seminar on carbon monoxide poisoning
Post on 16-Jul-2015
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Incomplete combustion –wood/charcoal
Automobile exhaust
Fire
Tobacco smoke
Endogenous CO from haeme degradation (non-toxic on its own)
Affinity to Hb: 230-270 times
Leftward shift of Oxyhaemoglobin dissociation curve
Decreased ability of carrying and releasing Oxygen
Acute Poisoning Early: Non-specific
2 classical features (rare):Cherry red colour – blood + tissues
Cutaneous bullae/blisters
Based on severity:Mild (COHb <30%)
Moderate (30-40%)
Severe (>40%)
Moderate Chest pain
Blurred vision
Confusion
Weakness
Increasing dyspnea
Tachycardia
Tachypnea
Ataxia
Severe Trismus
Muscle Spasms
Convulsions
Palpitations
Disorientation
Vent arrhythmias
Hypotension
MI
Resp failure
Coma
Headache, dizziness, confusion
Weakness, nausea, vomiting, abdominal pain
Paraesthesias
Visual disturbances
Hypertension
Hyperthermia
Cherry red skin
Palpitations
Aggravation of Angina
Alcoholic intoxication
Hyperventilation syndrome
Cerebrovascular accident
Meningitis/encephalitis
Migraine
Epilepsy
Food poisoning
Imm removal from contaminated env
100% O2 -endotracheal tube
Monitor C&D
Neurologic exam + CAT scan + fundoscopic exam
If ICT increased Hyperventilation
Head elevation
Mannitol
Cherry red colour of skin, blood, tissue
Cutaneous bullae
Pulmonary, cerebral edema
Necrosis and cavitation of Basal ganglia
Collect blood from peripheral vein. Preservative: Sodium fluoride.
Suicidal Increased suicide in western countries earlier by
‘Putting the head in the gas oven’
Now, suicide via inhaling exhaust fumes
Accidental Incomplete combustion of natural gas (ill-ventilated)
Defective exhaust system of automobile
Conglagration – large building in flames
• Homicidal (rare)
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