assessment of fecal incontinence
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Assessment of Fecal Incontinence. Why should we be interested?. Common problem Can be iatrogenic Results of surgery frequently imperfect C an have an adverse effect on quality of life Significant cost for the Society. Introduction. - PowerPoint PPT PresentationTRANSCRIPT
Assessment of Fecal Incontinence
Why should we be interested?
• Common problem• Can be iatrogenic• Results of surgery frequently imperfect• Can have an adverse effect on quality of
life• Significant cost for the Society
Common medical problem that is under-reported to physicians
Second leading cause of nursing home placement
3% of women who give birth by vaginal delivery will develop Some degree of FI
Introduction
Incidence and prevalence
Perry et al, 2002. Prevalence of faecal incontinence in adults aged 40 years or more living in the community
Background: definition• Faecal incontinence is defined as
involuntary loss of faeces• Commonly classified according to:
– character of leakage– symptom– presumed primary underlying cause
Diagnosis
• HISTORY
• EXAMINATION
• INVESTIGATION
History
• LISTEN to what is being said
• LISTEN to the problem
• LISTEN to the effect on their life
• Define incontinence: flatus vs. stool (liquid vs. solid) • Characterize frequency, duration, severity• Soiling?...fistula, prolapse, hemorrhoids • Urgency? ..... decreased rectal compliance • Medications: laxatives, antibiotics, pancreatic enzyme • Past surgical history: ano-rectal, obstetric
Initial evaluation History
Examination of the anus• Skin tags, fissures, fistulas• Descent• Gape• Strain• Length and angle• Muscle bulk• Voluntary contraction
The specific questions
• Defaecation• Consistency• Urgency• Frequency• Leakage
Pathophysiology and Etiology
Partial incontinence – loss of control to flatus and minor soiling
Major incontinence – frequent and regular deficiency in the ability to control stool of normal consistency
Normal Continence
External sphincter: - Somatic innervation - 15% continence
Internal sphincter: - Visceral innervation - 85% continence
Secondary Musclesof continencePrimary Muscles
of continence
External Anal Sphincter
Fecal Incontinencephysiologic factors
stool consistency rectal and anal sensation
rectal compliancepelvic floor function
can lead to a defective continence mechanism
Fecal Incontinence Altered stool consistency
Inflammatory bowel diseaseInfectious diarrheaLaxative abuseRadiation enteritisShort bowel syndromeMalabsorption syndrome
Fecal IncontinenceInadequate rectal compliance
Inflammatory bowel diseaseAbsent rectal reservoir (ileoanal, low ant. resection)Rectal neoplasmsRadiation TherapyCollagen vascular disease (scleroderma, amyloidosis, dermatomyositis)
Fecal Incontinence Inadequate rectal sensation
Dementia, CVA, MS, brain or spinal cord injury/neoplasm, sensory neuropathy
Diabetes – multifactorial, impaired rectal sensation is important
Overflow incontinenceFecal impaction – leading cause of incontinence in institutionalized elderly patients
Fecal IncontinenceDescending perineal syndrome
Constant straining during defecation
Traction neuropathy of the nerves
Denervation of puborectalis and EAS
The reflex responsiveness of the anal region
Fecal incontinence associated with spinal cord injury
Fecal IncontinenceSphincter defect (Internal and/or External)
Traumatic
Obstetric injury prolonged difficult labor (forceps
application) episiotomy complications
Anorectal surgery anal fistula surgery (most common)
hemorrhoidectomy
Incidence of Perineal Trauma
• 90% of incontinent women with an obstetric history have a sphincter defect (Burnett, S.J. BJS 1991)
• Women with 30/40 tear
– 74% Symptomatic– 59% Incontinent of Gas– 90% Sphincter Defect (Goffeng, A.R. Act.OGS 1998)
• 35% of Primiparous women will have a sphincter defect after delivery (13% symptomatic) (Sultan, A.H. NEJM 1993)
Childbirth & Fecal Incontinence
• 549 prospective fecal urgencyvag 7.3% vsCS 3.1% Chaliha 99 Obstet Gyn
259 consecutive women delivered single unit31 elective CS no FIPrimaparous delivered vaginally 13% FI
Abromowitz Dis Colon Rectum 2000
Incontinence after birth
MacLennan and collegues, BJOG 2000
No births
Caesareansection
Vaginal delivery
Instrumental delivery
Stress 11% 33% 41% 44%
Urge 4% 10% 19% 20%
Faecal 2% 4% 5% 11%
How often do these problems occur?
The Mechanism Of Obstetric Injury
Obstetric InjuryMechanisms
Rectovaginal septum - rectocoele
Ischaemic injury - fistula
Sphincter complex - incontinence
Investigations
FunctionAno-rectal
ManometryAno-rectal
Electrophysiology
StructureEndoanal Ultrasound Magnetic Resonance
ImagingDefecography
MorphologyEndoscopy
Anorectal manometry
Anorectal manometry
Measurement of both resting and voluntary sphincter squeeze pressure
Incontinent patients – low resting and voluntary squeeze pressure
Estimate threshold for rectal sensation/compliance, recto-anal inhibitory reflex
Anorectal manometry in fecal incontinence
Anal Endosonography
An ultrasound probe is placed in the anal canal or transvaginally to detect sphincter injuries and to evaluate pelvic floor structures
Normal anatomy as viewed by anal endosonography
Normal anatomy as viewed by anal endosonography
Faecal IncontinenceStructural Defect
Electrophysiologic testsEMG – needle electrodes into the superficial portion of the external sphincter or puborectalis muscle – myoelectric activit
Pudendal nerve terminal motor latency – measures the delay between the application of an electrical stimulus and external sphincter muscle response. Prolonged – pudendal neuropathy
SPHINCTEROPLASTYPNTML & Neuropathy
Is PNTML reliable in predicting poor outcome ?
• difficult to quantify neuropathy• cut-off value• value of unilateral prolonged latency
Defecography
Evacuation is monitored with flouroscopy
Assessment of the anorectal angle at rest and during defecation
Excessive perineal descent, failure of the puborectalis muscle to relax, rectocele and internal intususception
Summary• Listen to the story• Ask the questions• Examine the bottom• Do the tests• Fit the jigsaw together• Consider the alternatives for treatment