mammalian transport system ch. 8 part 4 heart function
TRANSCRIPT
Mammalian Transport System
Ch. 8 Part 4 Heart Function
Structures to Know• Atria (Atrium) or auricle
• Upper chambers• Receive blood from veins
• Ventricles• Lower chambers• Blood flows into from the atria and out through
arteries• Aorta• Pulmonary artery• Venae cavae (vena cava)• Pulmonary veins• Coronary arteries
• Bring oxygenated blood back to the heart• Septum
• Wall of muscle that separates chambers on the right side of the heart from the chambers on the left side of the heart
• Blood cannot pass through
Valves and Nodes to know• Atrioventricular valves (between the atrium and the
ventricle)• Mitral/bicuspid valve (LEFT SIDE OF HEART)• Tricuspid valve (RIGHT SIDE OF HEART)
• Semilunar valves• Pulmonary Valve• Aortic Valve
• Sinoatrial node (SAN)• Patch of specialized muscle fibers in RIGHT ATRIUM• Pacemaker
• Atrioventricular node (AVN)• Patch of conducting muscle fiber located in upper septum
• Purkyne tissue• Conducting muscle fibers running down septum, along the
right and left ventricles
Valves• Atrioventricular valves
(between the atrium and the ventricle)• Mitral/bicuspid valve
(LEFT SIDE OF HEART)
• Tricuspid valve (RIGHT SIDE OF HEART)
• Semilunar valves• Pulmonary Valve• Aortic Valve
Important!!!!• Heart is myogenic does NOT need outside nerve
impulses to initiate heart beat• Valves in heart do NOT actively open and close• Valves open and close due to PRESSURE changes in
chambers
Closing of valves create thump-thump
of heartbeat
Structure of Heart• Atrium walls thin, muscular walls
• Low pressure exerted• Ventricle walls thick and muscular
• Right ventricle small force needed to push blood to lungs
• Left ventricle large force needed to push blood all over body• VERY MUSCULAR• Greater pressure developed in
left ventricle than left atria
The Cardiac Cycle• Sequence of events that
make up 1 heart beat• Heart beats about 70x a
minute• 3 stages of the cycle
1. Atrial systole2. Ventricular Systole3. Ventricular Diastole
Systole ContractionDiastole Relaxation
Atrial Systole• Heart filled with blood• Muscle in atrial walls (very
thin) contract• Not a lot of pressure from
this contraction• Enough pressure to force
blood in atria through ATRIOVENTRICULAR VALVES (AV) into ventricles
• No back flow of blood into pulmonary veins or vena cava b/c of semilunar valves
Ventricular Systole• Ventricles contract 0.1 s after atria
contracts• Thick, muscular ventricle walls
push blood out (exert high pressure)
• AV valve shut when pressure in ventricles exceeds pressure in atria
• Semilunar valves open• Blood rushes up into aorta &
pulmonary artery• Lasts for 0.3 seconds
Ventricular Diastole • After 0.3 seconds, ventricular muscle
relaxes• Ventricle pressure decreases• Semilunar valves shut, preventing
backflow of blood• Blood only fills in cusps of valves• Blood from veins flow into the 2 atria• Blood is at low pressure• Walls of atria expand to accommodate
blood (very little resistance)• Some blood trickles into ventricles
through AV valve• Atria contracts and cardiac cycle begins
again
Control of the Heart Beat• Myogenic muscles
• Naturally contracts and relaxes• No nerve impulses required
• Sinoatrial Node (SAN) • Set out rhythm for all other muscle
cells to contract• SAN contraction rhythm slightly
faster than the rest of the heart• SAN contracts wave of excitation
(depolarization) sent across all of the atria & muscles of atria contract
Things NOT to say when describing nodes:
• “Signal”• “Wave” alone • “Pulse”• “Message”• “Nerve impulse”You SHOULD describe the function of SAN using:• “Wave of excitation”• “depolarization”• “impulse”
Ventricle contraction• SAN causes contraction of all atria muscles• Ventricle muscles delayed due to band of
fibers between the atria and ventricles that does NOT conduct electric impulses
• Only path for impulses to reach ventricles is through path of conducting fibers in septum called ATRIOVENTRICULAR NODE (AVN)
• AVN receive excitation from atria, delays it 0.1s and then passes it to another bunch of conducting fibers called the PURKINJE FIBERS or PURKYNE TISSUE
• Wave of excitation is sent up ventricle walls—bottom-up
Fibrillation• When muscular walls of heart flutter
rather than contract and relax as a whole
• Rapid, irregular, unsynchronized contraction of muscle cells
• Atrial fibrillation (non fatal) Afib• Can lead to stroke
• Ventrical fibrillation (fatal) Vfib• Faint, cardiac arrest
• Caused by:• Electric shock or damage to large
areas of muscle in walls of heart• Miss firing of electric impulse from
atria• Instead of electrical impulse going
from atria to AVN to Purkinje tissue in the ventricle, all muscle cells in ventricle get excited in all directions
Electrocardiograms (ECGs)• Graph plotting voltage vs. time• Records electrical potentials of heart
over time• Place electrodes over opposite sides of
heart• P= wave of excitation over atrial walls
• Q, R, S= wave of excitation over ventricle walls
• T = recovery of ventricle walls• Contraction time = time b/t Q and T• Filling time = time b/t T and Q
How To Read an ECG• EKG paper is a grid where time is measured along the horizontal axis. • Each small square is 1 mm in length and represents 0.04 seconds. • Each larger square is 5 mm in length and represents 0.2 seconds.
• Voltage is measured along the vertical axis.• 10 mm is equal to 1mV in voltage.
Calculating Heart from ECG
1. Determine rate of strip• Time of strip is given; measure strip with
ruler and divide length by sec of strip or…• May be given 20mm * s-1
2. Measure distance of one cardiac cycle• Beginning of one P to the beginning of the
next P