acute mi myocardial infarction

36
Management of patient with coronary vascular disorders Mrs. Samia Almusalhi

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Page 1: Acute MI Myocardial Infarction

Management of patient with coronary vascular disorders

Mrs. Samia Almusalhi

Page 2: Acute MI Myocardial Infarction

objectives

1. Patho, ss assessment, diagnosis, risk factor prevention medical and nursing management

2. Athero, angina and MI and acute coronary syndrome

3. Types of angina

4. Identify invasive coronary artery procedure

Page 3: Acute MI Myocardial Infarction

Acute coronary syndrome

• Includes all heart disease.

Page 4: Acute MI Myocardial Infarction

atherosclerosis• It is abnormal

Accumulation of fats or

lipids in the arterial of

blood vessels.

Page 5: Acute MI Myocardial Infarction

atherosclerosis

• Patho: fats deposits in intima (inner layer of blood vessels), ---- monocytes (macrophages) migrate to the site--- it release substance that attracts platelets and initiating clotting----- smooth cells of blood vessels proliferate and for fibrous cape called “ atherosclerosis” or “plaque”– it narrows the blood vessels causing less blood flow.

Page 6: Acute MI Myocardial Infarction

S&S

• Ischemia

• Angina (chest pain)

• MI

Page 7: Acute MI Myocardial Infarction

Risk factor

-Non modifiable:• Family history• Increased age• Gender (male more)• Race (more with african americans)-Modifiable:Hyperlipidemia, smoking, DM, obesity,

physical inactivity.

Page 8: Acute MI Myocardial Infarction

Prevention

1. Controlling cholesterol abnormalities: checking lipid profile p 759,

• Dietary measures: high in vegetables and fruits and less of meat, high fiber reduce fiber diet,

• Physical activity: it reduce LDL

• Medication: as Atrovastatin and simvastatin.

Page 9: Acute MI Myocardial Infarction

Cont’ prevention

2. Promoting cessation of tobacco: it affect endothelium leads to thrombus formation

3. Managing HTN: elevated BP leads to stiffness of blood vessels walls,

4. Controlling DM:

Page 10: Acute MI Myocardial Infarction

Coronary artery

Page 11: Acute MI Myocardial Infarction

Angina pectoris

• Pain or pressure in the anterior chest.

• Patho: when there is increased demand of O2 by cardiac muscle to meet its continous work----- and there is obstruction because of atherosclerosis--- blood flow will be affected----- ischemia result------- chest pain starts which is called angina pertoris

Page 12: Acute MI Myocardial Infarction

Factors causing angina pain:

• Physical exertion

• Exposure to cold

• Eating heavy meals

• Stress unstable angina is not associated with

pervious factors (at rest even)

Page 13: Acute MI Myocardial Infarction

Angina pectoris• S&S: severe chest pain

under the sternum impending

to death not relieved by rest

• Chest tightness

• Weakness and numbness in the arms

• Shortness with breathing

• Diaphoresis

• dizziness

• Stable angina relieved with rest or with nitroglycerides.

Page 14: Acute MI Myocardial Infarction

diagnosis

• Ask the patient: site of pain, if it is radiated, how is the pain, when did it begin, how long it last, what helps to release it, any other symptoms associated with it.

• St wave inversion (ischemia)

• Cardiac biomakers testing (CK mb, Troponin)

• Exercise or stress test (treadmill)

Page 15: Acute MI Myocardial Infarction
Page 16: Acute MI Myocardial Infarction

Medical management

1. Pharmacological therapy:

• Nitroglycerin: vasodilator

• Beta-adrenegic blocking agent

To reduce cardiac contractility

• Calcium channel blocking agent:

To slow HR, decrease strength

Of contraction

Page 17: Acute MI Myocardial Infarction

• Antiplatelet and anticoagulant medication:Prevent platelet aggregation and thrombus

formation.e.g: Aspirin and Heparin or Glycoprotien

Page 18: Acute MI Myocardial Infarction

2. Oxygen Administration

• Start O2 even if saturation is highat the onset of pain

• Monitor oxygen level

• Assess skin color, mucous membrane (central and peripheral cyanosis)

• Folwler position

Page 19: Acute MI Myocardial Infarction

• Assess level of pain continuously

• Perform repeated ECG to assess ST segment

• Measure vital signs every 15 minutes or half hour with pain level

Page 20: Acute MI Myocardial Infarction

• Reduce patient anxiety

• Home care related to ( diet, avoid vigorous exercise like stairs, stress management, follow up appointment, balance rest with activity, stop smoking, take sublingual medication once they feel pain, keep it with him or her all the time and not to stop medication by themselves, ,,,,,,,

Page 21: Acute MI Myocardial Infarction

MI

• It myocardial ischemia

Result in death of tissues.

• It is called coronary

occlusion, heart attack but proffered to be called MI.

• In MI there is complete occlusion of coronary artery leading to imbalance between demand and supply------ischemia ----infacrtio or tissue death occure.

Page 22: Acute MI Myocardial Infarction

MI (S&S(

• Severe chest pain

• Shortness of breathing

• Indigestion

• Nausea and anxiety

• Cool, pale skin

Page 23: Acute MI Myocardial Infarction

Diagnostic finding

• ECG

Page 24: Acute MI Myocardial Infarction

Diagnostic finding• Cardiac biomarkers (cardiac enzyme( CK

Mb and myoglobin which not specific to heart and Troponin

SGOT (serum glutamic oxaloacetic transaminase( or aspertate aminotransferase (AST)

• 2. Physical examination

• 3. Patient history: of pain and previous attach and family history.

• 4. ECG changes

Page 25: Acute MI Myocardial Infarction

Treatment guideline for acute MI

• Chart 27-7 page 744:

Transfere to hospital, ECG, blood test, Aspirine, IV heparine,

• MONA treatment

• Careful for side effect of morphin

Page 26: Acute MI Myocardial Infarction

Types of Angina:

• Stable angina: occurs on exertion and relieved by rest

• Unstable angina: pre-infarction angina is not relieved by rest, it increase in frequency caused by spasm in coronary

• Varient angina (Prinzmetal’s(: pain at rest with reversible st segment elevation caused by coronary artery vasospasm

• Silent ischemia: no pain ECG changes with stress test.

Page 27: Acute MI Myocardial Infarction

Invasive coronary artery procedure

1. Percutanious transluminal coronary angioplasty (PTCA(:

Is balloon tipped catheter is used to open blocked coronary artery and for blocked CABAG. It compress the atheroma thus improves blood

flow.

Page 28: Acute MI Myocardial Infarction

Coronary artery stent• After PTCA the area may close off partially

or completely, and the intima of this place has been injured and it might stimulate inflammatory process leading to vasoconstriction and clot formation so----- stent is placed there. Figure 28-8

Page 29: Acute MI Myocardial Infarction

atherectomy

• Is removal of atheroma by cutting or grinding by a catheter.

• It could be used with PTCA

Page 30: Acute MI Myocardial Infarction

brachytherapy

• Involves gamma or beta-radiation by catheter to destroy atheroma.

Page 31: Acute MI Myocardial Infarction

Surgical procedure: coronary artery revascularization

• It is called CABG

1.Traditional Coronary Artery Bypass Graft:

2.Alternative coronary artery bypass graft technique:

Page 32: Acute MI Myocardial Infarction

Traditional Coronary Artery Bypass Graft:

• The surgeon will do incision sternum and connects patient to cardiopulmonary bypass (CPB), next the blood vessels as saphenous vein is grafted distal to the coronary artery lesion (bypassing the obstruction) then the incision isclosed and patient is admitted

in ICU.

Page 33: Acute MI Myocardial Infarction

Traditional Coronary Artery Bypass Graft:

Page 34: Acute MI Myocardial Infarction

Alternative coronary artery bypass graft technique

• It is Off Pump CABG involves median sternotomy incision but without CPB (cardiopulmonary bypass). Beta adrenergic block used to slow HR, then anastomosis of the bypass graft into the coronary artery while the heart continues to beat. p781

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complication

• Bleeding, HTN, Hypovolemia,,,,,,,,,,,

Page 36: Acute MI Myocardial Infarction

Thank You