Download - Neonatal Bilious Vomiting- part1
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NEONATAL BILIOUS VOMITING- Part 1
A PROBLEM ORIENTED APPROACH
Dr.B.Selvaraj MS;Mch; FICS;Professor of Surgery
Melaka Manipal Medical CollegeMelaka 75150 Malaysia
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OBJECTIVESTo discuss the differential diagnosis of biliary emesis in neonates
To do appropriate workup to confirm the diagnosis
To select the various treatment options
To make you confident in managing a newborn with bilious vomiting
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Neonatal Bilious Vomiting-Causes
Meconium Peritonitis
Necrotising Enterocolitis
Hirschsprung’s Disease
Anorectal Malformation
Rarely Mesentric Cyst & Intestinal Duplication
Incarcerated inguinal hernia
Duodenal atresia/stenosis
Annular Pancreas
Malrotation&MGV
Intestinal Atresia: Jejunal&Ileal
Meconium Ileus
Meconium Plug
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Neonatal Bilious Vomiting-Causes
• MALROTATION & MGV• MESENTRIC CYST& DUPLICATION CYST• CONGENITAL BANDS LIKE VI DUCT BANDS
• MECONIUM ILEUS• MECONIUM PLUG• MECONIUM PERITONITIS
EXTRINSIC
• DUODENAL ATRESIA/STENOSIS• JEJUNAL/ILEAL ATRESIA• HIRSCHSPRUNG’S DISEASE• NECROTISING ENTEROCOLITIS
CAUSES
MURAL
INTRALUMINAL
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DUODENAL ATRESIA/STENOSIS
Failure of vacuolisation & recanalisation of solidcord state of duodenum at 7 to 10 wks of intrauterine life
Proximal Stomach & Duodenum get dilated and hypertrophied
Bilious vomiting in Postampullary type
Failure to pass meconium
Minimal upper abdominal distension
Hydramnios in mother& Down’s syndrome in the child
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DUODENAL ATRESIA/STENOSIS- Types
1. Membrane Type
a. Simple
b. Fenestrated
c. Windsock Anomaly2. Complete Mural discontinuity with connecting fibrous cord3. Complete Mural discontinuity without connecting fibrous cord
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DUODENAL ATRESIA/STENOSIS- Types
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Duodenal Atresia/StenosisWorkup
Antenatal USG Abdomen
Double Bubble appearance
Postnatal AXR
Classical Double Bubble Appearance
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Kimura’s Diamond ShapedDUODENODUODENOSTOMY
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Duodenal Atresia- Windsock anomaly
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Duodenal Atresia- Post op care
Dysmotility due to Megaduodenum may require a period of TPN
Transanastomotic feeding tube may obviate the need for TPN
Graded introduction of enteral feeds as bowel motility recovers
Prophylactic antibiotics for 48 hrs
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ANNULAR PANCREAS
A rim of pancreatic tissue encircles 2nd part of duodenum
A defect in rotation and fusion of ventral analgae with the dorsal analgae of pancreas
Clinical picture and radiological findings are akin to Duodenal Atresia
Treatment also same as that of Duodenal Atresia
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ANNULAR PANCREAS
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PREDUODENAL PORTAL VEIN
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MALROTATION- Embryology
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Physiological Umbilical Hernia in Fetus
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MALROTATION
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MALROTATION- Different Degrees
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MALROTATION
Any defect/ deviation of normal midgut rotation leads to Malrotation
60% of Malrotation patients present in neonatal period
Most common type of Malrotation is caused by Ladd’s band due to arrest of rotation at 180*
Midgut volvulus is due to narrow duodenocolic isthmus
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MALROTATION
Bilious Vomiting
Passing scanty meconium
Upper abdominal distension
In Midgut volvulusBleeding PR,abdominal distension and vomiting
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MALROTATION- IMAGING STUDIES
AXR- “ Double Bubble Appearance”
Upper GI Series:In Simple MalrotationAbsence of C loop; DJ flexure & jejunal loops on the right side of abdomenIn MGV “Corkscrew Appearance”
USG with Doppler scan:
Reversed position of SMA & SMV
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MALROTATION- IMAGING STUDIES
Double Bubble Appearance Corkscrew Appearance
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MALROTATION- IMAGING STUDIES
Absence of C LoopJejunum on Rt side
Reversed position ofSMA & SMV
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MALROTATION- Ladd’s Procedure
Division of Ladd’s band
Widening of Duodenocolic isthmus
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Malrotation with Midgut Volvulus
Derotation of Volvulus
If bowel is viable leave it
If bowel not viable Resection and EEAIf bowel viability is doubtful Second look laparotomyComplication Short bowel syndrome
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JEJUNAL & ILEAL ATRESIA
Due to mesenteric vascular accident during fetal life
Incidence 1 in 3000 livebirthsPresent within 24hrs with bilious vomiting,not passed meconium & abdominal distension Proximal obstruction earlier & more severe is the bilious vomiting Distal obstruction more severe is the abdominal distension
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JEJUNAL & ILEAL ATRESIA Types
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JEJUNAL & ILEAL ATRESIA Types
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JEJUNAL & ILEAL ATRESIA- AXRJejunal Atresia
Triple Bubble Appearance
ILeal atresia Multiple airfluid levels
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JEJUNAL & ILEAL ATRESIA
Barium Enema
Unused Microcolon
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JEJUNAL ATRESIA- Tapering Jejunoplasty
End to back Anastomosis
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Jejunal & Ileal Atresia- Operative Techniques
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NECROTISING ENTEROCOLITIS
Disease of paradoxes- unknown etiologyMost likely mechanism vascular compromise to GIT resulting bacterial invasion of portal venous system
Common in premature babies
Occurs during 1st or 2nd wk of life after starting oral feedings in babies weighing < 1.5 kgs
Distal Ileum & Rt colon are commonly involved
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NECROTISING ENTEROCOLITIS
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NECROTISING ENTEROCOLITIS
Affected bowel Dilated with mucosal necrosis and subserosal collection of gas
Bilious vomiting,abdominal distension,rectal bleeding and/or diarrheaAbdominal wall edema, erythema and fixed persistent loop of bowelAXR Pneumatosis intestinalis, Gas in portal vein and/or Free air in peritoneal cavity
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NECROTISING ENTEROCOLITIS- Staging
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NECROTISING ENTEROCOLITIS
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NECROTISING ENTEROCOLITIS- AXR
Pneumatosis Intestinalis
Portal Venous Gas—Pneumobilia
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NECROTISING ENTEROCOLITIS
ManagementStart aggressive medical treatment immediately
Keep NPO,NGT aspiration & TPN
Broadspectrum Antibiotics
Physical, radiographic and ultrasonographic evaluation Q6H for 1st 48 hrs in NICU
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NECROTISING ENTEROCOLITIS
Indications for SurgeryPneumoperitoneum & signs of peritonitis
Edematous & Erythematous anterior abdominal wall
Fixed persistent loop of bowel
Portal venous gas- Pneumobilia
Sudden deterioration of baby during medical treatment
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NECROTISING ENTEROCOLITIS Surgery
Operative strategy depends on extend of involvement of bowel
If perforation is small Direct suture closure or re section & primary anastomosis is adequate
In extensive bowel necrosis Remove all gross gangrenous bowel& do enterostomy
In doubtful bowel viability Second look laparotomy
In low birth weight infants with poor general condition do just peritoneal drainage
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TAKE HOME MESSAGE
“YELLOW COLOR VOMITUS IS THE RED SIGNAL OF INTESTINAL OBSTRUCTION UNLESS PROVED OTHERWISE”
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