summary of ecg abnormalities
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Summary of ECG Abnormalities
Abnormality ECG sign Seen in Pathology
Sinus rhythm regular p waves, andeach p wave is followedby a QRS. 60-100bpm
All leads (best tolook at the
rhythm strip)
None
Sinus Tachycardia Same as above, except>100bpm
All leads (best tolook at the
rhythm strip)
Does not represent cardiacpatholoy. May be a sign of anxiety,dehydration, recent exercise, orgeneral illness (e.g. sepsis,pneumonia, respiratory pathology,other illness)
Sinus bradycardia Same as above except
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Complete (third degree)heart block
90 P waves/min, onlyabout 38 QRS/min, and
not relationshipbetween the P waves
and the QRScomplexes. QRS will
often have anabnormal shape, and
be broad (>120ms).
However, the P-Pintervals will be regular,as will the R-R intervals they are just not intime with each other.
The rhythm of theventricles is the escape
rhythm.
Best in II and V1 This is an AV node block.Atrialactivity will be completely normal,
but this conductivity does not passinto the ventricles.
This always indicates underlyingdisease the disease is oftenfibrosis rather than ischaemia, butit can occur in MI.
RBBB right bundlebranch block
ECG may appearnormal. In some people
there may be 2 Rwaves. This creates a
distinctive pattern:V1 there is an Mshaped QRS this issometimes called anRSR patternV6 there is a Wshaped QRSWide QRS (120ms)
These are infra-Hisian blocks. Inbundle branch blockages, the
wave of depolarisation can stillreach the IV septum, then the
PRinterval will be normal andit is. However, the time taken for
the depolarisation to spreadthroughout the ventricles islonger thus QRS complex
duration is lengthened.In the acute setting it may be
caused by MIRBBB may indicate right sideddisease. The two R waves indicate
the depolarisation of the right andleft sides of the heart at differenttimes (the right depolarises afterthe left).You can remember the patternwith the word MarroW there is Min V1, and W in v6, and the rr tellsyou it is on the right!There is NOT specific treatment,and it is often caused by an atrialseptal defect.In the acute setting it may be
caused by MILBBB often indicatesleft sidedheart disease. Remember thepattern with WillaM.Causes:
Aortic stenosis, dilatedcardiomyopathy, acute MI, CADSymptoms:Syncope, and in more severecases heart failure. Those withsyncope and / or heart failure
will usually be treated with apacemaker.
LBBB left bundlebranch block
V1 there is an Wshaped QRSV6 there is a Mshaped QRSWide QRS (>120ms)
The axis can bedeviated either way inBBBs, but it is mostcommonly normal
Sinus bradycardia Normal rhythm
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immediately after MI
Sinus Tachycardia Normal rhythm>100bpm
Anywhere Associated with exercise, fear,pain, haemorrhage,
thyrotoxicosis
Supraventricularrhythms
This is any rhythmthat originates outside
the ventricle
Examples include:-
Sinus rhythms- LBBB- RBBB
Ventricular rhythms
(aka escape rhythms)Atrial escape
Junctional escape
Ventricular escape
Acceleratedidioventricular rhythm
Wide QRS complexes Anywhere
Abnormal p wave (e.g.inverted)
Normal QRSSome normal beats
after the abnormal one
Anywhere This occurs when the SA nodefails to depolarise. Instead,
some other part of the atriumdepolarises and sends the
signal to the ventricles.
No p wavesNormal QRS
Slightly slow rate (max75bpm)
The escape occurs somewhere atthe AV junction. It occurs when
the rate of depolarisation of the SAnode falls below the rate of the AVnode, thus the AV node starts the
beat instead. The resulting
bradycardia reduces cardiacoutput and can cause
symptoms similar to otherbradycardias such as:
-
Dizziness-
Light-headedness- Syncope- Hypotension
Usually the bradycardia can betolerated as long as it is above50bpm
Two types:-
Many p wavesper QRS(complete heartblock)
- Occasionalmissing p wave,followed by longgap, and then aventricular QRS,then normalrhythm
Somewhere along the line the pwaves isnt getting conducted to
the ventricles, and thus theventricles depolarise at their
normal escape rate.
Wide QRSRhythm of about
75bpmNo p waves
Abnormal T waves
Dont confuse this withventricular tachycardia which
requires a HR of >125pbm.Otherwise it looks very similar.
Usually benign and does notneed to be treated.Alsoassociated with MI
Extrasystoles(aka ectopics)
These are easy they are the same as ventricular escapes, except that wherein escapes the escape beat comes after a pause in the rhythm, in
extrasystole, there is an abnormal beat earlier than expected.The QRS complexes are the same as those of sinus rhythm, but there are
usually abnormal p waves that tend to come immediately before or immediatelyafter the QRS.
Inferior MI(probably the rightcoronary artery)
ST elevation II, III, aVF (theinferior leads)
The ST elevation in these leads isoften accompanied by ST
depression in the antero-lateralleads V1-V6, and possibly in
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lead I and aVL
Anterior MI(probably the left
anterior descending)
ST elevation V2-5 theanterior leads
This will also cause deep qwaves. The presence of Q waves
implies a full thicknessinfarction.
Posterior MI ST depression, tall Rwaves
V1-V3 Posterior MI is unusual! Thechanges that occur are oppositeto the changes of other type ofMI. thus the tall R waves are the
opposite of Q waves (remember Qwaves are negative), and ST
depression occurs in place of STelevation
ST elevation MI(STEMI)
ST elevation >2mm in2+ chest leads OR
>1mm in 2+ limbleads,
T-wave inversion (afterseveral hours)
Pathological Q waves(24 hours +)
T waveinversion occurs
within a fewhours of MI,
pathological Qwaves occurseveral daysafter initial MI
Both factors, if they occur, areusually permanent. In a full
thickness infarction then thereare pathological Q waves, and Twave inversion, but in a non-fullthickness MIthen there is only T
wave inversion. Thedifferentiation between full
/thickness and non full thickness ispretty much the same as ST
elevation / non-ST elevationNSTEMI Pathological Q waves
only
Ventriculartachycardia
Wide QRS, no pwaves, T waves difficult
to identify, rate>200bpm
? Can be difficult to differentiatefrom BBB. BBB has p waves, and
a QRS generally 120-160ms. VT ismore likely scenario after MI, and
has QRS >160ms
Supraventriculartachycardia
Narrow QRS
Ventricular fibrillation No discernable
pattern, no QRS, no P,no T
Patient is very likely to lose
consciousness thus thediagnosis is easy!
Wolff-Parkinson-White SYndrome
Delta waves present,right axis deviation,
short PR interval, shortQRS
Accessory pathway, usually fromthe left atria to the left ventricleallows direct transition of thesignal, bypassing the AV node,hence the shortened PR interval. Ithas a risk of mortality as it cancause re-entry tachycardiahowever, most patients aresymptomless and live with noproblems.
The digoxin effect Depression of ST,inverted T waves
widespread This causes a sloping ST segmentthat has a reversed tick look. Thisoccurs because digoxin blocks the
na/K pump, which increasesintracellular Ca2+
concentrations. (similarly,ischaemia causes reduced
production of ATP, and thusreduced pump activity)
Pericarditis T wave inversion (rare:also ST elevation)
Widespread If ST elevation does occur, thenthe ST waves will appear saddle
shaped thus helping you todifferentiate it from MI. also, theelevation in MI tends to be
confined to a certain area, but inpericarditis, it is widespread
P pulmonale Tall ,peaked T waves, p Lead II Seen in cor pulmonale, or pretty
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wave height >2mm inlead II
much anything that causes rightatrial enlargement (or
hypertrophy) such as tricuspidstenosis or pulmonary
hypertension
Bifid P waves (P-Mitrale)
P waves with twopeaks, broad lookslike an M hence the
name Mitrale
? Left ventricular hypertrophy
Bi-phasic T waves T waves with t peaks Can occur as a result of MIProlonged QT interval Prolonged QT The corrected QT, is the QT
interval as it would be at 60bpm. ifthis is long, then there is a risk ofsudden cardiac death. It can be
congenital, but also caused bydrugs
Hyperkalaemia Wide, tall, tented Twaves,
shortened/absent STsegment, small or
absent p waves, wideQRS
? Can lead to VF and AF
Left ventricularhypertrophy
S wave in V1 or V2 >35mm AND R wave in V5 or V6>35mm R in aVF >20mmR in aVL>11mm
Any chest lead >45mmR in lead I>12mm
Pacemaker Occasional P waves,not related to QRS,
QRS precede by largespike, QRS complexesbroad
? The large spike is pacemakerstimulus. The QRSs are wide
because the stimulus originates inthe ventricles
Axis deviation
Lead I Lead II Axis
+ + Normal
+ - LAD
- Either RADaVR should always be negative!If it is positive,it is called north-west axis. it could be due to incorrect limb lead placement,dextrocardia, or artificial pacing, due to the pacemaker wire - this enters the heart at the apex.Carotid sinus pressureBy applying pressure to the carotid sinus you can stimulate the AV and SA nodes via vagalstimulation. This will reduce the frequency of discharge of the SA node, and increase the time ofconduction across the AV node.Thus, by applying pressure to the carotid sinus you can:
Reduce the rate of some arrhythmias
Completely stop some arrhythmiasIt will have NO EFFECT ON VENTRICULAR TACHYCARDIAS thus is can help youdifferentiate.
Applying the pressure basically reduces the frequency of QRS complexes, and allows the
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underlying atrial arrhythmia to become more visible.Copyright 2009 - 2013 - Dr Tom Leach
Source URL (modified on 10/09/2014 - 04:06): http://almostadoctor.co.uk/content/sy stems/-cardiovascular-system/ecgs/summary-ecg-abnormalities