a
lpha
Mat
h D
iffic
ulty
Mat
h_D
iffic
ulty
_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EO
P4
Mat
h P
robl
ems
- esp
ecia
lly w
ord
prob
lem
sM
elan
chol
iaM
elan
chol
ia_2
CH
.bxd
2-C
hann
elY
Ent
rain
17H
zE
OT3
& T
4Tr
ains
the
ante
rior i
nsul
a on
eac
h si
de.
Mem
ory
Dec
lara
tive
Mem
ory_
Dec
lara
tive_
2CH
.bxd
2-C
hann
elY
Ent
rain
17H
zE
OT3
& T
4H
elps
dec
lara
tive
mem
ory
(Nam
es, A
ddre
sses
, Dat
es)
Mem
ory
Wor
king
Mem
ory_
Wor
king
_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EO
FzA
bsen
t min
dedn
ess
Mig
rain
e S
ensi
tivity
Mig
rain
e_S
ensi
tivity
_2C
H.b
xd2-
Cha
nnel
B I
Sup
pres
s 21
-37
Hz
EC
P3
& P
4C
omm
on /
Cla
ssic
al M
igra
ine
/ "S
ensi
tive
to li
ght &
noi
se"
Min
d C
hatte
rM
ind_
Cha
tter_
1CH
.bxd
1-C
hann
elB
Ent
rain
10H
zE
CT4
or C
z"G
olf C
ritic
", Le
arn
to "J
ust d
o it"
Mot
ility
Diff
icul
tyM
otili
ty_D
iffic
ulty
_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EO
Cz
Run
ning
, Kic
king
, Hop
ping
etc
.M
otiv
atio
nM
otiv
atio
n_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EO
T6P
rocr
astin
atio
n / L
ack
of d
rive
Mus
cle
Cra
mps
Mus
cle_
Cra
mps
_1C
H.b
xd1-
Cha
nnel
BE
ntra
in 1
2Hz
EC
Cz
Pro
mot
es re
laxa
tion
Mus
cle
Cra
mps
Mus
cle_
Cra
mps
_2C
H.b
xd2-
Cha
nnel
BE
ntra
in 1
2Hz
EC
C3
& C
4P
rom
otes
rela
xatio
nN
ight
mar
esN
ight
mar
es_2
CH
.bxd
2-C
hann
elB
Sup
pres
s 21
-37
Hz
EC
C3
& C
4A
ssoc
. with
Anx
iety
dur
ing
slee
pN
umbn
ess
(R,L
)N
umbn
ess_
1CH
.bxd
1-C
hann
elY
Ent
rain
17H
zE
O(L
eft:
P4;
Rig
ht: P
3)C
heck
for p
erip
hera
l ner
ve p
robl
emO
CD
OC
D_1
CH
.bxd
1-C
hann
elB
Sup
pres
s 21
-37
Hz
EC
Cz
Rep
etiti
ve B
ehav
ior /
Rel
ieve
s A
nxie
tyO
CD
OC
D_2
CH
.bxd
2-C
hann
elB
Sup
pres
s 21
-37
Hz
EC
C3
& C
4R
epet
itive
Beh
avio
r / R
elie
ves
Anx
iety
OD
DO
DD
_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EO
Oz
Wan
ts to
do
the
oppo
site
Opt
imiz
erO
ptim
izer
.bxd
2-C
hann
elY
Wid
eban
dE
OFp
1 &
Fp2
Gen
eral
Ent
rain
men
tO
vere
atin
gO
vere
atin
g_2C
H.b
xd2-
Cha
nnel
YE
ntra
in 1
7Hz
EC
P3
& P
4C
heck
for p
estic
ide
expo
sure
/ to
xici
ty in
hyp
otha
lam
usP
ain
Per
cept
ion
Pai
n_P
erce
ptio
n_1C
H1-
Cha
nnel
BE
ntra
in 1
0Hz
EC
F4P
erce
ptio
n of
pai
nP
erfo
rman
ce A
nxie
tyP
erfo
rman
ce_A
nxie
ty_2
CH
.bxd
2-C
hann
elB
Sup
pres
s 21
-37
Hz
EO
C3
& C
4P
eak
Per
form
ance
trai
ning
, with
anx
iety
Per
form
ance
Foc
usP
erfo
rman
ce_F
ocus
_2C
H.b
xd2-
Cha
nnel
YE
ntra
in 1
5Hz
EO
FPz
& F
zP
eak
Per
form
ance
trai
ning
, for
focu
sP
ick
Fron
tal D
emen
tiaP
ick_
Fron
tal_
Dem
entia
_2C
H.b
xd2-
Cha
nnel
YE
ntra
in 1
7Hz
EC
F3 &
F4
Pre
vent
s pr
ogre
ssio
nP
oor J
udge
men
tP
oor_
Judg
emen
t_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
5Hz
EO
Fp2
Hyp
erac
tivity
, Im
puls
ivity
PTS
DP
TSD
_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EC
Fpo2
May
nee
d m
ore
than
10
sess
ions
Rea
ctiv
e A
ttach
men
t Dis
orde
rR
eact
ive_
Atta
chm
ent_
Dis
orde
r_2C
H.b
xd2-
Cha
nnel
YE
ntra
in 1
7Hz
EC
F3 &
F4
"Neg
lect
ed In
fant
s"R
estle
ss L
egs
Res
tless
_Leg
s_2C
H.b
xd2-
Cha
nnel
BS
uppr
ess
21-3
7 H
zE
C(C
3 &
C4)
or (
F3 &
F4)
Can
't R
elax
or s
leep
Rew
ard
Def
icie
ncy
Rew
ard_
Def
icie
ncy_
2CH
.bxd
2-C
hann
elY
Ent
rain
17H
zE
CF3
& F
4C
omm
on in
alc
holic
s, P
TSD
, and
oth
er a
ddic
tions
Rew
ard
Def
icie
ncy
Rew
ard_
Def
icie
ncy_
1CH
.bxd
1-C
hann
elY
Ent
rain
17H
zE
CFp
o2C
omm
on in
alc
holic
s, P
TSD
, and
oth
er a
ddic
tions
Rum
inat
ions
Rum
inat
ion_
2CH
.bxd
2-C
hann
elB
Sup
pres
s 21
-37
Hz
EC
(T3
& T
4) o
r (P
z &
Oz)
"Una
ble
to s
hut m
ind
off"
Sen
sory
Inte
grat
ion
Dis
orde
rS
enso
ry_I
nteg
ratio
n_D
isor
der_
1CH
.bxd
1-C
hann
elY
Ent
rain
17H
zE
OP
z"H
yper
sens
itive
to li
ght,
soun
d, to
uch"
Sen
sory
Inte
grat
ion
Dis
orde
rS
enso
ry_I
nteg
ratio
n_D
isor
der_
2CH
.bxd
2-C
hann
elY
Ent
rain
17H
zE
OP
3 &
P4
"Hyp
erse
nsiti
ve to
ligh
t, so
und,
touc
h"S
low
Cog
nitiv
e P
roce
ssin
gS
low
_Cog
nitiv
e_P
roce
ssin
g_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EO
Pz
Unc
omm
on to
be
only
Pro
blem
Sno
ring
Sno
ring_
1CH
.bxd
1-C
hann
elY
Ent
rain
17H
zE
CFp
zH
elps
with
sle
ep a
pnia
/ In
crea
ses
tone
in p
hary
ngea
l mus
cles
Spe
lling
Spe
lling
_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EO
1" B
elow
T5
Dis
tinct
from
e ot
her l
earn
ing
diffi
culti
esS
tutte
rS
tutte
r_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EO
F7"V
erba
l Flu
ency
"Te
nsio
n H
eada
che
Tens
ion_
Hea
dach
e_2C
H.b
xd2-
Cha
nnel
B I
Ent
rain
13H
zE
CC
3 &
C4
Tens
ion
Hea
dach
eTI
C D
isor
der
TIC
Dis
orde
r_2C
H.b
xd2-
Cha
nnel
BS
uppr
ess
21-3
7 H
zE
OC
3 &
C4
Tour
ette
s or
Tic
Dis
orde
rTi
nnitu
s C
ant S
upre
ss S
ound
Tinn
itis_
Can
t_S
upre
ss_S
ound
_2C
H.b
xd2-
Cha
nnel
YE
ntra
in 1
7Hz
EO
T3 &
T4
"Can
't su
ppre
ss s
ound
" / In
ner e
ar o
rigin
/ S
timul
ate
that
par
t of t
he b
rain
Tinn
itus
Anx
iety
Tinn
itis_
Anx
iety
_2C
H.b
xd2-
Cha
nnel
BS
uppr
ess
21-3
7 H
zE
OT3
& T
4"C
an't
supp
ress
sou
nd" /
Ass
ocia
ted
with
Anx
iety
Tinn
itus
Hyp
erS
ensi
tive
Tinn
itis_
Hyp
erse
nsiti
ve_2
CH
.bxd
2-C
hann
elB
Sup
pres
s 21
-37
Hz
EO
T3 &
T4
"Hyp
erse
nsiti
ve to
sou
nd" /
Bra
in o
rigin
(e.g
Afte
r acc
iden
t)Tr
emor
- In
tent
ion
Cer
ebel
lar (
R,L
,GTr
emor
_Int
entio
n_C
ereb
ella
r_1C
H.b
xd1-
Cha
nnel
YE
ntra
in 1
7Hz
EO
(Lef
t: 02
;Rig
ht: 0
1;G
ait:
"Mis
s ob
ject
s yo
u ar
e try
ing
to to
uch"
Trem
or R
estin
g Tr
emor
Trem
or_R
estin
g_2C
H.b
xd2-
Cha
nnel
BS
uppr
ess
21-3
7 H
zE
OC
3 &
C4
Par
kiso
nian
- R
estin
g tre
mor
Ver
bal U
nder
stan
ding
Ver
bal_
Und
erst
andi
ng_1
CH
.bxd
1-C
hann
elY
Ent
rain
17H
zE
OT5
"Wha
t did
the
pers
on o
r boo
k sa
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Recommended Reading for Neurofeedback
Doing Neurofeedback, A Condensed and Comprehensive Guide to the Practice of NFB
Dr. Richard Soutar, PhD
Published by the author, updated 2007
Getting Started with Neurofeedback
(an introduction to theory and practice – useful reference for clinical approaches)
John N. Demos
Norton (2005)
Awakening the Mind – A Guide to Mastering the Power of Your Brain Waves
(hands-on tips for self-mastery; useful meditations and imagery for use with patients)
Anna Wise
Tarcher-Penguin (2002)
A Symphony in the Brain – The Evolution of The New Brain Wave Biofeedback
(must have to understand history of the development of NFB; part of the story)
Jim Robbins
Grove Press (2000)
A Guide to Neurofeedback
(great reference handbook-excellent neuroanatomy diagrams, clinical applications)
Thompson & Thompson
Published by The Association for Applied Psychophysiology and Biofeedback (2003)
Handbook of Neurofeedback – Dynamics and Clinical Applications
(theoretical, but excellent resource with cited references)
James R. Evans, PhD
Haworth Medical Press (2007)
Change Your Brain, Change Your Life
The Breakthrough Program for Conquering Anxiety, Depression, Obsessiveness, Anger and
Impulsiveness
Daniel G. Amen, MD
Three Rivers Press, NY (2004)
Power Up Your Brain (Dr. Perlmutter is a solid reference for nutritional concerns; discusses the spiritual side of brain fitness)
The NeuroScience of Enlightenment
Dr. David Perlmutter & Alberto Villoldo
Hay House (2011)
NeuroIntegration Intake Form
PERSONAL INFORMATION
Name Address
Date of birth
Age
/ /_
years City State Zip Email address
Gender M F
Home Phone Work Phone Occupation
Cell Phone Fax
Tell us more about your needs and desires regarding brain health.
How can we help? What are you hoping to address or achieve through our NeuroIntegration Program?
HEALTH INFORMATION
1. OVERALL HEALTH
On a scale of 1-10, how would you rate your current health? 1 2 3 4 5 6 7 8 9 10 (1 being the worst, 5 being average, 10 being the best)
2. SLEEP
Rate the quality of sleep you usually get in the past month. 1 2 3 4 5 6 7 8 9 10
At what time do you go to bed?
At what time do you rise in the morning? am/pm
am/pm
Are you able to sleep through the night? YES NO If NO, please describe:
Are you able to fall asleep easily most nights? YES NO If NO, please describe:
Do you wake refreshed? YES NO If YES, please describe any exceptions:
3. HEAD or NECK INJURY
Have you ever injured your head or neck? YES NO Ever had a concussion? YES NO If yes, have you suffered more than one concussion? YES NO Have you ever been in an auto, motorcycle or bicycle accident? YES NO Have you ever had a traumatic brain injury? YES NO Are you currently receiving care for this/these injuries? YES NO
Please describe your head or neck injuries using the reverse side of this page, thinking back over the years. Please consider the childhood and teen years, as well as adulthood, including home life, sports, accidents, slips/falls, etc.
4. CHRONIC HEALTH PROBLEMS?
Please list any chronic medical problems or brain health issues you have on the back side of this form.
5. HORMONES
Are you concerned that hormonal imbalances that may be contributing to your condition? YES NO
6. MOODS & EMOTIONS
How would you describe your general emotional state? (A brief sentence or short phrase of 3-4 words is fine.)
7. MEDICATIONS, SUPPLEMENTS & VITAMINS
If you haven’t previously listed these on our intake form, please provide a list here including name, dose, frequency and for what symptom you are taking each. Feel free to use the other side.
Medications Nutrition Supplements/Vitamins
ANY KNOWN MEDICATION ALLERGIES? YES NO
Please list any medication allergies you may have:
8. SUBSTANCES
Do you currently use psychoactive drugs, medications or alcohol to pick yourself up or calm yourself down? YES NO Have you ever used psychoactive drugs, medications or alcohol in the past to pick yourself up or calm yourself? YES NO Are you currently a smoker? YES NO Do you consider your current use of tobacco, alcohol or street drugs a problem? YES NO If yes on any of these substances, circle those currently taking.
Do you feel depressed or anxious at the present time? Depressed Anxious Neither Have you suffered from depression or anxiety in the past? Circle condition if yes. YES NO
9. ATTENTION & LEARNING
Any history of learning difficulties? YES NO
Any history of memory problems? YES NO
Any history of ADD/ADHD? YES NO In childhood? Adulthood? (please circle)
10. OTHER CONDITIONS
Any history of other psychiatric conditions in yourself, such as schizophrenia, bi-polar disorder , psychosis? YES NO Any history of other psychiatric conditions in family members, such as Schizophrenia, bi-polar, psychosis? YES NO
11. COUNSELING & PSYCHOTHERAPY
Are you currently working with a psychiatrist, therapist, counselor or clergy in matters regarding your mental health? YES NO If yes, please list name/names
12. SEIZURES or LIGHT SENSITIVITY?
Are you, or have you ever been, sensitive to lights or strobe lights, had or been diagnosed with migraines or epileptic seizures? YES NO
13. Is there anything that you would like to add?
Parent or Guardian of Minor, please complete this section
Parent/Guardian Name
Address City State Zip
Do you live with the patient? Y N Phone
SESSION DETAILS RECORD
Patient Name ____________________________
Date Settings today Session Notes
Session #____ ___/___/___ Site ___/____ TR time (min) = _________ _____ (#) X _____(min) Session #____ ___/___/___ Site ___/____ TR time (min) = _________ _____ (#) X _____(min)
Program # _____ Downtrain Uptrain Enhance _____ Hz (AVE) Color: B G Y none Eyes: open closed Reward _____ Media used: Music CD Movie DVD Stopping point: ___:___ Program # _____ Downtrain Uptrain Enhance _____ Hz Color: B Y none Eyes: open closed Reward _____ Media used: Music CD Movie DVD Stopping point: ___:___
PROTOCOL WORKSHEET & CONSENT FORM (Patient and Physician to retain copies)
Patient Name ____________________________________________________ Date of Birth ___/___/____ Brain Map & Assessment A 12-lead qEEG brain map may was recommended for the patient named above by ___________________(Practitioner Name, title, credentials) on ___/___/____. The brain map and assessment of outcome was performed on ___/___/____. Training Recommendations Pt initials here ____ I have been informed by ____________________(Neurofeedback practitioner name, title) that Neurofeedback is an emerging therapy that has shown benefits for many conditions, and is being actively researched for the treatment of depression, anxiety, insomnia, fibromyalgia, chronic pain, learning difficulties, PTSD and more. Recent studies demonstrating efficacy are available to review on our website and we will post any new studies as they become available. Number &Type of Training Sessions Pt initials here ____ Based on your brain map results, a total of____ (#) of NFB training sessions are recommended. We suggest that the following training sessions be considered as outlined here: Protocol _______________________ YBGnone EO EC Enhance/Suppress _____Hz Rational _____________________________________________________________________ Current symptoms _____________________________________________________________ Protocol _______________________ YBGnone EO EC Enhance/Suppress _____Hz Rational _____________________________________________________________________ Current symptoms _____________________________________________________________ Protocol _______________________ YBGnone EO EC Enhance/Suppress _____Hz Rational _____________________________________________________________________ Current symptoms _____________________________________________________________ Re-assessment Recommendation Pt initials here ____ I understand that a follow-up brain map is recommended to assess progress and/or revise training protocols after ____(#) NFB training sessions. Technical services at ___NAME OF CLINIC HERE____ will be managed and/or provided by ______________________(NFB practitioner name) under the direction of ___________________(licensed healthcare provider name, credentials, State in which licensed, license number). Neurofeedback is a form of brain-based biofeedback that offers potential benefits for improved cognitive, emotional, psychological, or metabolic functioning. My Training Goals As we have discussed, my primary brain health training goals are: patient writes his/her goals here
Precautions Pt initials here ____ If I am epileptic, have sensitivity to lights or a history of seizures, I have received clearance from a qualified healthcare professional to proceed with appropriate NFB training. Pt initials here ____ If you have had a brain injury or suffer from any mental disorder or psychiatric illness, I understand that it is recommended that I proceed with NFB training with the consent of my doctor, with the guidance of our qualified staff in-house. General Recommendations for Neurofeedback Training Pt initials here ____
1. Remain hydrated daily during your training period of ___(estimated #) weeks. Drink water or clear liquids appropriate to your physical condition.
2. Prior to your NFB training session, drink a 6-8 oz glass of water (15-30 minutes prior). 3. We will apply training programs and coaching as outlined above. Changes in protocol will be
discussed with you prior to implementation. 4. Arrive early for your session, if possible. To get the most out of your personal training session,
quiet your mind and relax your body as you enter the training room. 5. Most sessions are offered in a seated or semi-reclined position. Please let us know if you have
requirements or requests for specific seating arrangements. 6. During each session, our personal trainer for the brain will “check-in” with you. You may be
asked about your diet, sleep, exercise, moods, activities, etc. since your last NFB session. 7. If at any time during the session, you begin to feel discomfort, including (but not limited to)
vertigo, nausea, euphoria or déjà-vu, or if you experience a mental instability, please let your trainer know immediately. Your trainer will supervise your training sessions and may check-in with you periodically during each session.
8. We recommend that you begin your training with 3-5 sessions the first week, then ___ sessions/week starting at ___ weeks.
9. If your medications or supplements change during the time you are receiving brain health training, please let your trainer know as a change in protocol may be in order.
10. If new symptoms arise, please let your trainer know in person, by email or by telephone. Our number is listed on the front side of this sheet. Please send emails to:
Medical Release I understand that I agree to inform my primary care physician, therapists, and my NFB provider if taking any neurologic or psychiatric medications, or if suffering from a psychological disorder. With my informed consent, _______(NFB practitioner) may speak with my designated providers and therapists and/or fax them reports to them, if a team approach is desired. Appropriate Medical Release Forms will be obtained by me prior to the release of any medical information. Informed Consent I give my informed consent to participate in NFB training and/or brain health nutrition, exercise or behavioral coaching sessions at Revolutionary MD. Please check one for each line below: I ___do ___do not have epilepsy/seizure disorder..
I ___do ___do not have a family history of epilepsy/seizure disorder.
I ___do ___do not have bipolar disorder.
I ___do ___do not currently use psychoactive drugs
I___do ____do not currently use alcohol excessively.
Signature of patient or responsible party, parent or guardian Name_____________________________________________ Signature_____________________________________ Date _____________________ BCN/ RMD staff initial ___________________ Date_____________________