coma 1st aid by dr.mohamed el-abiad
TRANSCRIPT
Loss Of Loss Of ConsciousnessConsciousness
Dr. Mohamed El-Abiad
Approach to the Comatose Patient
Initial Treatment
Circulation Airway Breathing ABC - identify and address life threatening
inadequacies Treat rapidly progressive metabolic disorders Evaluate for intracranial hypertension and
imminent herniation and treat
Dr. Mohamed El-Abiad
Management of the Comatose Patient
Circulation
Is patient in shock? Check pulses, heart rate, blood pressure, perfusionRemember hypotension is late sign of shock
Start treatment for shockDo not restrict fluids in comatose patient with
inadequate intravascular volume. Cardiac output and cerebral perfusion are much
more important than fluid restriction
Dr. Mohamed El-Abiad
Use isotonic solutions and blood, as indicated.
Do not use hypotonic solutions to treat shock, particularly patients with coma or possible cerebral edema
Identify life threatening hemorrhage and control it.
Management of the Comatose Patient
Circulation
Dr. Mohamed El-Abiad
Management of the Comatose Patient Airway
Evaluate -- is airway patent. Can patient move air without obstruction. Is there trauma or foreign body obstructing airway
Try chin lift to help open airway -- protect cervical spine
Place airway if indicated - nasal or oral airway, intubation, or surgical airway
Dr. Mohamed El-Abiad
Management of the Comatose Patient
Breathing
Evaluate - is patient moving adequate air, is respiratory rate appropriate, is gas exchange adequate, are breath sounds adequate and symmetrical
Must assure oxygenation and ventilation If intubated don’t forget to ventilate Identify and immediately treat problems -
pneumothorax, airway obstruction, etc..Dr. Mohamed El-Abiad
Glasgow Coma ScaleGlasgow Coma Scale
Three components. Score derived by adding the score for each component.
• Eye opening (4 points)• Verbal response (5points)• Best motor response (6 points)
Dr. Mohamed El-Abiad
Eye opening• 4 - spontaneous• 3 - to speech• 2 - to pain• 1 - none
Verbal Response• 5 - oriented• 4 - confused conversation• 3 - inappropriate words• 2 - incomprehensible sounds• 1 - none
Best Motor Response• 6 - obeys• 5 - localizes• 4 - withdraws• 3 - abnormal flexion• 2 - abnormal extension• 1 - none
Glasgow Coma ScaleGlasgow Coma Scale
Dr. Mohamed El-Abiad
Glasgow Coma ScaleGlasgow Coma Scale
Score > 13 …… Fully Consciousness
Score < 3 …… Deeply Comatosed
Between 3-13 …… Disturbed Consciousness
Dr. Mohamed El-Abiad
Management and Evaluation of the Comatose Patient
Practicalities
During ABC’s and Tests:– Have someone start IV and obtain labs
ABG’sToxin screens
– As soon as IV in and giveGlucose (D25, 2 - 4 cc per kilogram)Consider thiamin
Dr. Mohamed El-Abiad
Intra CranialIntra Cranial
CausesCauses
Extra CranialExtra Cranial
Dr. Mohamed El-Abiad
Pupil Size
Pupil Direction
Deviation Of Angle Of mouth
Salivation
Buccinator Muscle
Hypotonia
Positive Babainiski SignDr. Mohamed El-Abiad
Pulse
Pupil
Patient
Blood Pressure Dr. Mohamed El-Abiad
Pulse
Pupil
Patient
Blood Pressure Dr. Mohamed El-Abiad
Pulse
Pupil
Patient
Blood Pressure Dr. Mohamed El-Abiad
Pulse
Pupil
Patient
Blood Pressure Dr. Mohamed El-Abiad
Pulse
Pupil
Patient
Blood Pressure Dr. Mohamed El-Abiad
HypertensionHypertension
Dr. Mohamed El-Abiad
EdemaEdema
HemorrhageHemorrhageDr. Mohamed El-Abiad
Subdural HematomaSubdural Hematoma
Dr. Mohamed El-Abiad
Acute epidural hematoma and midline shiftAcute epidural hematoma and midline shift
Dr. Mohamed El-Abiad
Herniation syndromesHerniation syndromes
Dr. Mohamed El-Abiad
HypertensionHypertensionLasix
Capoten
Nitroglycerin
Dr. Mohamed El-Abiad
DiabeticDiabetic
Dr. Mohamed El-Abiad
DiabeticDiabetic
Dr. Mohamed El-Abiad
Hypoglycemia (Low Blood Sugar)Hypoglycemia (Low Blood Sugar)
CAUSES: Too little food, too much insulin or diabetes medicine, or extra exercise.
ONSET: Sudden, may progress to insulin shock.BLOOD SUGAR: Below 70 mg/dL. Normal range: 70-115 mg/dL
WHAT CAN YOU DO?
Drink a cup of orange juice or milk or eat several hard candiesTest Blood sugarWithin 30 minutes after symptoms go away, eat a snack e.g. sandwich, and a glass of milkContact doctor if symptoms don't stop
Dr. Mohamed El-Abiad
Dr. Mohamed El-Abiad
Hyperglycemia (High Blood Sugar)Hyperglycemia (High Blood Sugar)
CAUSES: Too much food, too little insulin, illness or stress.
ONSET: Gradual, may progress to diabetic coma.
BLOOD SUGAR: Above 200 mg/dL.Normal range: 70-115 mg/dL
WHAT CAN YOU DO?Test blood sugarIf over 250mg/dL for several tests, CALL YOUR DOCTOR!
Dr. Mohamed El-Abiad
Diabetic ketoacidosisDiabetic ketoacidosis
Dr. Mohamed El-Abiad
Diabetic ketoacidosisDiabetic ketoacidosis
Symptoms
Nausea and vomiting Fruity breath (breath odor)
Stomach pain
Deep, rapid breathing
Flushed face
Dry skin and mouth
Muscle stiffness or aching Headache Shortness of breath Decreased consciousness Decreased appetite Abdominal pain FatigueBreathing difficulty while lying downFrequent urination or thirst for a day or moreMental stupor that may progress to coma Dr. Mohamed El-Abiad
Diabetic ketoacidosisDiabetic ketoacidosis
Dr. Mohamed El-Abiad
Testing for Diabetic KetoacidosisTesting for Diabetic Ketoacidosis
Ketone testing may be used in type 1 diabetes to screen for early ketoacidosis. The ketones test is done using a urine sample. Ketone testing is usually done at the following times:
•When the blood sugar is higher than 240 mg/dL •During an illness such as pneumonia, heart attack, or stroke •When nausea or vomiting occur •During pregnancy
Other tests that may be done to diagnose ketoacidosis include:
•Arterial blood gas •Blood glucose test •Blood pressure measurement •Amylase blood test •Potassium blood test
Dr. Mohamed El-Abiad
KetoacidosisKetoacidosis
Insulin
NaHCO3
IV Fluids
Dr. Mohamed El-Abiad
Hepatic EncephalopathyHepatic Encephalopathy
Dr. Mohamed El-Abiad
CausesCauses::
(1) Virus hepatitis(2) Cirrhosis of liver, (3) Biliary cirrhosis, (4) Toxic hepatitis, (5) Infantile cirrhosis of liver, (6) Hepato toxic drugs, (7) Carcinoma of liver, (8) Portal hypertension, (9) Toxic nitrogenous products are absorbed from the large gut, (10)Defective synthesis of urea in the liver, etc.
Dr. Mohamed El-Abiad
Triggered byTriggered by : :
•Dehydration•Eating too much protein•Electrolyte abnormalities (especially a decrease in potassium) from vomiting or taking diuretics•Bleeding from the intestines, stomach, or esophagus•Infections•Kidney problems•Low oxygen levels in the body•Shunt placement or complications (portosystemic shunt )•Surgery•Use of medications that suppress the central nervous system (such as barbiturates or benzodiazepine tranquilizers)
Dr. Mohamed El-Abiad
Signs and SymptomsSigns and Symptoms::
Symptoms many begin slowly and gradually worsen, or they may begin suddenly and be severe from the start.
)A (Stage of pre-coma
)1 (Alteration in behaviour.(2) Impairment of memory and other intellectual functions.(3) Confusion and even delirium.(4) Slurring of the speech.(5) Inversibn of sleep rhythm.(6) Convulsion—local or generalised.(7) Restlessness(8) Disorientation about space time and persons.(9) Flapping tremor in the outstretched hand and fingers, wrist and even in the shoulder (Bat’s wing tremor).
Dr. Mohamed El-Abiad
Signs and SymptomsSigns and Symptoms::
)B) Stage of coma
)1 (Patient is in deep coma,(2) Muscles are flaccid.(3) Planter may be extensor absent.(4) Deep reflexes absent. [Cerebral disturbance (encephalopathy)]
Dr. Mohamed El-Abiad
DiagnosisDiagnosis::
• Complete blood count or hematocrit to check for anemia• CT scan of the head • EEG• Liver function tests• Prothrombin time• Serum ammonia levels• Sodium level in the blood• Potassium level in the blood• creatinine to see how the kidneys are working
Signs of liver disease, such as:yellow skin eyes (jaundice) fluid collection in the abdomen (ascites)occasionally a musty odor to the breath and urine
Tests may include :
Dr. Mohamed El-Abiad
Dr. Mohamed El-Abiad
(1) Complete bed rest. (2) Diet should contain more carbohydrate and less protein. (3) Sedatives(4) Good food and polyvitamins may be effective. (5) Drugs—sedate cause of anxiety and treated(6) Gastrointestinal bleeding must be stopped (7) Infections, kidney failure, electrolyte abnormalities
(especially potassium) need to be treated. (8) Life support may be necessary to help with breathing
or blood circulation, particularly if the person is in a coma.(9) Lactulose may be given to prevent intestinal bacteria from
creating ammonia , and as a laxative to remove blood from the intestines. Neomycin may also be used to reduce ammonia production by intestinal bacteria.
(10) Medications containing ammonium (including certain antacids) should also be avoided.
TreatmentTreatment
Dr. Mohamed El-Abiad
ComplicationsComplications
Prognosis
Acute hepatic encephalopathy may be treatable. Chronic forms of the disorder often keep getting worse or continue to come back.
Both forms may result in irreversible coma and death. Approximately 80% (8 out of 10 patients) die if they go into a coma. Recovery and the risk of the conditionreturning vary from patient to patient.
•Brain herniation•Brain swelling•Increased risk of:
•Cardiovascular collapse•Kidney failure•Respiratory failure•Sepsis
•Permanent nervous system damage•Progressive, irreversible coma•Side effects of medications
Dr. Mohamed El-Abiad
Renal Failure
Earthy Looking
Puffiness of eyelid in the morning
Lower limb edema
Dr. Mohamed El-Abiad
Dr. Mohamed El-Abiad