ASSOCIATION FOR ACADEMIC SURGERY AND SOCIETY OF UNIVERSITY SURGEONS—ABSTRACTS342

59.6. A Novel Method Of Inducing Abdominal Aortic Aneu-rysms In Rodents. A. Laser,1 G. Lu,1 A. Ghosh,1 K. Roelofs,1

B. McEvoy,1 P. DiMusto,1 C. M. Bhamidipati,2 G. Ailawadi,2

J. L. Eliason,1 P. K. Henke,1 G. R. Upchurch1,2; 1University ofMichigan, Ann Arbor, MI; 2University of Virginia,Charlottesville, VA

59.7. The Mechanism Of Diabetes Pathophysiology DiminishesAAA Formation: Glucose Influences AGE-induced MMP-9Expression. G. Banker, S. Marsh, F. Zhang, J. Lengfeld, K.Kent, B. Liu; Department of Surgery, University of WisconsinSchool of Medicine, Madison, Wisconsin

59.8. Angiotensin Receptor Blockers Suppress AbdominalAortic Aneurysm (AAA) In Angiotensin II-inducedApoE Knockout Mice. Y. Iida,1 G. M. Schultz,1 M. M. Dua,1

N. Miyama,1 B. Xu,1 S. Rea Peterson,2 S. Sulaimon,2 R. L.Dalman1; 1Stanford University School of Medicine, Stanford,CA; 2Medtronic Vascular Inc., Santa Rosa, CA

59.9. Effectiveness Of A Novel Limited-Spectrum Matrix Met-alloprotease Inhibitor On Model Aneurysms. T. Ennis, J.Jin, B. Arif, J. A. Curci; Washington University School ofMedicine, St. Louis, MO

59.10. MembraneType1-MatrixMetalloproteinaseAsAPossibleMediator Of Estrogen-Stimulated Matrix DegradationAnd Migration In Vascular Smooth Muscle Cells. D. J.Mountain, S. S. Kirkpatrick, D. C. Cassada, S. L. Stevens,M. B. Freeman, M. H. Goldman, O. H. Grandas; University ofTennessee Graduate School of Medicine, Knoxville, TN

59.11. Effect Of Metabolic Syndrome On The Response To Ar-terial Injury. Y. Fu, E. A. Duru, K. A. Youker, M. G. Davies;Department of Cardiovascular Surgery, Methodist DeBakeyHeart and Vascular Center, Houston, TX

59.12. Changes In MMP2 System Following Arterial Injury InMetabolic Syndrome. Y. Fu, E. A. Duru, Z. Chen, M. G.Davies; Department of Cardiovascular Surgery, MethodistDeBakey Heart and Vascular Center, Houston, TX

59.13. Changes InUPAFollowingArterial Injury InMetabolicSyndrome. Y. Fu, E. A. Duru, Z. Chen, M. G. Davies;Department of Cardiovascular Surgery, Methodist DeBakeyHeart and Vascular Center, Houston, TX

59.14. Differential Modulation Of Proteases And Protease In-hibitors Expression In In Vitro Metabolic Syndrome.E. A. Duru, Y. Fu, M. G. Davies; The Methodist HospitalResearch Institute, Houston, TX

59.15. Modulation Of Leptin, Adiponectin, And Resistin Ex-pression In In Vitro Metabolic Syndrome. E. A. Duru,Y. Fu, M. G. Davies; Methodist Hospital Research Institute,Houston, TX

59.16. Modulation Of Inflammatory Markers By S-1-P In InVitro Metabolic Syndrome. E. A. Duru, Y. Fu, M. G.Davies; Methodist Hospital Research Institute, Houston, TX

59.17. Novel Adipokine Resistin Augments Injury-InducedIntimal Hyperplasia In A Murine Model. R. Ding1;1Department of Surgery, Stanford University, Stanford,California; 2Monta Vista High School, Cupertino, California;3Changhai Hospital, Shanghai, Shanghai; 4Department ofMolecular Environmental Biology, University of California,Berkley, Berkley, California

59.18. Adipocytokine Resistin Increases Endothelial Mono-layer Permeability In Human Coronary Artery Endo-thelial Cells. M. Jamaluddin, L. Zhang, Q. Yao, C. Chen;Baylor College of Medicine, Houston, TX

VASCULAR 3: ENDOTHELIAL CELLDYSFUNCTION

60.1. Pharmacologic Pre-conditioning With The Gasotrans-mitter Hydrogen Sulfide Occurs Through The XanthineOxidase Pathway. N. Jimenez, P. W. Henderson, A. J.Reiffel, Y. H. Millet, J. A. Spector; Weill Cornell MedicalCollege, New York, New York

60.2. Urokinase Requires NAD(P)HOxidase To TransactivateEGFR. M. G. Davies, E. A. Duru, Y. Fu; Methodist DeBakeyHeart and Vascular Center, Houston, TX

60.3. Transactivation Of EGFR1 By Urokinase Requires TheCo-receptor EGFR4. M. G. Davies, Y. Fu, E. A. Duru;Methodist DeBakey Heart and Vascular Center, Houston, TX

60.4. Ginsenoside Rb1 Reverses HomocysteineInduced Endo-thelial Dysfunction In Human Coronary Artery Endothe-lial Cells. L. Zhang,2 R. Ding,1 S. Powell,1 W. Zhou1;1Department of Surgery, Division of Vascular and EndovascularSurgery, Stanford University, Stanf, Stanford, California;2Department of Vascular Surgery, Changhai Hospital, SecondMilitary Medical University, Shanghai, Shanghai, Shanghai

60.5. Synergistic Effect Of Disturbed Flow With ChemicalStimulation By Thrombin On Tissue Factor ExpressionIn HUVEC. R. Abe B. E. Sumpio; Yale University School ofMedicine, New Haven, CT

60.6. Pomegranate Extract Blocks Ritonavir-Induced Endo-thelial Dysfunction In Porcine Pulmonary Arteries.S. M. Weakley, T. J. Beadle, C. Cheng, X. Wang, J. Jiang, P.Kougias, P. H. Lin, Q. Yao, C. Chen; Baylor College ofMedicine, Houston, TX