cva case pres
TRANSCRIPT
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Patients BackgroundPersonal Data
Name of Patient: Mr. X
Age: 64 y/o
Status: Married
Sex: Male
Admitting Diagnosis :T/C Sepsis S/S CVA
Final Diagnosis :CVA ( Cerebrovascular Accident)
Past Medical History :Hypertension, DM Type II
Vital Signs :BP = 130/80 ; PR = 72 ; RR = 22 ;
T = 37.5 C
Brief History:
The patient has been bedridden since he had stroke last April 2007, he also
had difficulty in swallowing and have a necrotic ulceration at the sacrum and
olecranon prior to admission. Mr. Ex-Men had a sedentary lifestyle, he loves eating
fatty foods and food with high calories, he also have a family background on
hypertension. He only stays at home and doesnt exercise. In 1987, he also had
stroke due to hypertension, but didnt take any maintenance drug after that. He
only takes Calcibloc 10mg, whenever he feels the signs and symptoms ofhypertension.
Review of Systems
- Physical Assessment
Vital signs : T 37.5 C PR 72 RR 22 BP 130/80
Consciousness : obtunted open eyes, responds slowly, confused
Bed-ridden
Hemi paresis - left side of the body
Wt: 47 kgs. Ht: 5 5
Weight ( loss [+] gain [ - ] ) Fever [ - ] Night Sweats [ - ]
Significance: weight loss of about 4-5 kgs.
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SKIN
Change in color [ - ] , Ulcers [ + ] ,Rashes [ - ] , Pruritus [ - ] , Bruising
[ - ] ,
Nevi ( change in color [ - ] / shape [ - ] / size [ - ] )
Significance: Sacrum - stage 3 decubitus ulcer (ulcer extends to
subcutaneous fat layer) , Olecranon process of ulna stage 2 decubitus
ulcer (skin is broken to epidermis, abrasion and blister present)
Oxygen is given via nasal canula at 2-3 lpm to deliver more oxygen to
blood
enhance wound healing
HEAD
Headache [ - ] , Injury [ - ] , Tenderness [ - ]
Hair & scalp condition ( infection/ infestation) [ - ], texture silky [ - ] ,
oily [ + ]
Significance: oily hair need to improve hygiene
EYES
Change in visual field [ - ] , Glasses [ - ] , Diplopia [ - ] , Inflammation [
- ] ,
Discharge [ - ]
Significance: none
NOSE
Allergies [ - ] , Sinus problem [ - ] , Obstruction [ - ] , Polyps [ - ] ,
Discharge [ - ], Epistaxis [ - ]
Significance: none
Throat
Toothaches [ - ] , Loose teeth [ - ] , Dentures [ - ] , Bleeding gums [ - ] ,
Mouth sores [ - ] , Difficulty in swallowing [ + ] , Ulceration or lesion on
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tongue, gums, buccal mucosa [ - ]
Significance: NG tube feeding
Respiratory
Chest pain [ - ] , Dyspnea [ + ] , Cough [ - ] , Amount and color of
sputum
[ - ] , Hemoptysis [ - ]
Significance: Ventilation machine
Oxygen is given via nasal canula at 2-3 lpm to ease labored breathing
Cardiovascular
Chest pain / pressure / tightness [ - ] , Orthopnea [ + ] , Claudication [ -
]
Significance: Bed reclined to ease breathing
Gastro-Intestinal
Dysphagia( solid & fluid ) [ + ] , Ulcer [ - ] , GERD [ + ] , Indigestion [ - ]
,
Diarrhea, Constipation [ - ] , Blood in stool [ - ] , Hemorrhoids [ - ],
Jaundice [ - ]
Significance: NG tube feeding
Genito-Urinary
Dysuria / Hematuria / Nocturia [ - ] , UTI [ - ] , Incontinence [ +/- ]
Significance: Bed reclined to ease breathing
Endocrine
Heat / Cold tolerance [ - ] , Weight change [ + ] , Polydisia [ - ] ,
Polyuria [ - ] ,
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Polyphagia [ - ]
Significance: Weight loss of about 4-5 kgs.
Musculo-Skeletal
Limitation of movement [ + ] , Stiffness [ - ] , Joint swelling/redness [ - ]
,
Arthritis [ - ] , Muscle spasm [ - ] , Muscle weakness [ + ]
Significance: Hemi paresis left side of the body
Psychiatric
Mood change [ - ] , Anxiety [ - ] , Suicidal thoughts/ attempts [ - ]
Significance: none
Laboratory Results
Hematology Report
Normal Values Results
emoglobin M 140 170 g/LF 120 160 g/L
96
ematocrit M 0.42 0.52F 0.37 0.470.28
ed Blood Cells Count (RBC) M 4.5 5.5 x 1012/LF 4.0 5.2 1012/L
2.73
hite Blood Cells Count (WBC) 4 10 x 109/L 15.5
atelet Count 150 400 x 109/L 395
hers:
fferential Count
gmenters 0.50 0.70 0.93
ymphocytes 0.20 0.40 0.05
onocytes 0.02 0.07 0.02
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Clinical Chemistry Report
Normal Values R
Creatinine B2 M 80 115 umol/LF (53-97)
1
SGPT B2 0 41 u/L 5
Total CHON B2 65 80 g/L 6
Albumin B2 35 50 g/L 3
Globulin B2 15 35 g/L 3
Cholesterol B2 3.6 6.5 mmol/L 2
Triglycerides B2 0.4 1.86 mmol/L
HDL B2 > 0.90 mmol/L 0
LDL B2 < 3.4 mmol/L
VLDL B2 0.18 0.84mmol/L 0
DRUG STUDY
NAME OF DRUG/
GENERIC NAME
INDICATION AND
DOSAGE
ACTION SIDE EFFECTS
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Name of Drug:
TAZOCINCommon Name:
2.25 g vial
Piperacillin Na 2 g
Tazobactam Na 250 mg4.5 g vial
Piperacillin Na 4 g
Tazobactam Na 500 mg
Indication:
Treatment of Systemic andor local bacterial
infections caused by
sensitive organisms;
UTI, lowerrespiratory tract,
intra-abdominal; skin
and skin structureinfections; bacterial
septicemia;
polymicrobicinfections.
Dosage:
Adult & Children > 12yr.
2.25-4.5 g/day 6-8
hours or 12 hourly byIM or IV infusion
(.20-30 min)
Penicillins are usually
bactericidal in action.They bind reversibly
to several enzymes
outside the bacterial
cytoplasmicmembrane. These
enzymes, known as
penicillin-bindingproteins (PBPs) are
involved in cell-wall
synthesis and celldivision.
Interference with
these processes
inhibits cell-wall
synthesis, causingrapid destruction of
the cell.
Diarrhea, constipation,
nausea, headache,insomnia, rash,
erythema, pruritus
vomiting, allergic
reactions, urticariaand super infection
NAME OF DRUG/
GENERIC NAME
INDICATION AND
DOSAGE
ACTION SIDE EFFECT
Name of Drug:
DUAVENT
Common Name:Per 2.5 ml pulmoneb
Ipatropium Br 500 mcg
Salbutamol sulfate 2.5mg
Indication: Bronchodilator
for treatment of
bronchospasmassociated with
COPD, including
chronic bronchitis,emphysema and
asthma
Dosage:
1-2 pulmoneb every 6-8hours
Inhibits vagally mediated
reflexes by
antagonizingacetylcholine
CNS nervousness,
dizziness, head
Cardiovascular palpitations
EENT blurred visio
epistaxisGI nausea, GI distr
dry mouth
Respiratory cough,
respiratory tracinfection, bronc
bronchospasm
Skin - rash
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NAME OF DRUG/
GENERIC NAME
INDICATION AND
DOSAGE
ACTION SIDE EFFECTS
Name of Drug:
AEKNILCommon Name:
Paracetamol
Indication:
Pyrexia of unknown origin.Fever and pain
associated with
common childhooddisorders, tonsillitis,
upper respiratory tract
infections, postimmunization
reactions, after
tonsillectomy andother conditions.
Prevention of febrileconvulsion.
Headache, cold,sinusitis, muscle pain,
arthritis and
toothache.Dosage:
2-3ml depending on the
severity of case, dosemay be repeated 4
hourly. In severe of
cases, dose may beadministered by IVvery slowly
May produce analgesic
effect by blockingpain impulses by
inhibiting
prostaglandin or painreceptors sensitizers.
May relieve fever by acting
in hypothalamus heat-regulating center
Hematologic hemo
anemia, neutropleucopenia,
thrombocytope
Hepatic liver dama(with toxic dos
jaundice
Metabolic
hypoglycemia
Skin rash, urticaria
Anatomy & Physiology
Lobes of the Brain
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The manifestations of stroke depend on which region and the extent of brain
affected.
The three main components of the brain:
Cerebrum - the largest and most developmentally advanced part of the
human brain. It is responsible for several higher functions, including higher
intellectual function, speech, emotion, integration of sensory stimuli of all
types, initiation of the final common pathways for movement, and fine control
of movement.
Cerebellum - second largest area, is responsible for maintaining balance and
further control of movement and coordination.
The brain stem is the final pathway between cerebral structures and the
spinal cord. It is responsible for a variety of automatic functions, such as
control of respiration, heart rate, and blood pressure, wake-fullness, arousal
and attention.
Cerebrum is divided into a right and a left hemisphere and is composed of
pairs of frontal, parietal, temporal, and occipital lobes.
The left hemisphere controls the majority of functions on the right side of the
body, while the right hemisphere controls most of functions on the left side of
the body The crossing of nerve fibers takes place in the brain stem. Thus,
injury to the left cerebral hemisphere produces sensory and motor deficits on
the right side, and vice versa.
Cerebellum and Brainstem
A stroke involving the cerebellum may result in a lack of coordination,
clumsiness, shaking, or other muscular difficulties. These are important to
diagnose early, since swelling may cause brainstem compression or
hydrocephalus
Strokes in the brainstem are usually due to basilar occlusion, although in
many cases the clinical syndrome may fit the criteria for a lacunar stroke.
Brainstem strokes can be serious or even fatal. People who survive may be
left with severe impairments or remain in a vegetative state.
Major Blood Vessels
Normal function of the brain's control centers is dependent upon adequate
supply of oxygen and nutrients through a dense network of blood vessels.
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Blood is supplied to the brain, face, and scalp via two major sets of vessels:
the right and left common carotid arteries and the right and left vertebral
arteries.
The common carotid arteries have two divisions.
The external carotid arteries supply the face and scalp with blood.
The internal carotid arteries supply blood to the anterior three-fifths of
cerebrum, except for parts of the temporal and occipital lobes.
The vertebrobasilar arteries supply the posterior two-fifths of the cerebrum,
part of the cerebellum, and the brain stem.
Any decrease in the flow of blood through one of the internal carotid arteries
brings about some impairment in the function of the frontal lobes. This
impairment may result in numbness, weakness, or paralysis on the side of
the body opposite to the obstruction of the artery. Occlusion of one of thevertebral arteries can cause many serious consequences, ranging from
blindness to paralysis.
Circle of Willis
At the base of the brain, the carotid and vertebrobasilar arteries form a
circle of communicating arteries known as the circle of Willis.
From this circle other arteries -- the anterior cerebral artery (ACA), the
middle cerebral artery (MCA), the posterior cerebral artery (PCA) - arise and
travel to all parts of the brain. Posterior Inferior Cerebellar Arteries (PICA),which branch from the vertebral arteries, are not shown.
Because the carotid and vertebrobasilar arteries form a circle, if one of
the main arteries is occluded, the distal smaller arteries that it supplies can
receive blood from the other arteries (collateral circulation).
Anterior Cerebral Artery
The anterior cerebral artery extends upward and forward from the
internal carotid artery. It supplies the frontal lobes, the parts of the brain that
control logical thought, personality, and voluntary movement, especially the
legs. Stroke in the anterior cerebral artery results in opposite leg weakness.
If both anterior cerebral territories are affected, profound mental symptoms
may result (akinetic mutism).
Middle Cerebral Artery
The middle cerebral artery is the largest branch of the internal carotid.
The artery supplies a portion of the frontal lobe and the lateral surface of the
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temporal and parietal lobes, including the primary motor and sensory areas
of the face, throat, hand and arm and in the dominant hemisphere, the areas
for speech. The middle cerebral artery is the artery most often occluded in
stroke.
Posterior Cerebral Artery
The posterior cerebral arteries stem in most individuals from the basilar
artery but sometimes originate from the ipsilateral internal carotid artery.
The posterior arteries supply the temporal and occipital lobes of the left
cerebral hemisphere and the right hemisphere.
When infarction occurs in the territory of the posterior cerebral artery, it
is usually secondary to embolism from lower segments of the vertebral
basilar system or heart.
Clinical symptoms associated with occlusion of the posterior cerebralartery, depend on the location of the occlusion and may include thalamic
syndrome, thalamic perforate syndrome, Weber's syndrome, contralateral
hemiplegia, hemianopsia and a variety of other symptoms, including color
blindness, failure to see to-and-fro movements, verbal dyslexia, and
hallucinations. The most common finding is occipital lobe infarction leading to
an opposite visual field defect.
Lenticulostriate Arteries
Small, deep penetrating arteries known as the lenticulostriate arteries
branch from the middle cerebral artery Occlusions of these vessels orpenetrating branches of the circle of Willis or vertebral or basilar arteries are
referred to as lacunar strokes. About 20% of all stokes are lacunar and have a
high incidence in patients with chronic hypertension and in the elderly CT
scanning shows signs of infarction in only approximately half of the most of
the common form of lacunar stroke (pure motor stroke), but MRI has
increased the yield
Pathophysiology
1.Disruption of blood to a part of the brain
2.Oxygenation of Neurons reduced
3.Anaerobic glycolysis
4. Sodium pump & neurotransmitter fail
5. Cerebral edema
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6. Cerebral blood Vessel walls swell; Circulation is compromised and vasospasm
combined with blood viscosity reduced cerebral perfusion
7. Outcomes of decreased cerebral perfusions: cerebral anoxia; cerebral
infarction; contralateral deficits; permanent deficit
Types of Stroke:
(1) Ischemic Stroke
Transient ischemic Stroke (TIA) - is a short-term stroke that lasts for less
than 24 hours. The oxygen supply to the brain is restored quickly, and
symptoms of the stroke disappear completely. A transient stroke needs
prompt medical attention as it is a warning of serious risk of a major stroke
Cerebral thrombosis occurs when a blood clot (thrombus) forms in an
artery (blood vessel) supplying blood to the brain. Furred-up blood vessels
with fatty patches of atheroma (arteriosclerosis) may make a thrombosismore likely. The clot interrupts the blood supply and brain cells are starved of
oxygen.
Cerebral embolism is a blood clot that forms somewhere in the body
before travelling through the blood vessels and lodging in the brain. This
causes the brain cells to become starved of oxygen. An irregular heartbeat or
recent heart attack may make you prone to forming emboli.
http://www.netdoctor.co.uk/diseases/facts/atherosclerosis.htmhttp://www.netdoctor.co.uk/diseases/facts/coronarythrombosis.htmhttp://www.netdoctor.co.uk/diseases/facts/atherosclerosis.htmhttp://www.netdoctor.co.uk/diseases/facts/coronarythrombosis.htm -
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(2) Hemorrhagic Stroke - occurs when a blood vessel bursts inside the brain andbleeds (hemorrhages). With a hemorrhage, extra damage is done to the brain tissue
by the blood that seeps into it..
Signs and Symptoms of CVA
Altered consciousness or unconsciousness
stertorous breathing
Unequal pupils ( larger pupil noted on same side as stroke)
Paralysis of one side
Perspiration
Aphasia (abnormal or absent of speech)
Blank stare
Dysphagia
Inability make decisions
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Loss of memory
Gait instability