ventricular tachycardia

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Ventricular Tachycardia Dr. Akshay Chincholi

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Page 1: Ventricular tachycardia

Ventricular Tachycardia

Dr. Akshay Chincholi

Page 2: Ventricular tachycardia

Ventricular Tachycardia

• Arrhythmia of three or more consecutive complexes in duration originating from the ventricles at a rate of ≥100 bpm

• Accounts for up to 80% of Wide complex tachycardia• Nonsustained VT

– Three or more beats in duration, terminating spontaneously in less than 30 s.

• Sustained VT– VT greater than 30 s in duration and/or requiring termination

due to hemodynamic compromise in less than 30 s.

Page 3: Ventricular tachycardia

• Rate 100 to 250 / min• Rhythm- Regular / slightly irregular • QRS duration– RBBB >140ms– LBBB >160ms

• QRS Axis – 20% of VTs have a northwest axis (sensitivity 20%,

specificity 96%)– Right axis deviation with LBBB suggests VT(sensitivity

20%, specificity 97%)– In patients with history of MI, axis deviation greater

than 40° from the baseline ECG

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• QRS Concordance – A concordant pattern is defined as predominant

QRS deflection, which is either all positive or all negative across the precordial leads V1 to V6

– occurs in only 15% of VTs with specificity of >90%– positive concordance seen in 18% of VTs with

RBBB-type pattern– 12% of LBBB-type VTs showed negative

concordance

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• AV dissociation – Complete AV dissociation - 20% to 50%– 1:1 VA relation - 30%– 2:1 retrograde (VA) conduction and retrograde

Wenckebach block - 15%–20%– sensitivity of 20%–50%, with specificity

approaching 100%

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Capture and Fusion QRS Complexes• A capture complex occurs when a supraventricular

impulse propagates through the normal HPS system between VT QRS complexes and excites both ventricles completely

• Capture complexes are narrow QRS complexes similar to sinus complexes

• Fused QRS complexes are those in which the QRS is a combination of 2 sources of ventricular activation (supraventricular and ventricular during VT)

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QRS mprphology• In Precordial leads

– Concordant– No R/S pattern– Onset of R to nadir longer than 100 msec

• RBBB pattern– qR, Rs or Rr´ in V1

– broad R (>40 ms) in lead V1– rS complex in lead V6

• LBBB pattern– r in V1 longer than 30 msec– R to nadir of S in V1 greater than 60 msec– Notching in the downstroke of the S wave– qR or qS in V6

• Other specific patterns in V1– Rs and W configuration in V1

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No RS Pattern

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KINDWALL CRITERIA

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KINDWALL CRITERIA

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MILLER CRITERIA

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Idiopathic Ventricular Tachycardia

• Refers to VT of unknown cause that occurs in the absence of structural heart disease or transient or reversible arrhythmogenic factors (e.g., electrolyte disorders, myocardial ischemia).

• Based on the location of the VT– Outflow tract tachycardia– Annular VT– Fascicular VT (left septal VT)

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Outflow Tract Tachycardia

• Accounts for 50% of idiopathic VT and 10% of all VTs

• Types – RVOT VT (80%)– LVOT VT (20%)

• Occurs in young to middle age patients • Mechanism – cAMP mediated delayed

afterdepolarizations

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• RVOT VT– LBBB pattern + Inferior axis – R wave transition at or later than V3

• LVOT VT – 2 patterns on ECG– RBBB pattern + Inferior axis ( aortomitral continuity) – LBBB pattern + Inferior axis + R wave transition V2 (basal

aspect of superior LV septum)• The prognosis for most patients RV/LV OT VT is good.• Vagal maneuvers, Beta blockers, verapamil & adenosine

can terminate the VT• exercise, stress,caffeine, isoproterenol infusion, and rapid

or premature stimulation often initiate or perpetuate the tachycardia.

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RVOT VTLBBB pattern and Inferior axis in frontal plane

The precordial R wave transition occurs at or later than V3

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LVOT VTLBBB pattern + Inferior axis - R wave transition occuring in V1

to V2 - prominent R wave is seen in lead I

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Annular Ventricular Tachycardia

• VTs arising from the mitral or tricuspid annulus• Accounts for 4% and 7% of cases of idiopathic

VT• Mitral annular VT – RBBB – S wave in V6

– monophasic R or Rs in leads V2 through V6

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Mitral Annular VT

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• Tricuspid Annular VT – foci generally arises from the septal region – LBBB morphology in V1– Early transition in precordial leads (V3)– Relatively narrower QRS complex

• Annular VTs behave similarly to outflow tract VT, both in prognosis and in drug response

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Fasicular VT(Left Septal VT)

• Accounts for 7-12% of idiopathic VT• Also called as verapamil sensitive tachycardia• Presents in young adulthood with slight male

preponderance• Mechanism – macroreentry using the left

posterior (or less commonly anterior) fascicle and abnormal purkinje or adjacent ventricular myocardium

• Prognosis is generally good

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Left Posterior Fasicular VTRBBB morphology + Left axis deviation

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Left Anterior Fasicular TachycardiaRBBB + Right Axis Deviation

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Bidirectional VT

• ECG– RBBB pattern– Axis alternating from −60 to −90 degrees to +120

to +130 degrees – regular rhythm

• Causes – Digitalis toxicity – CPVT

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Bidirectional VT

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Torsades de Pointes

• Refers to a VT characterized by QRS complexes of changing amplitude that appear to twist around the isoelectric line and occur at rates of 200 to 250/min

• characterized by prolonged ventricular repolarization with QT intervals generally exceeding 500 milliseconds

• VT that is similar morphologically to torsades de pointes and occurs in patients without QT prolongation, should generally be classified as polymorphic VT, not as torsades de pointes

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Torsades de Pointes

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Accelerated Idioventricular Rhythm

• Enhanced ectopic ventricular rhythm with at least 3 consecutive ventricular beats, which is faster than normal intrinsic ventricular escape rhythm but slower than ventricular tachycardia

• Ventricular rate between 50 and 100 bpm• Causes– acute myocardial infarction shortly after successful reperfusion– digitalis toxicity

• Does not affect prognosis in acute MI• No treatment required

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Accelerated Idioventricular Rhythm

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Ventricular Flutter

• Sine wave pattern – large regular oscillations without clear cut definitions of QRS complex and T waves ocurring at a rate of 150-300/min

• Difficult to distinguish between rapid VT & V.flutter

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Ventricular fibrillation

• Recognized by the presence of irregular undulations of varying contour and amplitude occuring at a rate of 400-600/min

• Distinct QRS complexes, ST segments, and T waves are absent.